西北燥证与心系疾病关联性研究
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摘要
目的:1)分析新疆地区心系疾病与西北燥证的罹患关系;2)分析新疆地区心系疾病病情、证情与西北燥证主兼证的相关性;3)分析新疆地区心系疾病患者与西北燥证罹患者素体状态的异同,从而揭示西北燥证对新疆地区心系疾病患者病证状态的影响。方法:运用文献学分析方法及临床流行病学方法,采用病例对照设计,共收集1314份调查问卷,其中病例组486例,对照组828例,对2组西北燥证罹患率、西北燥证证情、气血阴阳及五脏虚证罹患率进行比较。对病例组中燥证组非燥证组病情、临床证候证情、西北燥证证情、虚证证情进行比较。资料全部收回后采取EPIDATA3.0录入,然后导出数据。由SPSS16.0统计软件完成统计分析。病例组与对照组均进行人口学资料均衡性分析。计数资料采用卡方检验,计量资料采用t检验、方差分析。方差不齐者采用t’检验和Kruskal-Wallis秩和检验。所有的统计检验均采用双侧检验,P值小于或等于0.05将被认为所检验的差别有统计意义。计数资料多组间两两比较时,检验水准α' = 2α/k(k-1)。结果:1)心系疾病病例组西北燥证罹患率为37.86%,对照组为18.47%,病例组高于对照组(χ~2=60.329,P<0.01)。各病种病例组西北燥证罹患率均高于对照组(均有P<0.05)。各病种西北燥证罹患率排序从高到低依次为神经衰弱(62.65%)、皮肤瘙痒症(49.35%)、冠心病(31.48%)、高血压(31.37%)、痤疮(24.13%);2)心系疾病组西北燥证总积分高于对照组(P<0.01)。各证候除肺心脾风火燥证两组无差异外,其余主兼证西北燥证积分二者均有显著差异,病例组均高于对照组(P<0.01)。各证候均数差值以心肾阴虚证及肺卫孔窍皮肤燥证最高;3)除高血压外,各病种病例组外燥证均高于对照组(均有P<0.05),尤以皮肤瘙痒症、痤疮明显。高血压、冠心病、神经衰弱三病种内燥证均高于外燥证(均有P<0.05)。皮肤瘙痒症、痤疮内燥证与外燥证积分比较未显示出差异(P>0.05);4)高血压与神经衰弱燥证组病情积分均高于非燥证组(均见P<0.01)。皮肤瘙痒症与痤疮燥证组与非燥证组病情积分未显示出差异(P>0.05);5)高血压阴阳两虚证与燥证总证情相关性最强(r=0.864,P<0.01);典型相关分析提示,西北燥证主兼证与高血压各证有很强关联性,其中因变量组阴阳两虚证、痰湿壅盛证与自变量组脾胃蕴湿证、肝肾精血不足证相关性最强。神经衰弱气郁化火证与西北燥证总证情相关性最强(r=0.692,P<0.01);典型相关分析提示,西北燥证主兼证与神经衰弱各证有很强关联性,其中因变量组肝阴亏虚证、气郁化火证与自变量组肝肾精血不足证、心肾阴虚证相关性最强。皮肤瘙痒症血虚风燥证与西北燥证总证情相关性最强(r=0.517,P<0.01);典型相关分析提示,西北燥证主兼证与皮肤瘙痒症各证有很强关联性,其中因变量组血虚风燥证、湿热壅盛证与自变量组肺心脾风火燥证、脾胃阴虚证相关性最强。痤疮湿热证与西北燥证总证情相关性最强(r=0.391,P<0.01);典型相关分析提示,西北燥证主兼证与痤疮各证有很强关联性,其中因变量组湿热证、风热证与自变量组肺心脾风火燥证、肺卫孔窍皮肤燥证相关性最强;6)高血压病情与西北燥证证情有较强关联度(f_i=0.51)。类证中与肝肾精血不足证(f_i=0.49)、肺心脾风燥证(f_i=0.38)、心肾阴虚证(f_i=0.38)关联度也较强(P<0.01)。神经衰弱病情与西北燥证证情病证切合指数=0.31(P<0.01),提示神经衰弱病情与西北燥证证情有较强关联度。与各类兼证病证切合指数均有P<0.01,尤以肝肾精血不足证病证切合指数(f_i=0.40)最大,其次是脾胃蕴湿证(f_i=0.37)、心肾阴虚证(f_i=0.32)、脾胃阴虚证(f_i=0.32),与肺心脾风火燥证关联度最小(f_i=0.29)。皮肤瘙痒症病情除与肺心脾风火燥证(f_i=0.30,P<0.01)有较强关联度外,其余各证f_i均无统计学意义(P>0.05)。痤疮病情积分与西北燥证病证切合指数经检验均无统计学意义(P>0.05);7)心系疾病病例组气虚证、阳虚证、心虚证、肝虚证罹患率均高于对照组(P<0.05),其他虚证罹患二者无统计学差异(P>0.05)。冠心病组阳虚证、心虚证、肺虚证、肝虚证罹患率高于对照组(均有P<0.01)。高血压组心虚证、肝虚证罹患率高于对照组(P<0.05)。神经衰弱组气虚证、心虚证、肝虚证罹患率高于对照组(P<0.01),其他虚证罹患率与对照组比较无统计学差异(P>0.05)。皮肤瘙痒症组气虚证、阴虚证、阳虚证罹患率均高于对照组(P<0.05),尤以气虚证、阳虚证明显(P<0.01)。痤疮组各类虚证罹患情况与对照组无差异(P>0.05);8)冠心病燥证组气血阴阳虚证及五脏罹患率均高于非燥证组(P<0.01)。高血压燥证组气血阴阳及五脏虚证罹患率均高于非燥证组(P<0.01)。神经衰弱燥证组气虚证、阴虚证、心虚证、肺虚证、肝虚证罹患率高于非燥证组(P<0.01)。血虚证、阳虚证、脾虚证、肾虚证罹患率2组无差异(P>0.05)。皮肤瘙痒症燥证组阴虚证、阳虚证、心虚证、肝虚证、肾虚证罹患率高于非燥证组(P<0.05)。气虚证、血虚证、肺虚证、脾虚证、胃虚证罹患率2组无差异(P>0.05)。痤疮燥证组气虚证、血虚证、阴虚证、肝虚证高于非燥证组(P<0.05)。其他虚证罹患率2组无差异(P>0.05);9)病证组、唯病组、唯证组、非病证组4组虚证比较:既罹患心系疾病又罹患西北燥证的人群体质最差,各类虚证均较明显。其次为西北燥证罹患者,再次为仅患有心系疾病者,而以既无疾病又无西北燥证罹患的人群体质状态最佳。结论:1)西北燥证是新疆地区心系疾病的
危险因素。西北燥证是新疆地区心系疾病的共同临床伴随证候,并以心肾阴虚证为突出表现;2)新疆地区心系疾病病情和证情与西北燥证主兼各证均有较强相关性,随各病种特点,与西北燥证主兼证相关性亲疏不等;3)新疆地区心系疾病组虚证证情较对照组严重,突出表现为气虚、阳虚、心虚和肝虚证重而且多;4)冠心病组以阳虚为主,其次为气虚。五脏虚证中以心虚、肺虚、肝虚较为突出。其中肺虚证是新疆地区冠心病患者特殊素体状态。高血压组以心虚、肝虚为主,亦有气虚表现。这是新疆地区高血压患者有别于内地的体质特点。神经衰弱组气虚、心虚、肝虚较为突出。皮肤瘙痒症组气虚、阴虚、阳虚较为突出。痤疮组因年龄较轻,整体体质状况较好,但亦有肾虚表现。肾虚可能是新疆地区痤疮患者的潜在病理状态;5)西北燥证加重新疆地区心系疾病虚证证情。西北燥证是新疆民众亚健康状态的主要表现形式,防治西北燥证有助于提高新疆地区民众的健康水平。
Objectives: 1) to analyze the relationship of attack rate between the Northwest Dryness Syndrome and heart system diseases in Xinjiang Autonomous Region. 2) to analyze the correlation between the state of illness and syndrome of heart system diseases and NDS in Xinjiang. 3) to analyze the similarities and differences of the physique between the patients of NDS and heart system diseases and to reveal the impact of NDS on the patients’status of heart system diseases. Methods: By applying methods of philology analysis and clinical epidemiology, 1314 questionnaires were designed and collected. On the basis of case-control, disease group (486 cases) and control group (828 cases) are set up to compare the attack rate of northwest dryness syndrome (NDS), NDS integration, deficiency syndrome of Qi, blood, Yin and yang; to compare the state of illness, general syndrome differentiation in traditional Chinese medicine, integrations of NDS and deficiency syndrome of NDS group and non-NDS group in the disease group. After the collection of all questionnaires, all data were put into EPIDATA 3.0 and derived into SPSS 16.0 for statistic analysis. Equilibrium Analysis of demographic data is adopted in both disease group and control group. Enumeration data is analyzed by chi-square. Masurement data is analyzed by T-Test and analysis of variance. Heterogeneity of variance is analyzed with t’and Kruskal-Wallis testing method. Two-side test are applied in all statistic test. The tested difference will be considered to be Statistically Significant when P≤0.05. The significant level isα' = 2α/k(k-1) when the enumeration data is pairwise compared among groups. Results: 1) The attack rate of NDS in heart disease group (37.86%) is higher than control group (18.47%) (χ~2=60.329, P<0.01). The same situation exists in all other disease groups. The attack rate of NDS ranks in descending order as follows: neurasthenia (62.65%), prurituscutanea (49.35%), coronary heart disease (31.48%), hypertension (31.37%), acne (24.13%). 2) The total integration of NDS in heart disease group is higher than control group (P<0.01). There is obvious difference on the NDS integrations of main and accompanying syndromes between these two groups except the lung and heart wind-fire pathogen syndrome, i.e. Disease group is higher than control group. Among all standardized mean differences of syndromes, the highest ones are yin deficiencies of the heart and kidney syndrome and skin and opening of the body syndrome. 3) The outer dryness syndrome in all disease groups but group of hypertension is higher than control groups (P<0.05), especially prurituscutanea and acne. The integrations of inner dryness syndrome of coronary heart disease, hypertension and neurasthenia are higher than that of outer dryness syndrome (P<0.05). The comparison between the integrations of inner dryness syndrome of prurituscutanea and acne shows no difference (P>0.05). 4) The illness integration in groups of hypertension and neurasthenia are higher than those of non-dryness syndrome groups (P<0.01). The comparison between the integrations of dryness syndrome groups of prurituscutanea and acne shows no difference with those of non-dryness syndrome groups (P<0.05). 5) The deficiency of both Ying and Yang in group of hypertension and dryness are the most related with total NDS integration (r=0.864, P<0.01); The canonical correlation coefficient showed that the NDS main and the accompanied symptoms with hypertension has a strong correlation, in which the dependent variable set of deficiency of both yin and yang, phlegm wet obstruct the lung from the independent variable group of spleen-stomach damp syndrome, the deficiencies syndrome of essence and blood of the liver and the kidney are the most relevant. The syndrome of depressed qi transforming into fire of neurasthenia is the most related with total NDS integration (r=0.692, P<0.01); The canonical correlation coefficient showed that the NDS main and the accompanied symptoms with hypertension has a strong correlation, in which the dependent variable set of yin deficiencies of the liver syndrome, the syndrome of qi depression transforming into fire from the independent variable group of the deficiencies syndrome of essence and blood of the liver and the kidney, yin deficiencies of the heart and kidney syndrome are the most relevant. The blood deficiency and wind dryness syndrome in group of prurituscutanea is the most related with total NDS integration (r=0.517, P<0.01); The canonical correlation coefficient showed that the NDS main and the accompanied symptoms with prurituscutanea has a strong correlation, in which the dependent variable set of the blood deficiency and wind dryness syndrome, damp-heat filled syndrome from the independent variable group of the lung and heart wind-fire pathogen syndrome, yin deficiencies of the spleen and stomach syndrome are the most relevant.The damp-heat syndrome of acne is the most related with total NDS integration (r=0.391, P<0.01); The canonical correlation coefficient showed that the NDS main and
the accompanied symptoms with acne has a strong correlation, in which the dependent variable set of damp-heat syndrome, wind-heat syndrome from the independent variable group of the lung and heart wind-fire pathogen syndrome, Lung health skin pore opening dry permit are the most relevant.6) There is a strong correlation between the illness status of hypertension and syndromic state of NDS (f_i=0.51). The illness status of hypertension is also correlated with some accompanying syndromes (P<0.01), eg. deficiencies of essence and blood of the liver and the kidney syndrome (f_i=0.49); the lung and heart wind-fire pathogen syndrome (f_i=0.38), yin deficiencies of the heart and kidney syndrome (f_i=0.38). The corresponding index 0.31 (P<0.01) between the illness status of neurasthenia and syndromic state of NDS shows that there is a strong correlation between them. The corresponding index between the former and other accompanying syndromes are descending sorted as follows: deficiencies of essence and blood of the liver and the kidney syndrome (f_i=0.40), endogenous dampness syndrome (f_i=0.37), yin deficiencies of the heart and kidney syndrome (f_i=0.32), yin deficiencies of the lien and stomach syndrome (f_i=0.32), the lung and heart wind-fire pathogen syndrome (f_i=0.29). the illness status of prurituscutanea is only correlated with the lung and heart wind pathogen syndrome (f_i=0.30, P<0.01) but has no statistic significant with other syndromes (P>0.05). The integration of acne and its corresponding index with NDS show no statistic significant after testing. 7) The attack rates of Qi, Yang, Heart and Liver deficiency in case group are higher than those in control group (P<0.05). There is no difference in statistics on other deficiency syndromes. The attack rates of Yang deficiency (20.95%), heart deficiency (46.67%), lung deficiency (25.71%) and liver deficiency (40.48%) in heart system disease on blood vessel are higher than control group (P<0.05), especially on heart and liver deficiency. The attack rates of Qi deficiency (68.87%), heart deficiency (53.01%) and liver deficiency (43.37%) in heart system disease on mind are higher than control group. The attack rate of Yang deficiency (15.54%) is the only one which is higher than the control group in heart system disease on skin. 8) The attack rates of Qi, Xue, Ying and Yang deficiency syndrome and five internal organs of syndrome groups of coronary disease are higher than the non-dryness groups (P<0.01). The attack rates of Qi, Xue, Ying and Yang deficiency syndrome and five internal organs of syndrome groups of hypertension are higher than the non-dryness groups (P<0.01). The attack rates of Qi, Ying deficiency syndrome and heart deficiency, lung deficiency and liver deficiency syndrome groups of neurasthenia are higher than the non-dryness groups (P<0.01). The attack rates of Ying and Yang deficiency syndrome and heart deficiency, liver deficiency
and kidney deficiency syndrome in groups of prurituscutanea are higher than the non-dryness groups (P<0.01). The attack rates of Qi deficiency, Blood deficiency, Ying deficiency syndrome and liver deficiency syndrome in groups of acne are higher than the non-dryness groups (P<0.01). 9) To compare the disease and syndrome group, disease and the sole-illness group, the sole-syndrome group and the health group: The physical constitution of the disease and syndrome group are the poorest than other groups. The sole-syndrome group listed on the second and the sole-illness group on third. The physical constitution of the non-illness and syndrome group is the best. Conclusion: 1) The northwest dryness syndrome, with yin deficiencies of the heart and kidney syndrome as a prominent feature, is one of risk factors and common accompanying status for heart system disease in Xinjiang Region. 2) There is a strong correlation among the status of heart system diseases and its syndromes. The correlation with the main and accompanying syndromes of NDS varies with the different features of each disease. 3) The group of heart system disease in Xinjiang, asthenia syndrome condition is serious than the control group, which is mainly characterized as Qi deficiency, Yang deficiency, heart deficiency and liver deficiency. 4) Yang deficiency physique is most obvious in the group of coronary disease. The outstanding performance of the five internal organs is heart deficiency, lung deficiency and liver deficiency. Lung deficiency is the special physique for the patients of coronary disease in Xinjiang. The physique of heart deficiency and liver deficiency is evident in hypertension, Qi deficiency is also represented. This is a different physique feature of hypertension patients in Xinjiang and inland. The physique of Qi deficiency, heart deficiency and liver deficiency is evident in neurasthenia; the physique of Qi deficiency, Yin deficiency and Yang deficiency is evident in prurituscutanea. As a whole, the physique in the group of acne is better than other groups. Deficiency of the kidney may be the potential pathological status of the patients of acne in Xinjiang. 5) NDS accentuates the deficiency symptoms of heart system disease. The prophylaxis and treatment of NDS will contribute to improve people's health because it is the dominant manifestation of the sub-health status of people live in Xinjiang.
