DβH-ir terminals form asymmetric synapses with NK1R-ir neurons in the pre-BötC.
dAIH induces an increase in DβH expression in the pre-BötC.
dAIH evokes increases in the number and area of NK1R-ir processes in the pre-BötC.
Close appositions of DβH- and NK1R-ir profiles are significantly elevated in the pre-BötC after dAIH exposure.