危险因素。西北燥证是新疆地区心系疾病的共同临床伴随证候,并以心肾阴虚证为突出表现;2)新疆地区心系疾病病情和证情与西北燥证主兼各证均有较强相关性,随各病种特点,与西北燥证主兼证相关性亲疏不等;3)新疆地区心系疾病组虚证证情较对照组严重,突出表现为气虚、阳虚、心虚和肝虚证重而且多;4)冠心病组以阳虚为主,其次为气虚。五脏虚证中以心虚、肺虚、肝虚较为突出。其中肺虚证是新疆地区冠心病患者特殊素体状态。高血压组以心虚、肝虚为主,亦有气虚表现。这是新疆地区高血压患者有别于内地的体质特点。神经衰弱组气虚、心虚、肝虚较为突出。皮肤瘙痒症组气虚、阴虚、阳虚较为突出。痤疮组因年龄较轻,整体体质状况较好,但亦有肾虚表现。肾虚可能是新疆地区痤疮患者的潜在病理状态;5)西北燥证加重新疆地区心系疾病虚证证情。西北燥证是新疆民众亚健康状态的主要表现形式,防治西北燥证有助于提高新疆地区民众的健康水平。
Objectives: 1) to analyze the relationship of attack rate between the Northwest Dryness Syndrome and heart system diseases in Xinjiang Autonomous Region. 2) to analyze the correlation between the state of illness and syndrome of heart system diseases and NDS in Xinjiang. 3) to analyze the similarities and differences of the physique between the patients of NDS and heart system diseases and to reveal the impact of NDS on the patients’status of heart system diseases. Methods: By applying methods of philology analysis and clinical epidemiology, 1314 questionnaires were designed and collected. On the basis of case-control, disease group (486 cases) and control group (828 cases) are set up to compare the attack rate of northwest dryness syndrome (NDS), NDS integration, deficiency syndrome of Qi, blood, Yin and yang; to compare the state of illness, general syndrome differentiation in traditional Chinese medicine, integrations of NDS and deficiency syndrome of NDS group and non-NDS group in the disease group. After the collection of all questionnaires, all data were put into EPIDATA 3.0 and derived into SPSS 16.0 for statistic analysis. Equilibrium Analysis of demographic data is adopted in both disease group and control group. Enumeration data is analyzed by chi-square. Masurement data is analyzed by T-Test and analysis of variance. Heterogeneity of variance is analyzed with t’and Kruskal-Wallis testing method. Two-side test are applied in all statistic test. The tested difference will be considered to be Statistically Significant when P≤0.05. The significant level isα' = 2α/k(k-1) when the enumeration data is pairwise compared among groups. Results: 1) The attack rate of NDS in heart disease group (37.86%) is higher than control group (18.47%) (χ~2=60.329, P<0.01). The same situation exists in all other disease groups. The attack rate of NDS ranks in descending order as follows: neurasthenia (62.65%), prurituscutanea (49.35%), coronary heart disease (31.48%), hypertension (31.37%), acne (24.13%). 2) The total integration of NDS in heart disease group is higher than control group (P<0.01). There is obvious difference on the NDS integrations of main and accompanying syndromes between these two groups except the lung and heart wind-fire pathogen syndrome, i.e. Disease group is higher than control group. Among all standardized mean differences of syndromes, the highest ones are yin deficiencies of the heart and kidney syndrome and skin and opening of the body syndrome. 3) The outer dryness syndrome in all disease groups but group of hypertension is higher than control groups (P<0.05), especially prurituscutanea and acne. The integrations of inner dryness syndrome of coronary heart disease, hypertension and neurasthenia are higher than that of outer dryness syndrome (P<0.05). The comparison between the integrations of inner dryness syndrome of prurituscutanea and acne shows no difference (P>0.05). 4) The illness integration in groups of hypertension and neurasthenia are higher than those of non-dryness syndrome groups (P<0.01). The comparison between the integrations of dryness syndrome groups of prurituscutanea and acne shows no difference with those of non-dryness syndrome groups (P<0.05). 5) The deficiency of both Ying and Yang in group of hypertension and dryness are the most related with total NDS integration (r=0.864, P<0.01); The canonical correlation coefficient showed that the NDS main and the accompanied symptoms with hypertension has a strong correlation, in which the dependent variable set of deficiency of both yin and yang, phlegm wet obstruct the lung from the independent variable group of spleen-stomach damp syndrome, the deficiencies syndrome of essence and blood of the liver and the kidney are the most relevant. The syndrome of depressed qi transforming into fire of neurasthenia is the most related with total NDS integration (r=0.692, P<0.01); The canonical correlation coefficient showed that the NDS main and the accompanied symptoms with hypertension has a strong correlation, in which the dependent variable set of yin deficiencies of the liver syndrome, the syndrome of qi depression transforming into fire from the independent variable group of the deficiencies syndrome of essence and blood of the liver and the kidney, yin deficiencies of the heart and kidney syndrome are the most relevant. The blood deficiency and wind dryness syndrome in group of prurituscutanea is the most related with total NDS integration (r=0.517, P<0.01); The canonical correlation coefficient showed that the NDS main and the accompanied symptoms with prurituscutanea has a strong correlation, in which the dependent variable set of the blood deficiency and wind dryness syndrome, damp-heat filled syndrome from the independent variable group of the lung and heart wind-fire pathogen syndrome, yin deficiencies of the spleen and stomach syndrome are the most relevant.The damp-heat syndrome of acne is the most related with total NDS integration (r=0.391, P<0.01); The canonical correlation coefficient showed that the NDS main and
the accompanied symptoms with acne has a strong correlation, in which the dependent variable set of damp-heat syndrome, wind-heat syndrome from the independent variable group of the lung and heart wind-fire pathogen syndrome, Lung health skin pore opening dry permit are the most relevant.6) There is a strong correlation between the illness status of hypertension and syndromic state of NDS (f_i=0.51). The illness status of hypertension is also correlated with some accompanying syndromes (P<0.01), eg. deficiencies of essence and blood of the liver and the kidney syndrome (f_i=0.49); the lung and heart wind-fire pathogen syndrome (f_i=0.38), yin deficiencies of the heart and kidney syndrome (f_i=0.38). The corresponding index 0.31 (P<0.01) between the illness status of neurasthenia and syndromic state of NDS shows that there is a strong correlation between them. The corresponding index between the former and other accompanying syndromes are descending sorted as follows: deficiencies of essence and blood of the liver and the kidney syndrome (f_i=0.40), endogenous dampness syndrome (f_i=0.37), yin deficiencies of the heart and kidney syndrome (f_i=0.32), yin deficiencies of the lien and stomach syndrome (f_i=0.32), the lung and heart wind-fire pathogen syndrome (f_i=0.29). the illness status of prurituscutanea is only correlated with the lung and heart wind pathogen syndrome (f_i=0.30, P<0.01) but has no statistic significant with other syndromes (P>0.05). The integration of acne and its corresponding index with NDS show no statistic significant after testing. 7) The attack rates of Qi, Yang, Heart and Liver deficiency in case group are higher than those in control group (P<0.05). There is no difference in statistics on other deficiency syndromes. The attack rates of Yang deficiency (20.95%), heart deficiency (46.67%), lung deficiency (25.71%) and liver deficiency (40.48%) in heart system disease on blood vessel are higher than control group (P<0.05), especially on heart and liver deficiency. The attack rates of Qi deficiency (68.87%), heart deficiency (53.01%) and liver deficiency (43.37%) in heart system disease on mind are higher than control group. The attack rate of Yang deficiency (15.54%) is the only one which is higher than the control group in heart system disease on skin. 8) The attack rates of Qi, Xue, Ying and Yang deficiency syndrome and five internal organs of syndrome groups of coronary disease are higher than the non-dryness groups (P<0.01). The attack rates of Qi, Xue, Ying and Yang deficiency syndrome and five internal organs of syndrome groups of hypertension are higher than the non-dryness groups (P<0.01). The attack rates of Qi, Ying deficiency syndrome and heart deficiency, lung deficiency and liver deficiency syndrome groups of neurasthenia are higher than the non-dryness groups (P<0.01). The attack rates of Ying and Yang deficiency syndrome and heart deficiency, liver deficiency
and kidney deficiency syndrome in groups of prurituscutanea are higher than the non-dryness groups (P<0.01). The attack rates of Qi deficiency, Blood deficiency, Ying deficiency syndrome and liver deficiency syndrome in groups of acne are higher than the non-dryness groups (P<0.01). 9) To compare the disease and syndrome group, disease and the sole-illness group, the sole-syndrome group and the health group: The physical constitution of the disease and syndrome group are the poorest than other groups. The sole-syndrome group listed on the second and the sole-illness group on third. The physical constitution of the non-illness and syndrome group is the best. Conclusion: 1) The northwest dryness syndrome, with yin deficiencies of the heart and kidney syndrome as a prominent feature, is one of risk factors and common accompanying status for heart system disease in Xinjiang Region. 2) There is a strong correlation among the status of heart system diseases and its syndromes. The correlation with the main and accompanying syndromes of NDS varies with the different features of each disease. 3) The group of heart system disease in Xinjiang, asthenia syndrome condition is serious than the control group, which is mainly characterized as Qi deficiency, Yang deficiency, heart deficiency and liver deficiency. 4) Yang deficiency physique is most obvious in the group of coronary disease. The outstanding performance of the five internal organs is heart deficiency, lung deficiency and liver deficiency. Lung deficiency is the special physique for the patients of coronary disease in Xinjiang. The physique of heart deficiency and liver deficiency is evident in hypertension, Qi deficiency is also represented. This is a different physique feature of hypertension patients in Xinjiang and inland. The physique of Qi deficiency, heart deficiency and liver deficiency is evident in neurasthenia; the physique of Qi deficiency, Yin deficiency and Yang deficiency is evident in prurituscutanea. As a whole, the physique in the group of acne is better than other groups. Deficiency of the kidney may be the potential pathological status of the patients of acne in Xinjiang. 5) NDS accentuates the deficiency symptoms of heart system disease. The prophylaxis and treatment of NDS will contribute to improve people's health because it is the dominant manifestation of the sub-health status of people live in Xinjiang.
引文
[1]周铭心.西北燥证研究概述[J].上海中医药杂志, 2005, 39(11):43-45.
[2]牟全胜,周铭心.西北多燥说[J].新疆中医药, 1991,卷(4):1-6.
[3]周铭心.燥邪病因属性辨析[J].新疆中医药, 2005, 23(6):1-5.
[4]王燕,周铭心.吴鞠通治燥特色探讨—西北燥证治法相关文献研究[J].新疆中医药, 2006, 24(4):1-3.
[5]姜德,周铭心.燥邪病因与咳嗽的关系探讨--西北燥证病因相关研究[J].新疆中医药, 2006, 24(5):7-9
[6]王学武,刘浩,周铭心.古今燥证论治的研究概况[J].新疆中医药, 2003,21(3): 53-57.
[7]周铭心,陶培勇.沙漠燥证初探-沙漠石油工人保健研究[J].中医杂志, 1997,38 (8):493-496.
[8]周铭心,宋晓平,单丽娟,等.新疆各地不同民族居民西北燥证罹患情况流行病学调查分析[J].新疆医科大学学报, 2006, 29(11):1034-1038.
[9]吕光耀,李杰,周铭心.燥证及西北燥证病因病机研究概述[J].新疆医科大学学报, 2007, 30(1):26-28.
[10]周铭心,宋晓平,单丽娟,等.西北燥证证候类型分析[J].新疆医科大学学报, 2007, 30(1):1-6.
[11]李鹏,周铭心.新疆和田地区维吾尔族居民西北燥证罹患情况及主要症状分析[J].新疆医科大学学报, 2007, 30(1):14-16.
[12]吕光耀,李杰,周铭心.新疆不同地区不同民族居民生存质量与西北燥证关系的分析[J].新疆医科大学学报, 2007, 30(1):6-10
[13]张育善,周铭心.疏风宣卫合剂治疗银屑病70例疗效观察[J].新疆中医药, 1986(3):45.
[14]周铭心,单丽娟,宋晓平,等.西北燥证外感病因六淫构成情况因子分析.新疆医科大学学报, 2006, 29(12):1123-1130.
[15]赵存娥,李明奎.中医病因病机学[M].北京:科学出版社, 2000.
[16]沙塔娜提,孙红友.周铭心.环境地理因素与亚健康状态的关系初探—西北燥证背景研究[J].新疆中医药, 2006, 24(4):74-76.
[17]沈绍功,韩学杰.中医心病学正名与标准化研究[J].中国中医基础医学杂志, 2007, 13(7):485-487.
[18]韩学杰.中医心病学诊疗规范性研究,中华中医药学会内科心病第八届学术年会,安徽芜湖, 2006:20-31.
[19] Andrew Moran, Dong Zhao, Dongfeng Gu, et, al. The future impact of population growth and aging on coronary heart disease in China:projections from the Coronary Heart Disease Policy Model-China[J]. BMC Public Health 2008, 8:394.
[20] International Database, U. S. Census Bureau [http://www. census. gov/ipc/www/ idb/]
[21] National Bureau of Statistics of China[http://www. stats. gov. cn/english/]
[22] Wang L, Kong L, Wu F, Bai Y, Burton R:Preventing chronic diseases in China. Lancet. 2005, 366(9499):1821-1824.
[23] Omran AR:The epidemiologic transition. A theory of the epidemiology of population change[J]. Milbank Memo Fund Q. 1971, 49(4):509-538.
[24] Yusuf S, Reddy S, Ounpuu S, Anand. S:Global burden of cardiovascular diseases: part I:general considerations, the epidemiologic transition, risk factors, and impact of urbanization. Circulation. 2001, 104(22):2746-2753.
[25] Murray CJ, Lopez AD:Alternative projections of mortality and disability by cause 1990-2020:Global Burden of Disease Study. Lancet 1997, 349(9064):1498-1504.
[26] Gaziano TA:Reducing the growing burden of cardiovascular disease in the develo- ping world. Health Aff(Millwood)2007, 26(1):13-24.
[27] He J, Gu D, Wu X, et, al. :Major causes of death among men and women in China. New Engel J Med 2005, 353(11):1124-1134.
[28] He J, Gu D, Wu X, Wildman RP, Klag MJ, Whelton PK:Major causes of death among men and women in China. New Engel J Med 2005, 353(11):1124-1134.
[29] Leeder S, Raymond S, Greenberg H, Liu H, Esson K:A Race Against Time:the Challenge of Cardiovascular Disease in Developing Countries. New York, NY: Columbia University Center for Global Health and Economic Development; 2004. 10.
[30] Murray CJ, Lopez AD:Mortality by cause for eight regions of the world:Global Burden of Disease Study. Lancet 1997, 349(9061):1269-1276.
[31] Critchley J, Liu J, Zhao D, et, al. Cape well S:Explaining the increase in coronary heart disease mortality in Beijing between 1984 and 1999. Circulation 2004,110(10): 1236-1244.
[32] Weinstein MC, Coxson PG, Williams LW, Pass TM, Stason WB, Goldman L. Forecasting coronary heart disease incidence, mortality, and cost:the Coronary Heart Disease Policy Model. Am J Public Health 1987, 77(11):1417-1426.
[33] Kochanek KD, Murphy SL, et al. Final Data for 2002. National Static’s Reports. Vol53. Hyattsville, Maryland:National Center for Health Statistics;
[34] Anonymous. Heart Disease and Stroke Statistics. American Heart Association;2006.
[35] Mackay J, Mensah GA. The Atlas of Heart Disease and Stroke. Geneva:WHP;2004.
[36] Ridker PM, Hennekens CH, Buring JE, Rifai N. C-reactive protein and other markers of inflammation in the prediction of cardiovascular disease in women. N Eng J Med. 2000,342(12):836-43.
[37] D’Aiuto F, Parkar M, Andreou G, Brett PM, Ready D, Tonetti MS. Periodontitis and atherogenesis:causal association or simple coincidence J Clin Periodontol. 2004,31 (5):402-11.
[38] Ridker PM, Hennekens CH, Buring JE, Rifai N. C-reactive protein and other markers of inflammation in the prediction of cardiovascular disease in women. N Eng J Med. 2000,342(12):836-43.
[39] Noack B, Genco RJ, Trevisan M, Grossi S, Zambon JJ, De Nardin E. Periodontal infections contribute to elevated systemic C-reactive protein level. J Periodontol. 2001, 72(9):1221-7.
[40] Wu T, Trevisan M, Genco R, et al. Examination of the relation between periodontal health status and cardiovascular risk factors:serum total and high-density lipoprotein cholesterol, C-reactive protein, and plasma fibrinogen. Am J Epidermal. 2000, 151:273-82.
[41] Ajwani S, Mattila KJ, Tilvis RS, Ainamo A. Periodontal disease and mortality in an aged population. Spec Care Dent. 2003, 23(4):125-30.
[42] Glurich I, Grossi S, Albini B, et al. Systemic inflammation in cardiovascular and periodontal disease:comparative study. Clin Diagn Lab Immunol. 2002,9(2):425-32.
[43] Janket SJ, Baird AE, Chuang SK, Jones JA. Meta-analysis of periodontal disease and risk of coronary heart disease and stroke. Oral Surg Oral Med Oral Pathol Oral Radiol Endo. 2003, 95(5):559-69.
[44] Khader YS, Albashaireh ZS, Alomari MA. Periodontal diseases and the risk of coronary heart and cardiovascular diseases:a meta-analysis. J Periodontal. 2004, 75(8):1046-53.
[45] Linda L. Humphrey, Rongwei Fu, David I. Buckley, et, al. Periodontal Disease and Coronary Heart Disease Incidence:A Systematic Review and Meta-analysis.
[46] Marmot M, Shipley M, Brunner E, Hemingway H. Relative contribution of early life and adult socioeconomic factors to adult morbidity in the Whitehall II study. J Epidemiol Community Health. 2001, 55:301-307.
[47] Davey Smith G, Ben-Shlomo Y, Lynch J. Life course approaches to inequalities in coronary heart disease risk. In:Stansfeld S, Marmot M, eds. Stress and the Heart. London, England:BMJ Books. 2002,20-49.
[48] Notkola V, Punsar S, Karvonen MJ, Haapakoski J. Socioeconomic conditions in childhood and mortality and morbidity caused by coronary heart disease in adult- hood in rural Finland. Soc Sci Med. 1985, 21:517-523.
[49] Kaplan GA, Salonen JT. Socioeconomic conditions in childhood and ischemic heart disease during middle age. BMJ. 1990, 301:1121-1123.
[50] Gliksman MD, Kawachi I, Hunter D, et al. Childhood socioeconomic status and risk of cardiovascular disease in middle-aged US women:a prospective study. J Epidemiol Community Health. 1995, 49:10-15.
[51] Wannamethee SG, Whincup PH, Shaper G, Walker M. Influence of fathers’social class on cardiovascular disease in middle-aged men. Lancet. 1996, 348:1259-1263.
[52] Davey Smith G, Hart C, Blane D, Hole D. Adverse socioeconomic conditions in childhood and cause-specific adult mortality:prospective observational study. BMJ. 1998, 316:1631-1635.
[53] Frankel S, Davey Smith G, Gunnell D. Childhood socioeconomic position and adult cardiovascular mortality:the Boyd Orr cohort. Am J Epidemiol. 1998,150:1081- 1084.
[54] Davey Smith G, McCarron P, Okasha M, McEwen J. Social circumstances in childhood and cardiovascular disease mortality:prospective observational study of Glasgow University students. J Epidemiol Community Health. 2001, 55:340-341.
[55] Heslop P, Davey Smith G, Macleod J, Hart C. The socioeconomic position of women, risk factors and mortality. Soc Sci Med. 2001, 53:477-485.
[56] Wamala SP, Lynch J, Kaplan GA. Women’s exposure to early and later-life socioeconomic disadvantage and coronary heart disease risk:the Stockholm Female Coronary Risk Study. Int J Epidemiol. 2001, 30:275-284.
[57] Claussen B, Davey Smith G, Thelle D. Impact of childhood and adulthood socioeconomic position on cause specific mortality:the Oslo Mortality Study. J Epidemiol Community Health. 2003, 57:40-45.
[58] Paffenbarger RS, Hyde RT, Wing AL, Lee I-M, Jung DL, Kampert JB. The association of changes in physical-activity level and other lifestyle characteristics with mortality among men. N Engl J Med. 1993,328:538-545.
[59] Stamler J, Stamler R, Neaton JD, et al. Low riskfactor profile and long-termcardiovascular and noncardiovascular mortality and life expectancy:findings for 5 large cohorts of young adult and middle-aged men and women. JAMA. 1999, 282:2012-2018.
[60] Yusuf S, Reddy S, Ounpuu S, Anand S. Global burden of cardiovascular diseases. Part II:variations in cardiovascular disease by specific ethnic groups and geographic regions and prevention strategies. Circulation. 2001, 104:2855-2864.
[61] Yusuf S, Hawken S, Ounpuu S, et al. Effect of potentially modifiable risk factors associated with myocardial infarction in 52 countries(the INTERHEART study):case-control study. Lancet. 2004, 364:937-952.
[62] Magnus P, Beaglehole R. The real contribution of the major risk factors to the coronary epidemics:time to end the“only-50%”myth. Arch Intern Med. 2001, 161:2657-2660.
[63] Greenland P, Knoll MD, Stamler J, et al. Major risk factors as antecedents of fatal and nonfatal coronary heart disease events. JAMA. 2003, 290:891-897.
[64] Lantz PM, House JS, Lepkowski JM, Williams DR, Mero RP, Chen J. Socioeconomic factors, health behaviors, and mortality:results from a nationally representativeprospective study of US adults. JAMA. 1998, 279:1703-1708.
[65] Mika Kivim?ki, Debbie A. Lawlor, George Davey Smith, et al. Socioeconomic Position, Co-Occurrence of Behavior-Related Risk Factors, and Coronary Heart Disease:the Finnish Public Sector Study[J]. American Journal of Public Health, May 2007, 97( 5):874-879.
[66] Debbie A. Lawlor, George Davey Smith, Shah Ebrahim. Association Between Childhood Socioeconomic Status and Coronary Heart Disease Risk Among Postmenopausal Women:Findings From the British Women’s Heart and Health Study[J]. American Journal of Public Health, August 2004, 94( 8):1386-1392.
[67] Marilyn Hravnak, Jeff Whittle, Mary E. Kelley et al. Symptom Expression in Coronary Heart Disease and Revascularization Recommendations for Black and White Patients[J]. American Journal of Public Health, September 2007,97(9):1701- 1708.
[68] Lee H, Bahler R, Park O, Kim C, Lee HY, Kim Y. Typical and atypical symptoms of myocardial infarction among Black, Whites and Koreans. Crit Care Nurs Clin N Am. 2001, 13:531-539.
[69] Teresa T Fung, Vasanti Malik, Kathryn M Rexrode. Sweetened beverage consump- tion and risk of coronary heart disease in women[J]. American Society for Nutrition,2009, 89:1037-1041.
[70] Brook RD, Brook JR, Urch B, Vincent R, Rajagopalan S, Silverman F. 2002. Inhalation of fine particulate air pollutionand ozone causes acute arterial vasocon- striction in healthy adults. Circulation. 2002, 105(13):1534-1536.
[71] Donaldson K, Tran L, Jimenez LA, Duffin R, Newby DE, Mills N, et al. 2005b. Combustion-derived nanoparticles:a review of their toxicology following inhalation exposure. Part Fibre Toxicol 2:10.
[72] National High Blood Pressure Education Program Working Group on High Blood Pressure in Children and Adolescents. The fourth report on the diagnosis, evaluation, and treatment of high blood pressure in children and adolescents. Pediatrics. 2004, 114:555-576.
[73] Chobanian AV, Bakris GL, Black HR, et al. Seventh report of the Joint National Committee on Prevention, Detection, Evaluation, and Treatment of High Blood Pressure. Hypertension. 2003, 42:1206-1252.
[74] Sorof JM, Lai D, Turner J, Poffenbarger T, Portman RJ. Overweight, ethnicity, and the prevalence of hypertension in school-aged children. Pediatrics. 2004,113:475- 482.
[75] Flynn JT, Mitsnefes M, Pierce C, Cole SR, Parekh RS, Furth SL, Warady BA. Blood pressure in children with chronic kidney disease:a report from the Chronic Kidney Disease in Children study. Hypertension. 2008, 52:631-637.
[76] van den Elzen AP, de Ridder MA, Grobbee DE, Hofman A, Witteman JC, Uiterwaal CS. Families and the natural history of blood pressure:a 27-year follow-up study. Am J Hypertens. 2004, 17:936-940.
[77] Chobanian AV, Bakris GL, Black HR, et al. Seventh report of the Joint National Committee on Prevention, Detection, Evaluation, and Treatment of High Blood Pressure[J]. Hypertension, 2003, 42:1206-1252.
[78] Qureshi AI, Suri MF, Kirmani J et al. Prevalence and trends of prehypertension and hypertension in United States:National Health and Nutrition Examination Surveys 1976 to 2000[J]. Med Sci Monit, 2005, 11:CR403-CR409.
[79] Burt VL, Whelton P, Roccella EJ, et al. . Prevalence of hypertension in the US adult population:results from the Third National Health and Nutrition Examination Survey, 1988-1991[J]. Hypertension. 1995, 25:305-313.
[80] Vasan RS, Beiser A, Seshadri S, et al. Residual lifetime risk for developing hypertension in middle-aged women and men:the Framingham Heart Study[J].JAMA. 2002, 287:1003-1010.
[81] Birkenhager WH, Staessen JA. Convergence of atherosclerosis and Alzheimer’s disease. Lancet. 2004, 363:2091.
[82] Kilander L, Nyman H, Boberg M, Hansson L, Lithell H. Hypertension is related to cognitive impairment:a 20-year follow-up of 999 men. Hypertension. 1998,31:780- 786.
[83] Launer LJ, Ross GW, Petrovitch H, Masaki K, Foley D, White LR, Havlik RJ. Midlife blood pressure and dementia:the Honolulu-Asia Aging Study. Neurobiol Aging. 2000, 21:49-55.
[84] Duron E, Hanon O. Hypertension, cognitive decline and dementia. Arch Cardiovasc Dis. 2008, 101:181-189.
[85] Singh-Manoux A, Marmot M. High blood pressure was associated with cognitive function in middle-age in the Whitehall II Study. J Clin Epidemiol. 2005,58:1308- 1315.
[86] Waldstein SR, Brown JR, Maier KJ, Katzel LI. Diagnosis of hypertension and high blood pressure levels negatively affect cognitive function in older adults. Ann Behav Med. 2005, 29:174-180.
[87] Elias MF, Elias PK, Sullivan LM, Wolf PA, D’Agostino RB. Lower cognitive function in the presence of obesity and hypertension:the Framingham Heart Study[J]. Int J Obes Relat Metab Disord. 2003, 27:260-268.
[88] Wolf PA, Beiser A, Elias MF, Au R, Vasan RS, Seshadri S. Relation of obesity to cognitive function:importance of central obesity and synergistic influence of concomitant hypertension-the Framingham Heart Study[J]. Curr Alzheimer Res. 2007, 4:111-116.
[89] Waldstein SR, Katzel LI. Interactive relations of central versus total obesity and blood pressure to cognitive function[J]. Int J Obes(Lond). 2006, 30:201-207.
[90] Bolger N, Zuckerman A. A framework for studying personality in the stress process[J]. J Pers Soc Psychol 1995, 69:890-902.
[91] Larsen RJ, Ketelaar T. Personality and susceptibility to positive and negative emotional states[J]. J Pers Soc Psychol 1991, 61:132-140.
[92] McCrae RR, Costa PT Jr. Adding liebe und arbeit:The full five-factor model and well-being[J]. Personality & Social Psychology Bulletin 1991, 17.
[93] Costa, PT, Jr;McCrae, RR. The revised neo personality inverntory(neo pi-r) professional manual[J]. Odessa, FL;Psychological Assessment Resources:1992.
[94] Watson D, Clark LA. On traits and temperament:General and specific factors of emotional experience and their relation to the five-factor mode[J] . J Pers 1992, 60:441-476.
[95] Izard CE, Libero DZ, Putnam P, Haynes OM. Stability of emotion experiences and their relations to traits of personality[J]. J Pers Soc Psychol 1993, 64:847-860.
[96] Bolger N, Schilling EA. Personality and the problems of everyday life:The role of neuroticism in exposure and reactivity to daily stressors[J]. J Pers 1991,59:355-386.
[97] Pickering, A. Gray, JA. The neuroscience of personality. In:Pervin, L;John, O, editors. Handbook of personality:Theory and research. New York:Gilford Press. 2001. 227-299.
[98] Canli T, Sivers H, Whitfield SL, et, al. Amygdala response to happy faces as a function of extraversion[J]. Science 2002, 296-2191.
[99] Canli T, Zhao Z, Desmond JE, Kang E, Gross J, Gabrieli JD. An fMRI study of personality influences on brain reactivity to emotional stimuli[J]. Behav Neurosci 2001, 115:33-42.
[100]Zald DH, Mattson DL, Pardo JV. Brain activity in ventromedial prefrontal cortex correlates with individual differences in negative affect[J]. Proceedings of the National Academy of Sciences of the United States of America 2002,99:2450-2454.
[101]Gusnard DA, Ollinger JM, Shulman GL, Cloninger CR, Price JL, Van Essen DC, Raichle ME. Persistence and brain circuitry[J]. Proceedings of the National Academy of Sciences of the United States of America 2003, 100:3479-3484.
[102]Schwartz CE, Wright CI, Shin LM, Kagan J, Rauch SL. Inhibited and uninhibited infants“grown up”:Adult amygdalar response to novelty. Science 2003,300:1952- 1953.
[103]Canli T, Amin Z, Haas B, Omura K, Constable RT. A double dissociation between mood states and personality traits in the anterior cingulate[J]. Behav Neurosci 2004, 118:897-904.
[104]Gray JR, Burgess GC, Schaefer A, Yarkoni T, Larsen RJ, Braver TS. Affective personality differences in neural processing efficiency confirmed using fMRI. Cogn Affect Behav Neurosci 2005, 5:182-190.
[105]Omura K, Todd Constable R, Canli T. Amygdala gray matter concentration is associated with extraversion and neuroticism[J]. Neuroreport 2005, 16:1905-1908.
[106]Rauch SL, Milad MR, Orr SP, Quinn BT, Fischl B, Pitman RK. Orbitofrontal thickness, retention of fear extinction, and extraversion[J]. Neuroreport 2005,16:1909-1912.
[107]Johnson DL, Wiebe JS, Gold SM, Andreasen NC, Hichwa RD, Watkins GL, Boles Ponto LL. Cerebral blood flow and personality:A positron emission tomography study[J]. Am J Psychiatry 1999, 156:252-257.
[108]Christopher, Wright, Eric Feczko, Bradford Dickerson, et, al. Neuroanatomical Correlates of Personality in the Elderly Neuroimage[J]. 2007, 35(1):263-272.
[109]Coccaro EJ, Seiver JL, Klar HM, et al. Serotonergic studies in patients with affective and personality disorders[J]. Archives of General Psychiatry 1989,46:587- 599.
[110]O'Keane VO, Moloney E, O'Neill H, O'Connor A, Smith R, Dian TG. Blunted prolactin responses to d-Fenfluramine in sociopathy:Evidence for subsensitivity of central serotonergic function[J]. British Journal of Psychiatry 1992, 160:643-646.
[111]Cleare A, Bond A. Does central serotonergic function correlate inversely with aggression:A study using D-fenfluramine in healthy subjects[J].Psychiatry Research 1997, 69:89-95.
[112]Cleare AJ, Bond AJ. The effect of tryptophan depletion and enhancement on subjective and behavioural aggression in normal male subjects[J]. Psychopharma- cology, 1995, 118:72-81.
[113]Coccaro EF. Impulsive aggression and central serotonergic system function in humans:an example of adimensional brain-behavior relationship[J]. Int Clin Psychopharmacol, 1992, 7:3-12.
[114]Marsh DM, Dougherty DM, Moeller FG, Swann AC, Spiga R. Laboratory- measured aggressive behavior of women:Acute tryptophan depletion and augmentation[J]. Neuropscyhopharmacology, 2002, 26:660-671.
[115]Beverly H. Brummett, Stephen H. Boyle, Cynthia M. Kuhn. Associations among central nervous system serotonergic function and neuroticism are moderated by gender[J]. Biol Psychol. 2008, 78(2):200-203.
[116]Nishizawa S, Benkelfat C, Young SN, Leyton M, Mzengeza S, de Montigny C, et al. Differences between males and females in rates of serotonin synthesis in human brain[J]. Proceedings of the National Academy of Science:USA 1997,94:4823- 4824.
[117]Amy C Iversen, Lauren van Staden1, Jamie Hacker Hughes, et, al. The prevalence of common mental disorders and PTSD in the UK military:using data from a clinical interview-based study[J]. BMC Psychiatry 2009, 9:68.
[118]Hoge CW, Lesikar SE, Guevara R, et, al. Mental disorders among U. S. military personnelin the 1990s:association with high levels of health care utilizationand early military attrition[J]. Am J Psychiatry 2002, 159(9):1576-1583.
[119]Rosenheck R, Leda C, Gallup P, et, al. Initial assessment data from a 43-site program for homeless chronic mentally ill veterans[J]. Hosp Community Psychiatry 1989, 40(9):937-942.
[120]Iversen A, Nikolaou V, Greenberg N, et, al. What happens to British veterans when they leave the armed forces[J]. Eur J Public Health 2005, 15(2):175-184.
[121]Hoge CW, Castro CA, Messer SC, McGurk D, Cotting DI, Koffman RL:Combat duty in Iraq and Afghanistan, mental health problems, and barriers to care[J]. N Engl J Med, 2004, 351(1):13-22.
[122]Dlugosz LJ, Hocter WJ, Kaiser KS, Knoke JD, Heller JM, Hamid NA, Reed RJ, Kendler KS, Gray GC:Risk factors for mental disorder hospitalization after the Persian Gulf War:US Armed Forces, June 1, 1991-September 30,1993.J Clin Epidemiol.1999, 52(12):1267-1278.
[123]Maud Lemoine, Aurélie Revaux, Claire Francoz, et, al. Albumin liver dialysis as pregnancy-saving procedure in cholestatic liver disease and intractable pruritus[J]. World J Gastroenterol, 2008, 14(42):6572-6574.
[124]Adam Reich, Jacek C. Szepietowski1 Mediators of Pruritus in Psoriasis[J]. Med- iators of Inflammation, 2007, 10:1-6.
[125]Seppo AM Saari, Kirsi H Juuti, Joanna H Paloj?rvi, et, al. Demodex gatoi- associated contagious pruritic dermatosis in cats-a report from six households in Finland[J]. Acta Veterinaria Scandinavica, 2009, 51:40.
[126]S M Bates, G Dilke-Wing. Determination of iron status in women attending genitourinary clinics with pruritus vulvae[J]. Sex Transm Infect, 2006, 82:489-490.
[127]Maryam Akhyani1, Mohammad-Reza Ganji2, Nasrin Samadi, et, al. Pruritus in hemodialysis patients[J]. BMC Dermatology, 2005, 5:7.
[128]Yonova D. Pruritus in certain internal diseases[J]. HIPPOKRATIA 2007,11(2): 67-71.
[129]Greaves MW, Davies MG. The current status of histamine receptors in human skin:indirect evidence[J]. Br. J Dermatol 1982, 23(suppl):101-105.
[130]Stephen E. Waisglass, Avery Gillick. Pruritus possibly associated with a porto -systemic shunt in a bichon frise puppy[J]. Can Vet J 2006, 47:1109-1111.
[131]TA Salami, SO Samue, KC Eze, et, al. Prevalence and characteristics of aquagenicpruritus in a young African population[J]. BMC Dermatology, 2009, 9:4.
[132]Zouboulis CC. Acne:current aspects on pathology and treatment[J]. Dermatological Experience,1999, 4:6-37.
[133]Chu TC. Acne and other facial eruptions. Medicine, 1997, 25:30-3.
[134]Witkowski JA, Parish LC. The assessment of acne:an evaluation of grading and lesion counting in the measurement of acne[J]. Clin Dermato, 2004, 22:394-7.
[135]Bataille V, Snieder H, MacGregor AJ, et, al. The influence of genetics and environmental factors in the pathogenesis of acne:a twin study of acne in women[J]. J Invest Dermatol,2002, 119:1317-22.
[136]鞠强,石继海,夏隆庆.白介素-1与痤疮.国外医学·皮肤性病学分册. 2001, 27:24-26.
[137]Homemaunn S, SeltmarnH, Kodelia V, et al. Interleukin and protein are expressed in eultured human sehacyes at seady-teate and their leves are harely influneed by lipoolysanchandes. J Invest Der-matol,1997;108:382.
[138]Zalluga E. Skin reactions to antigens of Propionihacteriom acnes in patientswith acne vulgaris treatedwith aumvaccine. J British Journal of Dermatology,2001, 43:389-397.
[139]Valacckj G, Rimbach G, Salou C, et al. Effect of henxoyl perox-side on antioxidant status, NF-kappaB activity and interleukin-lalpa gene espresson in human keratno -cutes. J Tosicology 2001, 165:225-234.
[140]Yaneoka J, Kume T, Akinori A, et al. Suppressive effect of zine ion on iNOS expression induced by interferongamma or tumor necro-sis factor-slpha in murine keratnocytes. J Dermatological Sciemce 2000, 20:27-35.
[141]高宇.心系证候规范性研究[D]:[硕士学位论文].江苏南京:南京中医药大学, 2006. 11-12.
[142]耿建领,黄涛.张子和心系疾病学术经验略要[J].光明中医, 2003,18(109): 47-48.
[143]丁书文,李晓,李运伦.热毒学说在心系疾病中的构建与应用[J].山东中医药大学学报, 2004, 28(6):413-416.
[144]董玉山.781例心系疾病证治特点的研究[D]. [硕士学位论文].山东:山东中医药大学, 2001.
[145]戴天芳,柳芳.《金匮要略》治心七法[J].中医研究, 2005, 18(2):7-8.
[146]李素清.《伤寒论》“悸”的辨治规律探讨[J].中医药研究, 1998, 14(4):1-2.
[147]顾宁,陈红景.《伤寒论》心悸证治浅析[J].中医药研究, 2000, 16(1):3-4.
[148]陈国英.《伤寒论》中温法在心系疾病中的运用考释[J].中医药学刊, 2004,22(5):904-905.
[149]谈文峰,丁倩.柴胡疏肝散治疗心系疾病验案举隅[J].江苏中医,1998,19(10): 33.
[150]白雪,王明杰.从玄府论治心系疾病的经验浅析[J].首都医药, 2005,11(17): 41-42.
[151]李必旭.林钟香治疗心系疾病验案三则[J].辽宁中医杂志,2007,34(12): 1796-1797.
[152]丁成华,曹晓瑞,马丽珍.略论心系疾病从脾胃论治[J].江西中医学院学报, 2007, 19(5):17-18.
[153]刘明武.伤寒论对心系疾病的辨治规律探析[J].辽宁中医, 1997, 24(3):107-108.
[154]胡爱萍.试论《伤寒论》中心系疾病的用药原则与特点[J].甘肃中医, 2000,3:2-3.
[155]苏明.栗锦迁教授经验介绍--心系疾病治疗中化痰法的应用[J].天津中医药, 2006, 23(2):103-104.
[156]梁凤信,郭茂松,孙元莹.郭文勤教授治疗心系疾病学术思想[J].黑龙江中医药, 2001, 6:2-3.
[157]蔚永运,郭艳.从心系疾病论心与表的关系[J].山东中医杂志1999,18(10): 435-436.
[158]范磊,张成博,杨海燕.“心部于表”的理论认识及其临床意义[J].山东中医杂志, 2005, 29(4):261-262.
[159]刘莹,李平.论李永成从脾胃从神论治皮肤病[J].辽宁中医, 2008,35(5): 772-773.
[160]刘大文.面部痤疮从心论治[J].北京中医药, 2008, 27(1):20-21.
[161]谷玉琴,孙月吉.心理因素与痤疮[J].中华皮肤科杂志2006, 39(2):119-120.
[162]李旺,张淑萍.刘完素燥证论治探析[J].浙江中医杂志, 2001, (5):209.
[163]孟繁洁.刘完素燥论阐微[J].中国医药学报, 2004, 19(4):200.
[164]王爱民.吴鞠通论下焦温病阴伤证治浅析[J].江苏中医, 1999, 20(12):6.
[165]马伯艳,李冀.《温病条辨》保阴护津思想探析[J]. ACMP, 2003, 31(1):56.
[166]张晓光.吴鞠通养阴保津学术思想探要[J].实用中医内科杂志, 2003, 17(4):261.
[167]马爱莲.治疗温病当注重养阴[J].现代中西医结合杂志, 2005, 14(13):1698.
[168]周铭心,王树芬.中医时间医学[M].长沙:湖南科学技术出版社, 1989:76
[169]蔡能,史鸿璋,陈钟舜,等.心身病[M].上海:上海科技文献出版社, 1985, 88.
[170]Schafer T, Nienhaus A, Vieluf D, et al. Epidemiology of acne in the general population:the risk of smoking[J]. Br J Dermato, 2001, 145:100-104.
[171]刘叶兰.中医对寻常痤疮病因病机的认识[J].河南中医, 2006, (26)4:6-8.
[172]毛文姣.痤疮病因概述[J].辽宁中医药大学学报, 2008, 10(12):70-71.
[173]张爱清,李克华,王甘棠,等.痤疮患者个性特征及情绪反应的研究[J].中国皮肤性病学杂志, 1997, 13(1):17.
[174]钱江,杨柳,兰海梅,等.南方地区寻常痤疮中医临床流行病学调查分析[J].医学信息, 2006, 19(10):1807-1809.
[175]陈慧.痤疮的流行病调查及中医分型和积雪甙对造模大鼠炎症结节作用机理探讨[D].湖北:湖北中医学院, 2006,硕士学位论文.
[176]何弘.乌鲁木齐市女性青春期后痤疮病因初探[J].中国麻风皮肤病杂志, 2007, 23(10):921.
[177]李立明.流行病学[M]. 4版.北京:人民卫生出版社, 2000:237.
[178]EzzatiM, LopeZ AD, Rodgers A, et a. l Selected major risk factor and global and regional burden of disease[J]. Lancet, 2002, 360:1347-1360.
[179]程祖亨,汪师贞.新疆维吾尔族自治区不同民族高血压流行病学因素的调查研究[J].新疆医学院学报, 1982, 5(2):103-111.
[180]Hiroshi Kawamura, Medet. J, Hiromim. 24-hour, blood pressure in Uygur, Kazakh and han elderly subjects in China[J]. Hypertens Res. 2000, 23(2):177-185.
[181]李江,王建平.新疆南疆农村居民饮水现状与相关地方病问题[J].地方病通报, 1997, 1.
[182]王连方,郑宝山,王生玲.新疆水砷及其对开发建设的影响[J].地方病通报, 2002, 17(1).
[183]寇风荣,摆宏萍,居尼斯别克,景兆兰,阮华.米泉市农村饮用水水氟水砷含量的检测[J].现代预防医学, 2007, 14.
[184]史红,周铭心.便秘的病因研究.新疆中医药, 2007;25(3):107-111.
[185]丁建中,龚权,朱惠玲,等.外燥对小鼠呼吸道分泌功能与抗体的影响[J].长江大学学报(自然版). 2005, 2(12):341-342, 351.
[186]丁建中,张六通,陈刚,等.外燥对小鼠气管?肺与皮肤影响的病理学研究[J].时珍国医国药, 2007, 18(3):527-528.
[187]陈刚,丁建中,张六通,等.细菌攻击对外燥小鼠气道与肠道分泌及免疫功能的影响[J].湖北中医杂志, 2007, 29(2):3-5.
[188]丁建中,龚权,陈刚,等.外燥对小鼠气管细胞凋亡与bcl-2基因及bax基因表达的影响[J].时珍国医国药, 2007, 18(1):15-16.
[189]李永军,赵刚,司小莉,等.戈壁气候对新兵肺通气功能的影响[J].总装备部医学学报, 2008, 10(4):195-196.
[190]刘浩,周铭心.西北燥证(新疆)与常年性变应性鼻炎?慢性支气管炎的关系研究.新疆医科大学2005级硕士论文.中国优秀硕士学位论文全文数据库
[191]杨正志,杨利华.气象因素对老年高血压患者血压的影响[J].世界中西医结合杂志, 2009, 4(6):418-419.
[192]王玲,白原,刘小云,等.高血压与气象因素的关系[J].医学综述, 2007,13(3): 239-240.
[193]黄如训,肖小华,李玲,等.气象因素促发卒中的实验研究[J].中华老年医学杂志, 2001, 20(5):366-368.
[194]董蕙青,郭琳芳.脑卒中发病与气象要素变化关系分析[J].广西气象, 2006,20 (2):40-43.
[195]董蕙青,李雄,郭琳芳,等.南宁市脑卒中疾病死亡与气象要素的关系[J].广西科学院学报, 2005, 21(2):127-130.
[196]王健,张新英,窦月华.乌鲁木齐市高血压病的气象诱因和指数预报初探[J].华西医学, 2009, 12:289-295.
[197]陈凤辉,程祖亨,玛依拉·吾甫尔,等.新疆“塔克拉玛干沙漠人”高血压与摄盐量摄钠量相关性研究[J].新疆医科大学学报, 2009. 32(7):882-884.
[198]Momityama Y, Hirano R, Taniguchi H, et al. Effects of interleukin-1gene polymorphisms on the development of coronary artery disease associated with Chlamydia Pneumoniae infection[J]. J Am Coll Cardiol, 2001, 38(3):712-7.
[199]Francis SE, Camp NJ, Dewberry RM, et al. Interleukin-1 receptor antagonist gene polymorphism and coronary artery disease[J]. Circulation, 1999, 99(7):861-6.
[200]Miranda-Malpica E, Martinez-Rios MA, Fragoso JM, et al. The interleukin 1B-511 polymorphism is associated with the risk of developing restenosis after coronary stenting in Mexican patients[J]. Hum Immunol, 2008, 69(2):116-21.
[201]Duff GW. Evidence for genetic variation as a factor in maintaining health[J]. Am J Clin Nutr, 2006, 83(suppl):431S-5S.
[202]Soylu O, Yildirim A, Coker A, et al. Interleukin-1B(-511)gene polymorphism is associated with acute coronary syndrome in the Turkish population[J]. Eur Cytokine Netw, 2008, 19(1):42-8.
[203]Latkovskis G, Licis N, Kalnins U. C-reactive protein levels and common polymorphisms of the interleukin-1 gene cluster and interleukin-6 gene in patients with coronary heart disease[J]. Eur J Immunogenet, 2004, 31(5):207-13.
[204]骆秦,李南方,周玲.β2-肾上腺素能受体基因Arg16/G ly多态性与新疆哈萨克族高血压的关系[J].科学技术与工程, 2010, 10(28):6859-6966.
[205]杨忠伟,冯秀丽,吕继荣.新疆不同民族冠心病患者eNOS基因894G/T多态性研究[J].新医学, 2010, 41(1):21-23.
[206]王慧,刘奇迹,黎莉. LRPS/6基因多态与中国汉族人群冠心病的关联分析[J].国际遗传学杂志, 2009, 32(4):12-14.
[207]林雪,尚玉红,马丽.环境地理因素与更年期高血压的关系探微—西北燥证背景研究[J].中华中医药杂志2008, 23(2):125-127.
[208]王素娥.神经衰弱的中医辨证分型治疗[J].中国乡村医药杂志, 2003, 10(6):17.
[209]李慧杰,李德新.郁证之症因及证治规律探析[J].辽宁中医药大学学报, 2007, 9(3):24-25.
[210]徐海燕.神经衰弱的中医辨证施治体会[J].中西医结合与祖国医学, 2005, 9(9):836-837.
[211]高华,沙塔娜提·穆罕默德,周铭心.西北燥证与心理素质关系探讨[J].新疆医科大学学报, 2007, 30(1):17-19.
[212]潘祥龙,谢珉宁,丁佳频.润燥止痒胶囊治疗皮肤瘙痒症的临床研究[J].上海中医药杂志, 2004, 38(8):33-34.
[213]马宽玉,朱璐.润肤止痒汤治疗老年性皮肤瘙痒症30例临床观察[J].四川中医, 2005, 25(12):95-96.
[214]丁仁明,李谦,刘复兴.辨证分型治疗单纯性皮肤瘙痒症62例疗效观察[J].云南中医中药杂志, 2007, 28(4):12-13.
[215]沈乐.痤疮论治杂谈[J].光明中医, 2006, 21(4):14.
[216]李东海,肖红利,林少健,等.从肺热脾虚论治寻常性痤疮64例[J].广州中医药大学学报, 2006, 23(1):32-34.
[217]孟军,王蕾,徐信杰,等.辨病辨证相结合治疗痤疮临床观察[J].中国中西医结合皮肤性病学杂志, 2008, 7(2):93-94.
[218]许嗣立,严石林.小议痤疮从肝阳虚论治[J].四川中医, 2007, 25(6):27-28.
[219]周喜芬.浅析痤疮从肝论治之机理[J].内蒙古中医药, 2008, 1:5-6.
[220]余晓琳,陈军平,林晨.从肝郁肾虚论治寻常性痤疮48例[J].辽宁中医药大学学报, 2008, 10(6):93-94.
[221]张秀美,王秀莲.痤疮从湿热论治探讨[J].天津中医药, 2007, 24(6):521-522.
[222]田维军,魏桂芝.中医虚证全书[M].江西科学技术出版社, 1997, 12.
[223]孙广仁,中医基础理论,普通高等教育”十五”国家级规划教材.北京:中国中医药出版社, 2002, 8:209.
[224]匡调元.中医病理研究[M].上海:上海科学技术出版社, 1980:66.
[225]王琦.中医体质学[M].北京:人民卫生出版社, 2005. 8.
[226]王琦,盛增秀.中医体质学说M.南京:江苏科学技术出版社, 1982:2.
[227]母国成.中医体质学说及其异化[J].新中医, 1983, 15(9):9.
[228]田代华.论体质与证候[J].山东中医学院学报, 1983, 7(1):6.
[229]朱庭仪.中医体质分型100例分析[J].成都中医药大学学报, 1996, 19(4):48.
[230]罗慰慈.鉴别咳嗽症状时应重视环境因素临床肺科杂志[J]. 2009, 14(3):285.
[231]王燕,周铭心.新疆各地不同民族阴阳虚证流行病学调查分析[J]. 2008. 49(4):345-348.
[232]王琦,朱燕波.中国一般人群中医体质流行病学调查—基于全国9省市21948例流行病学调查数据[J].中华中医药杂志, 2009, 24(1):7-12.
[233]吴承玉,骆文斌,王娜娜等.江苏地区2043例中医体质流行病学调查研究[J].中国中医基础医学杂志, 2009, 15(2):138-140.
[234]李杰.中医体质分类的流行病学调查及阳虚体质的相关研究[].江苏:南京中医药大学,博士学位论文, 2008.
[235]周晓平,杨进.中医气象学现代研究方法探讨[J].中国中医药信息杂志, 2007, 14(2):6.
[236]张学忠,董得平.中国青少儿生长发育不同年龄阶段与地理气象因素关系的灰色关联分析[J].中国体育科技, 1996, 32(4):62.
[237]蔡民坤.心气虚证与气虚证的相互关系浅析[J].实用中医内科杂志, 2008, 22(5):27.
[238]李燕.王昕.心气虚证与心功能指标相关性的meta分析[J].中外医疗, 2009, 12:174-175.
[239]王阶,何庆勇,姚魁武. 1069例冠心病脏腑虚证的多中心前瞻性研究[J].中华中医药杂志, 2009, 1:208-213.
[240]宋红普,何裕民. 476例原发性高血压患者体质特点研究[J].上海中医药大学学报, 2001, 15(2):33-35.
[241]林谦,陈焱木,金法. 371例原发性高血压患者的中医体质辨证研究[J].现代中医药, 2004, 3:17-18.
[242]姚晓天,胡伟,程志清.高血压影响因素与中医体质类型相关性研究[J].中医药学刊, 2004, 22(12):2314-2316.
[243]吴宏东,王琦,董静.高血压病体质因素的多元Logistic回归分析[J].北京中医药大学学报(中医临床版)2009, 16(5):12-13.
[244]魏源.神经衰弱患者五大价格特质研究[J].中国临床康复, 2004, 8(30):6585-6587.
[245]钱铭怡.心理咨询与心理治疗[M].北京:北京大学出版社, 1984:232-54.
[246]冯栋年,刘燕玲,张新,等.阳虚证患者遗传学特征探讨[J].中西医结合杂志, 1989, 9(1):24.
[247]国维,范瑞强,尹玉贞,等.中药消痤灵治疗痤疮的多中心随机对照研究[J].广州中医药大学学报, 1995, 12(3):6-8.
[248]成改霞.寻常痤疮五脏辨治规律及五脏分级相关性研究[D].福建:福建中医学院硕士学位论文, 2005.
[249]郑雨佳.座疮200例中医体质流行病学调查[D].四川:成都中医药大学,硕士学位论文, 2008.
[1]吴辉,于扬文,吴伟等. 116例冠心病患者中医证候及病因分析[J].江苏中医, 2004, 25(10):30.
[2]李芳,樊相军.胸痹诱发因素与辨证分型的调查分析[J].人民军医, 2001, 4(8):484-486
[3]李军,王杰.病证结合的冠心病心绞痛病因病机探讨[J].中国中医基础医学杂志, 2003, 13(7):531-533
[4]林谦.冠心病(胸痹心痛)的病因病机及辨证论治[J].中国临床医生, 2000,28(10): 11-14
[5]杨维益.心绞痛诊治一得.浙江中医杂志, 1981, (9):386.
[6]阁洪臣.略谈胸痹心痛的病理与证治.陕西新医药, 1979, (6):49.
[7]黄河清.《伤寒论》少阴寒化证与冠心病心肌缺血相关性探讨.中医杂志, 1999, 40(10):585.
[8]黄宗鉴.益气活血法治疗冠心病心绞痛临床疗效观察.福建中医药, 1988,(l):27.
[9]张伯臾,蓝建华.中医内科学.北京:人民卫生出版社, 1990:215.
[10]王秀珍等.论心生痰与心痛.黑龙江中医药, 1985, (4):11.
[11]林功铮.曹锡寅论治心绞痛.辽宁中医杂志, 1983, (6):5.
[12]田养年.从痰瘀论治冠心病.辽宁中医杂志, 1983, (4):25.
[13]胡冬裴.胸痹古今证型归类研究团.上海中医药大学学报, 2004, 18(4):8-9
[14]邱少波.浅谈胸痹的病因病机.国医论坛. 2007, 22(l):13-14
[15]李秀峰,张瑜珠.高血压病的病因病机?辨证论治及体会.中国医药指南, 2009, 7(4):109-110
[16]黄力,史载祥.高血压病的病因病机和辨证论治.中国临床医生, 1999, 27(12):10-11
[17]古炽明,丁有钦.高血压病证候文献分析述评[J].中医药学刊, 2003, 21(7):1156.
[18]赵奕,方文岩,赵治等.高血压病中医辨证分型与基底动脉血流动力学探讨[J].天津中医药, 2005, 22(4):297.
[19]冯向阳.辨证分型治疗高血压病112例临床观察[J].中医药导报, 2006, 12(8):34.
[20]白春锦,周濠,王丽等.不同中医证型高血压患者的心血管危险因素分层?高血压分期?分级及血脂等指标变化特征[J].中医康复研究, 2005, 9(23):145.
[21]柳静,张旭生.高血压病从气血论治[J].吉林中医药, 2007, 27(1):14.
[22]董德保.清脑熄风汤治疗高血压病40例小结[J].光明中医, 2004, 19(6):52.
[23]张喜奎.陈亦人教授医话(连载)[J].国医论坛, 2000, 15(3):14.
[24]吴华堂,范金茄.风眩(高血压病)病因病机证治探析[J].中医药学刊, 2003, 21(3):467-468
[25]李玉兰.从中医学角度探讨高脂血症的病因病机[J].四川中医, 2010, 28(3):17.
[26]郭技,郭宗英.降脂饮治疗高脂血症的临床观察.辽宁中医杂志, 1996(2):69.
[27]张秉志.中药泡饮治疗高脂血症.江苏中医, 1995, 16(2):24.
[28]李长生,郭凤仗,盛子敬. 49例高脂血症的临床观察,实用中西医结合杂志, 1996, 9(2):98.
[29]熊文生,李任先,刘国普等.辨治高脂血症重在理脾化痰.新中医, 1996(11):4.
[30]刘剑,王献民,周心芳等.自制神农双降丸治疗高脂血症临床观察.中医药研究, 1996, 9(1):26-28
[31]曾昭龙,李小敏,尹炳生等.正脂丸对血脂影响的研究.辽宁中医杂志, 1999, 26(5):234-235
[32]屠光英,周晓军. 44例高脂血症的临床观察.上海中医药杂志, 1991(9):8-9
[33]董玉宽.健脾为主治疗老年性高指血症27例.辽宁中医杂志, 1994,21(12): 545-546
[34]王静宇,陈培玉,张立营等.山明乌参三黄二泽汤治疗高脂血症80例临床观察.四川中医, 1994(12):24-25
[35]吴少萍.“降脂汤”治疗高脂血症40例观察.上海中医, 1998(9):21.
[36]胡正松.高脂血症从痰淤论治五法.江苏中医, 1995, 16(12):32-33
[37]张三林,方亚伦,张正臣等.黄炭降脂片治疗高脂血症92例临床分析.实用中西医结合杂志, 1995, 8(4):249.
[38]崔衍富.血塞通结合辨证治疗高脂血症100例.辽宁中医杂志, 1997, 24(8):403
[39]张子明,吴振萍,侯健民等.降脂口服液治疗高脂血症120例.实用中西医结合杂志, 1993, 6(5):279-280
[40]巫奕丽,董卫.降脂饮治疗高脂血症53例临床观察.中医药研究, 1998, 14(3):15-16
[41]周海虹.谈高脂血症的中医证治.实用中西医结合杂志, 1995, 8(7):418.
[42]刘杏鑫,清脂饮治疗高脂血症88例临床分析.实用中西医结合杂志, 1997, 10(7):630.
[43]陈川.补肾、化痰和活血对血脂代谢影响的比较.上海中医药杂志, 1997(2):22-25
[44]郑清莲,薛东波.刘茂甫教授对高脂血症病因病机的认识及治疗经验.中医药研究, 1993(6):17-18
[45]杨大男,杨代勇.张惠琴逍遥丸治疗高脂血症84例?陕西中医, 1995, 16(3):109-110
[46]张绮桂.降脂汤治疗高脂血症30例观察.中医药研究, 1992(6):33-34
[47]解荣.“三子汤”治疗高脂血症30例观察.上海中医药杂志, 1997(10):28.
[48]杨相彬.降脂汤治疗高胆固醉血症临床观察.实用中西医结合杂志, 1995, 8(6):377.
[49]刘少云.外用消脂贴治疗高脂血症60例疗效分析.天津中医, 1999, 16(8):15-16
[50]刘国凤,周静.降脂化淤胶囊治疗高脂血症20例.河北中医, 1999, 21(2):90-91
[51]胡晓灵,李以义,曾斌芳等.复方降脂散治疗老年高脂血症临床观察.中医药研究, 1997, 13(3):11-13
[52]俞兴祥,张受韵.自拟降脂通脉汤治疗高脂血症36例.上海中医药杂志, 1995(4):23-24
[53]杨文瑞,沈王安.降脂叶袋泡茶治疗高脂血症35例报道.上海中医药杂志, 1993(12):29.
[54]朱丽华.自拟降脂汤治疗高脂血症41例临床分析.中医药研究, 1998, 14(3):15.
[55]姚克裘,周曾绮,陈敏先.血通胶囊对老年高脂血症的影响临床研究.上海中医药杂志, 1995(l):37-38
[56]拓步雄,李慧,车玉英.舒肝胶囊对实验性高脂血症及脂肪肝的影响.中医药研究, 1998, 14(5):41-43
[57]李金德,李峰.中西医结合治疗原发性高脂血症62例临床观察.北京中医, 1997(5):17-18
[58]张春荣,王平.中医辨证联合静点丹参?刺五加治疗高脂血症.实用中西医结合杂志, 1996, 9(7):440.
[59]高尚社.中风后郁证的辨证论治探析[J].中国中医基础医学杂志, 2005, 11(8):616.
[60]李慧杰,李德新.郁证之症因及证治规律探析[J].辽宁中医药大学学报, 2007, 9(3):24-25
[61]王威.不寐的证治探识[J].实用中医内科杂志, 2006, 20(1):39-40
[62]吕慰秋.顽固性失眠的治疗心法[J].山东中医杂志, 1999, 18(3):105-106
[63]徐凌云.女性失眠探讨[J].中国中医基础医学杂志, 2004, 10(1):75.
[64]方少琼.小儿失眠的病因病机的探讨[J].中国中医基础医学杂志, 1997, (3):23.
[65]武永刚.《伤寒杂病论》治疗失眠证七法辨析[J].中医药学报, 2004, 32(2):5-6
[66]安妮,马智.马智教授治疗不寐经验撷菁[J].时珍国医国药, 2008, 19(5):1288-1289
[67]夏隆庆,赵春霞.瘙痒的发生机制、分型和治疗[J].临床皮肤科杂志, 2003, 32(11):687-689
[68]沈柱,刘玉峰.瘙痒发生与治疗的研究进展[J].中华皮肤科杂志, 2003, 36(7):415-417
[69]尹冀源,秦晓娟,柴立等.瘙痒症患者血浆中阿片样肽的检测[J].临床皮肤科杂志, 2000, 29(6):340-341
[70]徐宜厚,王保方,张赛英.皮肤病中医诊疗学[M].北京:人民卫生出版社, 1997, 14-15
[71]陆德铭.中医外科学汇[M].上海:上海科学技术出版社, 1997, 18:120.
[72]张坤.皮肤痛痒症辨证论治浅析[J].新中医, 2004, 36(7):60.
[73]赵炳南.赵炳南临床经验集[M].第一版.北京:人民卫生出版社, 1974.
[74]刘艳平,刘艳杰.七味地黄益母汤治疗老年皮肤瘙痒症[J].湖北中医杂志, 2002, 24(8):44.
[75]谢贻志.克痒汤治疗皮肤瘙痒症30例[J].实用中医药杂志, 2002, 18(9):32.
[76]贺芳,蔡爱萍.中药治疗皮肤瘙痒58例[J].现代中西医结合杂志, 2000, 9(19):1939-1940
[77]陈达灿,国维.皮肤性病科专病中医临床诊治[M].北京:人民卫生出版社, 2002:281.
[78]江超,姜华静,柯黎黎.仙方活命饮治疗痤疮80例[J].山东中医杂志, 2001, 20(8):475.
[79]艾军.从温病辨治痤疮的体会[J].广西中医药, 1998, 21(4):42.
[80]贾喜花,唐汉钧,林立真.唐汉钧辨证分型治疗痤疮的经验[J].上海中医药杂志, 2002, (6):31-32
[81]徐学武.增液承气汤加味治疗寻常痤疮110例[J].湖北中医杂志, 1991, 13(1):18-19
[82]阎德文.浅谈痤疮的发病、辨证与分型诊断[J].深圳中西医结合杂志, 2000, 10(2):70-71
[83]金起风,周德英.中医皮肤病学[M].北京:中国医药科技出版社, 2000, 376.
[84]李艳萍,张友堂.痤疮病因病机探讨[J].中国中医基础医学杂志, 2004, 10(11):60.
[85]李明亮,杨柳,黄文婧等.基于杨柳教授4种证型分类的痤疮病因病机探讨[J].时珍国医国药, 2010, 21(2):456-457
[86]钱江,杨柳,兰海梅等.南方地区寻常痤疮中医临床流行病学调查分析[J].临床医学, 2006, 19(10):1807.
[87]王作顺.马连珍老中医治疗冠心病经验[J].中国中医急症, 2003, 12(2):145.
[88]李洁,解品启.从瘀论治冠心病心绞痛[J].中国中医急症, 2007, 16(2):179-180
[89]许从真.冠心病心绞痛辨证论治[J].浙江中西医结合杂志, 2003, 13(1):59.
[90]于厚波.冠心病心肌梗死的中医辨证施治[J].黑龙江医学, 2005, 29(6):479.
[91]黄永生.冠心病心绞痛临床诊治体会[J].长春中医学院学报, 2005, 21(1):16-17
[92]王文革,姚红芳.冠心病心绞痛的中医辨证分析[J].中西医结合心血管杂志, 2004, 2(12):730.
[93]袁建平,朱玉兰,周会丽.中医辨证加西药治疗冠心病心绞痛疗效观察[J].吉林中医, 2005, 25(7):47-48
[94]李幼勋,王德华.胸痹的病因病机及其证治述略[J].实用中医内科杂志, 2002, 16(4):184-185
[95]张敏州,邹旭,李新梅等.脚屏心痛证冠状动脉造影100例临床分析[J]浙江中西医结合杂志, 2001, 11(8):472-473
[96]徐瑛.中医辨证治疗冠心病60例[J].辽宁中医杂志, 2002, 29(7)409-410
[97]梁秀香.中药治疗胸痹心痛126例临床观察.北京中医, 1997, (3):23-25
[98]宋秀明.王左治疗冠心病的经脸.湖北中医杂志, 1999, 21(5):197.
[99]康路景.辨证论治冠心病心绞痛198例分析.内蒙古中医药, 1996, 15(2):5-7
[l00]高荣林等.路志正调理脾胃法治疗胸痹300例临床观察报告.中医杂志, 1996, 37(10):606.
[101]旷惠挑.冠心病心绞痛临床证型分类探讨.中医杂志, 1997, 38(12):782.
[102]国家中医药管理局.中药新药临床研究指导原则(试行)[M].北京:中国医药科技出版社, 2002:73-77
[103]秦建国,王亚红,粱晋普等.郭维琴治疗高血压病经验[J].中医杂志, 2007, 48(7):586-587
[104]顾月珍,刘旭玲.高血压病的辨证治疗[J].黑龙江中医药, 2007(2):27
[105]王丽艳.高血压病辨证论治探要[J].中医函授通讯, 1999, 18(5):32
[106]魏霞,穆广梅.高血压辨证体会[J].中国医药导报, 2004, 19(1):59
[107]吕伍文.中药治疗高血压病84例临床观察[J].中医药学报, 2004, 132(13):38
[108]张荣珍,陈志祥,胡素颖等. 3 578例高血压中医证型构成临床调查研究[J].中医药临床杂志, 2007, 19(4):360-364
[109]罗玉环,梁昌年.中医辨证分型治疗高血压病72例[J].安徽医药, 2008, 12(3):242-244
[110]王严冬,刘志龙,杨华伟.珠海地区300名高血压患者中医证型与不同类降压药效果分析[J].甘肃中医, 2008, 21(9):54-55
[111]武煦峰.辨证治疗高血压病120例[J].实用中西医结合临床, 2008, 8(2):26-26
[112]谭洁,滕久祥,彭芝配.湖南省家族性高血压病中医证型分布的流行病学调查研究[J].湖南中医药大学学报, 2009, 29(1):70-71
[113]刘福明,陈晓虎,杜午奇等.高血压病中医证型分布规律的临床研究[J].江苏中医药, 2009, 41(10):33-34
[114]吴世才.中医治疗原发性高血压137例的体会[J].河南学院学报, 2009, 15(11):1433-1434
[115]杨秀飞.百合安神治疗神经衰弱30例[J].实用中医内科杂志, 2002, 16(1):28.
[116]林秋兰.中医辨证施治职业因素引起的神经衰弱[J].职业与健康, 2001, 17(11):29.
[117]张赐安.中西医结合治疗神经衰弱70例[J].新中医, 2002, 34(7):61.
[118]李淑萍.针刺兴奋疗法治疗神经衰弱120例[J].中医药学刊, 2001, 18(1):81.
[119]周彬,巨春青.健脑宁神治疗神经衰弱68例[J].湖北中医杂志, 2002, 24(7):31.
[120]李彬,李岩,魏久贞.辨证分型治疗神经衰弱60例[J].山东中医杂志, 2002, 21(3):151.
[121]曹文胜.针药并用辨证治疗神经衰弱96例[J].针灸临床杂志, 2000, 16(11):5.
[122]孙明军,李海军.天麻钩藤饮加减治疗神经衰弱500例[J].河南中医药学刊, 1998, 13(4):52.
[123]梁煜,李逊,曾令芬.辨证治疗神经衰弱30例[J].实用中医药杂志, 1998, 14(11):12.
[124]吕品雅.神经衰弱的证与治[J].南京医学情报, 1998, 18(3):20.
[125]孙秀,于春雷,战惠娟等. 100例神经衰弱综合征的针刺及调护的探讨[J].中医药信息, 1998, 2:53.
[126]冯占元.神经衰弱中医诊断与疗效标准的探讨[J].山东中医杂志, 1997, 16(10):435.
[127]邓禄清.神经衰弱从肾治[J].广东医学, 1992, 13(3):138.
[128]朱仁康.朱仁康临床经验集--皮肤外科[M].北京:人民卫生出版社. 2005:144-150
[129]孙凤琴.许连霈教授辨证分型治疗皮肤瘙痒症经验[J].北京中医药大学学报, 2003, 10(1):31-32
[130]金强.中药为主治疗痤疮75例[J].浙江中医杂志, 1995, 30(9):391-391
[131]李元亚,刘瓦利.中药内服外用治疗寻常痤疮58例疗效分析[J].中级医刊, 1996, 31(11):54-55
[132]吴克永,张新民,沈自尹.辨证分型治疗寻常性痤疮234例[J].上海中医药杂志, 1998, 11(5):28-29
[133]张虹亚,刘峥,葛明等.辨证论治痤疮69例[J].安徽中医学院学报, 1997, 16(4):29-30
[134]赵志新.辨证治疗痤疮190例[J].河北中医, 2000, 22(4):172.
[135]孟涛.辨证治疗痤疮120例[J].陕西中医, 2001, 22(4):212.
[136]杨健芳. 148例痤疮中医辨证分型与临床疗效观察[J].湖南医科大学学报, 2001, 26(3):219.
[137]王上游.清肺法治疗青春期痤疮205例[J].浙江中医杂志, 1994, 29(9):421-422
[138]李征.辨证分型治疗寻常性痤疮136例疗效观察[J].中国皮肤性病学杂志, 1997, 11(5):294.
[139]游曦闽,辨证分型治疗痤疮83例[J].福建中医学院学报, 2001, 11(3):71.
[140]余方. 80例痤疮的中医分型与治疗[J].广西中医药, 1995, 18(4):5-6
[141]杨艳华中药辨证分型治疗痤疮80例[J].现代中西医结合杂志, 1999, 8(10):1642-1643
[142]李惠林.中医辨证治疗痤疮的体会[J].新中医11, 43(1):131-132
[1]秦长林.燥证琐谈[J].湖北中医杂志, 2001, 23(8):5-6.
[2]清·王冰.黄帝内经素问[M].北京:人民卫生出版社, 1963:499, 391.
[3]金·刘完素.素问玄机原病式(注释本)[M].北京:人民卫生出版社, 1983:206.
[4]孙莹,连博,杨梅.费伯雄治疗燥证经验探要[J].云南中医学院学报, 2009, 32(2):55-57.
[5]陈亦宜,陈涛.新安医家余国佩对燥邪的认识初探[J].中医药临床杂志, 2004, 16(6):598-599.
[6]王殿华.关于中医辛润法的理论思考[J].山东中医药大学学报, 1999, 38(8):493.
[7]张年顺.燥邪的阴阳属性争鸣概述[J].北京中医学院学报, 1990, 13(5):10-12.
[8]魏妮,孟凡冰,王宗柱.浅谈“燥”的阴阳属性[J].国医论坛, 2007, 22(6):9-10.
[9]林旭红.浅议燥邪的发病季节[J].时珍国医国药, 2006, 17(9):1804.
[10]牟全胜,周铭心.西北多燥说[J].新疆中医药, 1991, 9(4):1-6.
[11]周铭心,陶培勇.燥邪病因属性辨析[J].新疆中医药, 2005, 23(6):1-5.
[12]周铭心.西北燥证研究概述[J].上海中医药杂志, 2005, 39(11):43-45.
[13]周铭心,陶培勇.沙漠燥证初探——沙漠石油工人保健研究[J].中医杂志, 1997, 38(8):493-496.
[14]沙塔拉提,孙红友,周铭心.环境地理因素与亚健康状态的关系初探——西北燥证背景研究[J].新疆中医药, 2006, 24(4):74-76.
[15]周铭心,宋晓平,单丽娟,等.新疆各地不同民族居民西北燥证罹患情况流行病学调查分析[J].新疆医科大学学报, 2006, 29(11):1034-1038.
[16]王燕,周铭心.吴鞠通治燥特色探讨—西北燥证治法相关文献研究[J].新疆中医药, 2006, 24(4):1-3.
[17]明·李梃.医学入门[M].上海:上海锦章图书局印行本. 1930.
[18]连博,孙莹,杨梅.浅析喻嘉言《医门法律》燥证辨治之贡献[J].云南中医学院学报, 2009, 32(2):58-59.
[19]何廉臣.重印全国名医验案类编.福州:福建科技出版社, 2003:234.
[20]白君伟,梁苹茂.《医原》“燥?湿”论及其在2型糖尿病中的应用[J].中医杂志, 2008, 49(4):377-378.
[21]王秀莲.《医原》外感燥湿论探析[J].江苏中医, 2000, 21(11):6-8.
[22]余杨,张德新.从“冬三月,水冰地坼”浅谈辛润治燥法的运用[J].时珍国医国药, 2005, 16(1):80.
[23]何宽其.类燥证探讨[J].中医杂志, 2009, 50(6):569.
[24]丁建中,张六通,邱幸凡.外燥致病特点与辨证论治综议[J].时珍国医国药, 2006, 17(3):442-443.
[25]李民,路伟.阳生阴长法治燥浅识[J].湖北中医杂志, 2001, 23(1):20.
[26]周光,刘爱玲,周铭心.燥证证治八法解析[J].辽宁中医杂志, 2009, 36(3):360-361.
[2]牟全胜,周铭心.西北多燥说[J].新疆中医药, 1991,卷(4):1-6.
[3]周铭心.燥邪病因属性辨析[J].新疆中医药, 2005, 23(6):1-5.
[4]王燕,周铭心.吴鞠通治燥特色探讨—西北燥证治法相关文献研究[J].新疆中医药, 2006, 24(4):1-3.
[5]姜德,周铭心.燥邪病因与咳嗽的关系探讨--西北燥证病因相关研究[J].新疆中医药, 2006, 24(5):7-9
[6]王学武,刘浩,周铭心.古今燥证论治的研究概况[J].新疆中医药, 2003,21(3): 53-57.
[7]周铭心,陶培勇.沙漠燥证初探-沙漠石油工人保健研究[J].中医杂志, 1997,38 (8):493-496.
[8]周铭心,宋晓平,单丽娟,等.新疆各地不同民族居民西北燥证罹患情况流行病学调查分析[J].新疆医科大学学报, 2006, 29(11):1034-1038.
[9]吕光耀,李杰,周铭心.燥证及西北燥证病因病机研究概述[J].新疆医科大学学报, 2007, 30(1):26-28.
[10]周铭心,宋晓平,单丽娟,等.西北燥证证候类型分析[J].新疆医科大学学报, 2007, 30(1):1-6.
[11]李鹏,周铭心.新疆和田地区维吾尔族居民西北燥证罹患情况及主要症状分析[J].新疆医科大学学报, 2007, 30(1):14-16.
[12]吕光耀,李杰,周铭心.新疆不同地区不同民族居民生存质量与西北燥证关系的分析[J].新疆医科大学学报, 2007, 30(1):6-10
[13]张育善,周铭心.疏风宣卫合剂治疗银屑病70例疗效观察[J].新疆中医药, 1986(3):45.
[14]周铭心,单丽娟,宋晓平,等.西北燥证外感病因六淫构成情况因子分析.新疆医科大学学报, 2006, 29(12):1123-1130.
[15]赵存娥,李明奎.中医病因病机学[M].北京:科学出版社, 2000.
[16]沙塔娜提,孙红友.周铭心.环境地理因素与亚健康状态的关系初探—西北燥证背景研究[J].新疆中医药, 2006, 24(4):74-76.
[17]沈绍功,韩学杰.中医心病学正名与标准化研究[J].中国中医基础医学杂志, 2007, 13(7):485-487.
[18]韩学杰.中医心病学诊疗规范性研究,中华中医药学会内科心病第八届学术年会,安徽芜湖, 2006:20-31.
[19] Andrew Moran, Dong Zhao, Dongfeng Gu, et, al. The future impact of population growth and aging on coronary heart disease in China:projections from the Coronary Heart Disease Policy Model-China[J]. BMC Public Health 2008, 8:394.
[20] International Database, U. S. Census Bureau [http://www. census. gov/ipc/www/ idb/]
[21] National Bureau of Statistics of China[http://www. stats. gov. cn/english/]
[22] Wang L, Kong L, Wu F, Bai Y, Burton R:Preventing chronic diseases in China. Lancet. 2005, 366(9499):1821-1824.
[23] Omran AR:The epidemiologic transition. A theory of the epidemiology of population change[J]. Milbank Memo Fund Q. 1971, 49(4):509-538.
[24] Yusuf S, Reddy S, Ounpuu S, Anand. S:Global burden of cardiovascular diseases: part I:general considerations, the epidemiologic transition, risk factors, and impact of urbanization. Circulation. 2001, 104(22):2746-2753.
[25] Murray CJ, Lopez AD:Alternative projections of mortality and disability by cause 1990-2020:Global Burden of Disease Study. Lancet 1997, 349(9064):1498-1504.
[26] Gaziano TA:Reducing the growing burden of cardiovascular disease in the develo- ping world. Health Aff(Millwood)2007, 26(1):13-24.
[27] He J, Gu D, Wu X, et, al. :Major causes of death among men and women in China. New Engel J Med 2005, 353(11):1124-1134.
[28] He J, Gu D, Wu X, Wildman RP, Klag MJ, Whelton PK:Major causes of death among men and women in China. New Engel J Med 2005, 353(11):1124-1134.
[29] Leeder S, Raymond S, Greenberg H, Liu H, Esson K:A Race Against Time:the Challenge of Cardiovascular Disease in Developing Countries. New York, NY: Columbia University Center for Global Health and Economic Development; 2004. 10.
[30] Murray CJ, Lopez AD:Mortality by cause for eight regions of the world:Global Burden of Disease Study. Lancet 1997, 349(9061):1269-1276.
[31] Critchley J, Liu J, Zhao D, et, al. Cape well S:Explaining the increase in coronary heart disease mortality in Beijing between 1984 and 1999. Circulation 2004,110(10): 1236-1244.
[32] Weinstein MC, Coxson PG, Williams LW, Pass TM, Stason WB, Goldman L. Forecasting coronary heart disease incidence, mortality, and cost:the Coronary Heart Disease Policy Model. Am J Public Health 1987, 77(11):1417-1426.
[33] Kochanek KD, Murphy SL, et al. Final Data for 2002. National Static’s Reports. Vol53. Hyattsville, Maryland:National Center for Health Statistics;
[34] Anonymous. Heart Disease and Stroke Statistics. American Heart Association;2006.
[35] Mackay J, Mensah GA. The Atlas of Heart Disease and Stroke. Geneva:WHP;2004.
[36] Ridker PM, Hennekens CH, Buring JE, Rifai N. C-reactive protein and other markers of inflammation in the prediction of cardiovascular disease in women. N Eng J Med. 2000,342(12):836-43.
[37] D’Aiuto F, Parkar M, Andreou G, Brett PM, Ready D, Tonetti MS. Periodontitis and atherogenesis:causal association or simple coincidence J Clin Periodontol. 2004,31 (5):402-11.
[38] Ridker PM, Hennekens CH, Buring JE, Rifai N. C-reactive protein and other markers of inflammation in the prediction of cardiovascular disease in women. N Eng J Med. 2000,342(12):836-43.
[39] Noack B, Genco RJ, Trevisan M, Grossi S, Zambon JJ, De Nardin E. Periodontal infections contribute to elevated systemic C-reactive protein level. J Periodontol. 2001, 72(9):1221-7.
[40] Wu T, Trevisan M, Genco R, et al. Examination of the relation between periodontal health status and cardiovascular risk factors:serum total and high-density lipoprotein cholesterol, C-reactive protein, and plasma fibrinogen. Am J Epidermal. 2000, 151:273-82.
[41] Ajwani S, Mattila KJ, Tilvis RS, Ainamo A. Periodontal disease and mortality in an aged population. Spec Care Dent. 2003, 23(4):125-30.
[42] Glurich I, Grossi S, Albini B, et al. Systemic inflammation in cardiovascular and periodontal disease:comparative study. Clin Diagn Lab Immunol. 2002,9(2):425-32.
[43] Janket SJ, Baird AE, Chuang SK, Jones JA. Meta-analysis of periodontal disease and risk of coronary heart disease and stroke. Oral Surg Oral Med Oral Pathol Oral Radiol Endo. 2003, 95(5):559-69.
[44] Khader YS, Albashaireh ZS, Alomari MA. Periodontal diseases and the risk of coronary heart and cardiovascular diseases:a meta-analysis. J Periodontal. 2004, 75(8):1046-53.
[45] Linda L. Humphrey, Rongwei Fu, David I. Buckley, et, al. Periodontal Disease and Coronary Heart Disease Incidence:A Systematic Review and Meta-analysis.
[46] Marmot M, Shipley M, Brunner E, Hemingway H. Relative contribution of early life and adult socioeconomic factors to adult morbidity in the Whitehall II study. J Epidemiol Community Health. 2001, 55:301-307.
[47] Davey Smith G, Ben-Shlomo Y, Lynch J. Life course approaches to inequalities in coronary heart disease risk. In:Stansfeld S, Marmot M, eds. Stress and the Heart. London, England:BMJ Books. 2002,20-49.
[48] Notkola V, Punsar S, Karvonen MJ, Haapakoski J. Socioeconomic conditions in childhood and mortality and morbidity caused by coronary heart disease in adult- hood in rural Finland. Soc Sci Med. 1985, 21:517-523.
[49] Kaplan GA, Salonen JT. Socioeconomic conditions in childhood and ischemic heart disease during middle age. BMJ. 1990, 301:1121-1123.
[50] Gliksman MD, Kawachi I, Hunter D, et al. Childhood socioeconomic status and risk of cardiovascular disease in middle-aged US women:a prospective study. J Epidemiol Community Health. 1995, 49:10-15.
[51] Wannamethee SG, Whincup PH, Shaper G, Walker M. Influence of fathers’social class on cardiovascular disease in middle-aged men. Lancet. 1996, 348:1259-1263.
[52] Davey Smith G, Hart C, Blane D, Hole D. Adverse socioeconomic conditions in childhood and cause-specific adult mortality:prospective observational study. BMJ. 1998, 316:1631-1635.
[53] Frankel S, Davey Smith G, Gunnell D. Childhood socioeconomic position and adult cardiovascular mortality:the Boyd Orr cohort. Am J Epidemiol. 1998,150:1081- 1084.
[54] Davey Smith G, McCarron P, Okasha M, McEwen J. Social circumstances in childhood and cardiovascular disease mortality:prospective observational study of Glasgow University students. J Epidemiol Community Health. 2001, 55:340-341.
[55] Heslop P, Davey Smith G, Macleod J, Hart C. The socioeconomic position of women, risk factors and mortality. Soc Sci Med. 2001, 53:477-485.
[56] Wamala SP, Lynch J, Kaplan GA. Women’s exposure to early and later-life socioeconomic disadvantage and coronary heart disease risk:the Stockholm Female Coronary Risk Study. Int J Epidemiol. 2001, 30:275-284.
[57] Claussen B, Davey Smith G, Thelle D. Impact of childhood and adulthood socioeconomic position on cause specific mortality:the Oslo Mortality Study. J Epidemiol Community Health. 2003, 57:40-45.
[58] Paffenbarger RS, Hyde RT, Wing AL, Lee I-M, Jung DL, Kampert JB. The association of changes in physical-activity level and other lifestyle characteristics with mortality among men. N Engl J Med. 1993,328:538-545.
[59] Stamler J, Stamler R, Neaton JD, et al. Low riskfactor profile and long-termcardiovascular and noncardiovascular mortality and life expectancy:findings for 5 large cohorts of young adult and middle-aged men and women. JAMA. 1999, 282:2012-2018.
[60] Yusuf S, Reddy S, Ounpuu S, Anand S. Global burden of cardiovascular diseases. Part II:variations in cardiovascular disease by specific ethnic groups and geographic regions and prevention strategies. Circulation. 2001, 104:2855-2864.
[61] Yusuf S, Hawken S, Ounpuu S, et al. Effect of potentially modifiable risk factors associated with myocardial infarction in 52 countries(the INTERHEART study):case-control study. Lancet. 2004, 364:937-952.
[62] Magnus P, Beaglehole R. The real contribution of the major risk factors to the coronary epidemics:time to end the“only-50%”myth. Arch Intern Med. 2001, 161:2657-2660.
[63] Greenland P, Knoll MD, Stamler J, et al. Major risk factors as antecedents of fatal and nonfatal coronary heart disease events. JAMA. 2003, 290:891-897.
[64] Lantz PM, House JS, Lepkowski JM, Williams DR, Mero RP, Chen J. Socioeconomic factors, health behaviors, and mortality:results from a nationally representativeprospective study of US adults. JAMA. 1998, 279:1703-1708.
[65] Mika Kivim?ki, Debbie A. Lawlor, George Davey Smith, et al. Socioeconomic Position, Co-Occurrence of Behavior-Related Risk Factors, and Coronary Heart Disease:the Finnish Public Sector Study[J]. American Journal of Public Health, May 2007, 97( 5):874-879.
[66] Debbie A. Lawlor, George Davey Smith, Shah Ebrahim. Association Between Childhood Socioeconomic Status and Coronary Heart Disease Risk Among Postmenopausal Women:Findings From the British Women’s Heart and Health Study[J]. American Journal of Public Health, August 2004, 94( 8):1386-1392.
[67] Marilyn Hravnak, Jeff Whittle, Mary E. Kelley et al. Symptom Expression in Coronary Heart Disease and Revascularization Recommendations for Black and White Patients[J]. American Journal of Public Health, September 2007,97(9):1701- 1708.
[68] Lee H, Bahler R, Park O, Kim C, Lee HY, Kim Y. Typical and atypical symptoms of myocardial infarction among Black, Whites and Koreans. Crit Care Nurs Clin N Am. 2001, 13:531-539.
[69] Teresa T Fung, Vasanti Malik, Kathryn M Rexrode. Sweetened beverage consump- tion and risk of coronary heart disease in women[J]. American Society for Nutrition,2009, 89:1037-1041.
[70] Brook RD, Brook JR, Urch B, Vincent R, Rajagopalan S, Silverman F. 2002. Inhalation of fine particulate air pollutionand ozone causes acute arterial vasocon- striction in healthy adults. Circulation. 2002, 105(13):1534-1536.
[71] Donaldson K, Tran L, Jimenez LA, Duffin R, Newby DE, Mills N, et al. 2005b. Combustion-derived nanoparticles:a review of their toxicology following inhalation exposure. Part Fibre Toxicol 2:10.
[72] National High Blood Pressure Education Program Working Group on High Blood Pressure in Children and Adolescents. The fourth report on the diagnosis, evaluation, and treatment of high blood pressure in children and adolescents. Pediatrics. 2004, 114:555-576.
[73] Chobanian AV, Bakris GL, Black HR, et al. Seventh report of the Joint National Committee on Prevention, Detection, Evaluation, and Treatment of High Blood Pressure. Hypertension. 2003, 42:1206-1252.
[74] Sorof JM, Lai D, Turner J, Poffenbarger T, Portman RJ. Overweight, ethnicity, and the prevalence of hypertension in school-aged children. Pediatrics. 2004,113:475- 482.
[75] Flynn JT, Mitsnefes M, Pierce C, Cole SR, Parekh RS, Furth SL, Warady BA. Blood pressure in children with chronic kidney disease:a report from the Chronic Kidney Disease in Children study. Hypertension. 2008, 52:631-637.
[76] van den Elzen AP, de Ridder MA, Grobbee DE, Hofman A, Witteman JC, Uiterwaal CS. Families and the natural history of blood pressure:a 27-year follow-up study. Am J Hypertens. 2004, 17:936-940.
[77] Chobanian AV, Bakris GL, Black HR, et al. Seventh report of the Joint National Committee on Prevention, Detection, Evaluation, and Treatment of High Blood Pressure[J]. Hypertension, 2003, 42:1206-1252.
[78] Qureshi AI, Suri MF, Kirmani J et al. Prevalence and trends of prehypertension and hypertension in United States:National Health and Nutrition Examination Surveys 1976 to 2000[J]. Med Sci Monit, 2005, 11:CR403-CR409.
[79] Burt VL, Whelton P, Roccella EJ, et al. . Prevalence of hypertension in the US adult population:results from the Third National Health and Nutrition Examination Survey, 1988-1991[J]. Hypertension. 1995, 25:305-313.
[80] Vasan RS, Beiser A, Seshadri S, et al. Residual lifetime risk for developing hypertension in middle-aged women and men:the Framingham Heart Study[J].JAMA. 2002, 287:1003-1010.
[81] Birkenhager WH, Staessen JA. Convergence of atherosclerosis and Alzheimer’s disease. Lancet. 2004, 363:2091.
[82] Kilander L, Nyman H, Boberg M, Hansson L, Lithell H. Hypertension is related to cognitive impairment:a 20-year follow-up of 999 men. Hypertension. 1998,31:780- 786.
[83] Launer LJ, Ross GW, Petrovitch H, Masaki K, Foley D, White LR, Havlik RJ. Midlife blood pressure and dementia:the Honolulu-Asia Aging Study. Neurobiol Aging. 2000, 21:49-55.
[84] Duron E, Hanon O. Hypertension, cognitive decline and dementia. Arch Cardiovasc Dis. 2008, 101:181-189.
[85] Singh-Manoux A, Marmot M. High blood pressure was associated with cognitive function in middle-age in the Whitehall II Study. J Clin Epidemiol. 2005,58:1308- 1315.
[86] Waldstein SR, Brown JR, Maier KJ, Katzel LI. Diagnosis of hypertension and high blood pressure levels negatively affect cognitive function in older adults. Ann Behav Med. 2005, 29:174-180.
[87] Elias MF, Elias PK, Sullivan LM, Wolf PA, D’Agostino RB. Lower cognitive function in the presence of obesity and hypertension:the Framingham Heart Study[J]. Int J Obes Relat Metab Disord. 2003, 27:260-268.
[88] Wolf PA, Beiser A, Elias MF, Au R, Vasan RS, Seshadri S. Relation of obesity to cognitive function:importance of central obesity and synergistic influence of concomitant hypertension-the Framingham Heart Study[J]. Curr Alzheimer Res. 2007, 4:111-116.
[89] Waldstein SR, Katzel LI. Interactive relations of central versus total obesity and blood pressure to cognitive function[J]. Int J Obes(Lond). 2006, 30:201-207.
[90] Bolger N, Zuckerman A. A framework for studying personality in the stress process[J]. J Pers Soc Psychol 1995, 69:890-902.
[91] Larsen RJ, Ketelaar T. Personality and susceptibility to positive and negative emotional states[J]. J Pers Soc Psychol 1991, 61:132-140.
[92] McCrae RR, Costa PT Jr. Adding liebe und arbeit:The full five-factor model and well-being[J]. Personality & Social Psychology Bulletin 1991, 17.
[93] Costa, PT, Jr;McCrae, RR. The revised neo personality inverntory(neo pi-r) professional manual[J]. Odessa, FL;Psychological Assessment Resources:1992.
[94] Watson D, Clark LA. On traits and temperament:General and specific factors of emotional experience and their relation to the five-factor mode[J] . J Pers 1992, 60:441-476.
[95] Izard CE, Libero DZ, Putnam P, Haynes OM. Stability of emotion experiences and their relations to traits of personality[J]. J Pers Soc Psychol 1993, 64:847-860.
[96] Bolger N, Schilling EA. Personality and the problems of everyday life:The role of neuroticism in exposure and reactivity to daily stressors[J]. J Pers 1991,59:355-386.
[97] Pickering, A. Gray, JA. The neuroscience of personality. In:Pervin, L;John, O, editors. Handbook of personality:Theory and research. New York:Gilford Press. 2001. 227-299.
[98] Canli T, Sivers H, Whitfield SL, et, al. Amygdala response to happy faces as a function of extraversion[J]. Science 2002, 296-2191.
[99] Canli T, Zhao Z, Desmond JE, Kang E, Gross J, Gabrieli JD. An fMRI study of personality influences on brain reactivity to emotional stimuli[J]. Behav Neurosci 2001, 115:33-42.
[100]Zald DH, Mattson DL, Pardo JV. Brain activity in ventromedial prefrontal cortex correlates with individual differences in negative affect[J]. Proceedings of the National Academy of Sciences of the United States of America 2002,99:2450-2454.
[101]Gusnard DA, Ollinger JM, Shulman GL, Cloninger CR, Price JL, Van Essen DC, Raichle ME. Persistence and brain circuitry[J]. Proceedings of the National Academy of Sciences of the United States of America 2003, 100:3479-3484.
[102]Schwartz CE, Wright CI, Shin LM, Kagan J, Rauch SL. Inhibited and uninhibited infants“grown up”:Adult amygdalar response to novelty. Science 2003,300:1952- 1953.
[103]Canli T, Amin Z, Haas B, Omura K, Constable RT. A double dissociation between mood states and personality traits in the anterior cingulate[J]. Behav Neurosci 2004, 118:897-904.
[104]Gray JR, Burgess GC, Schaefer A, Yarkoni T, Larsen RJ, Braver TS. Affective personality differences in neural processing efficiency confirmed using fMRI. Cogn Affect Behav Neurosci 2005, 5:182-190.
[105]Omura K, Todd Constable R, Canli T. Amygdala gray matter concentration is associated with extraversion and neuroticism[J]. Neuroreport 2005, 16:1905-1908.
[106]Rauch SL, Milad MR, Orr SP, Quinn BT, Fischl B, Pitman RK. Orbitofrontal thickness, retention of fear extinction, and extraversion[J]. Neuroreport 2005,16:1909-1912.
[107]Johnson DL, Wiebe JS, Gold SM, Andreasen NC, Hichwa RD, Watkins GL, Boles Ponto LL. Cerebral blood flow and personality:A positron emission tomography study[J]. Am J Psychiatry 1999, 156:252-257.
[108]Christopher, Wright, Eric Feczko, Bradford Dickerson, et, al. Neuroanatomical Correlates of Personality in the Elderly Neuroimage[J]. 2007, 35(1):263-272.
[109]Coccaro EJ, Seiver JL, Klar HM, et al. Serotonergic studies in patients with affective and personality disorders[J]. Archives of General Psychiatry 1989,46:587- 599.
[110]O'Keane VO, Moloney E, O'Neill H, O'Connor A, Smith R, Dian TG. Blunted prolactin responses to d-Fenfluramine in sociopathy:Evidence for subsensitivity of central serotonergic function[J]. British Journal of Psychiatry 1992, 160:643-646.
[111]Cleare A, Bond A. Does central serotonergic function correlate inversely with aggression:A study using D-fenfluramine in healthy subjects[J].Psychiatry Research 1997, 69:89-95.
[112]Cleare AJ, Bond AJ. The effect of tryptophan depletion and enhancement on subjective and behavioural aggression in normal male subjects[J]. Psychopharma- cology, 1995, 118:72-81.
[113]Coccaro EF. Impulsive aggression and central serotonergic system function in humans:an example of adimensional brain-behavior relationship[J]. Int Clin Psychopharmacol, 1992, 7:3-12.
[114]Marsh DM, Dougherty DM, Moeller FG, Swann AC, Spiga R. Laboratory- measured aggressive behavior of women:Acute tryptophan depletion and augmentation[J]. Neuropscyhopharmacology, 2002, 26:660-671.
[115]Beverly H. Brummett, Stephen H. Boyle, Cynthia M. Kuhn. Associations among central nervous system serotonergic function and neuroticism are moderated by gender[J]. Biol Psychol. 2008, 78(2):200-203.
[116]Nishizawa S, Benkelfat C, Young SN, Leyton M, Mzengeza S, de Montigny C, et al. Differences between males and females in rates of serotonin synthesis in human brain[J]. Proceedings of the National Academy of Science:USA 1997,94:4823- 4824.
[117]Amy C Iversen, Lauren van Staden1, Jamie Hacker Hughes, et, al. The prevalence of common mental disorders and PTSD in the UK military:using data from a clinical interview-based study[J]. BMC Psychiatry 2009, 9:68.
[118]Hoge CW, Lesikar SE, Guevara R, et, al. Mental disorders among U. S. military personnelin the 1990s:association with high levels of health care utilizationand early military attrition[J]. Am J Psychiatry 2002, 159(9):1576-1583.
[119]Rosenheck R, Leda C, Gallup P, et, al. Initial assessment data from a 43-site program for homeless chronic mentally ill veterans[J]. Hosp Community Psychiatry 1989, 40(9):937-942.
[120]Iversen A, Nikolaou V, Greenberg N, et, al. What happens to British veterans when they leave the armed forces[J]. Eur J Public Health 2005, 15(2):175-184.
[121]Hoge CW, Castro CA, Messer SC, McGurk D, Cotting DI, Koffman RL:Combat duty in Iraq and Afghanistan, mental health problems, and barriers to care[J]. N Engl J Med, 2004, 351(1):13-22.
[122]Dlugosz LJ, Hocter WJ, Kaiser KS, Knoke JD, Heller JM, Hamid NA, Reed RJ, Kendler KS, Gray GC:Risk factors for mental disorder hospitalization after the Persian Gulf War:US Armed Forces, June 1, 1991-September 30,1993.J Clin Epidemiol.1999, 52(12):1267-1278.
[123]Maud Lemoine, Aurélie Revaux, Claire Francoz, et, al. Albumin liver dialysis as pregnancy-saving procedure in cholestatic liver disease and intractable pruritus[J]. World J Gastroenterol, 2008, 14(42):6572-6574.
[124]Adam Reich, Jacek C. Szepietowski1 Mediators of Pruritus in Psoriasis[J]. Med- iators of Inflammation, 2007, 10:1-6.
[125]Seppo AM Saari, Kirsi H Juuti, Joanna H Paloj?rvi, et, al. Demodex gatoi- associated contagious pruritic dermatosis in cats-a report from six households in Finland[J]. Acta Veterinaria Scandinavica, 2009, 51:40.
[126]S M Bates, G Dilke-Wing. Determination of iron status in women attending genitourinary clinics with pruritus vulvae[J]. Sex Transm Infect, 2006, 82:489-490.
[127]Maryam Akhyani1, Mohammad-Reza Ganji2, Nasrin Samadi, et, al. Pruritus in hemodialysis patients[J]. BMC Dermatology, 2005, 5:7.
[128]Yonova D. Pruritus in certain internal diseases[J]. HIPPOKRATIA 2007,11(2): 67-71.
[129]Greaves MW, Davies MG. The current status of histamine receptors in human skin:indirect evidence[J]. Br. J Dermatol 1982, 23(suppl):101-105.
[130]Stephen E. Waisglass, Avery Gillick. Pruritus possibly associated with a porto -systemic shunt in a bichon frise puppy[J]. Can Vet J 2006, 47:1109-1111.
[131]TA Salami, SO Samue, KC Eze, et, al. Prevalence and characteristics of aquagenicpruritus in a young African population[J]. BMC Dermatology, 2009, 9:4.
[132]Zouboulis CC. Acne:current aspects on pathology and treatment[J]. Dermatological Experience,1999, 4:6-37.
[133]Chu TC. Acne and other facial eruptions. Medicine, 1997, 25:30-3.
[134]Witkowski JA, Parish LC. The assessment of acne:an evaluation of grading and lesion counting in the measurement of acne[J]. Clin Dermato, 2004, 22:394-7.
[135]Bataille V, Snieder H, MacGregor AJ, et, al. The influence of genetics and environmental factors in the pathogenesis of acne:a twin study of acne in women[J]. J Invest Dermatol,2002, 119:1317-22.
[136]鞠强,石继海,夏隆庆.白介素-1与痤疮.国外医学·皮肤性病学分册. 2001, 27:24-26.
[137]Homemaunn S, SeltmarnH, Kodelia V, et al. Interleukin and protein are expressed in eultured human sehacyes at seady-teate and their leves are harely influneed by lipoolysanchandes. J Invest Der-matol,1997;108:382.
[138]Zalluga E. Skin reactions to antigens of Propionihacteriom acnes in patientswith acne vulgaris treatedwith aumvaccine. J British Journal of Dermatology,2001, 43:389-397.
[139]Valacckj G, Rimbach G, Salou C, et al. Effect of henxoyl perox-side on antioxidant status, NF-kappaB activity and interleukin-lalpa gene espresson in human keratno -cutes. J Tosicology 2001, 165:225-234.
[140]Yaneoka J, Kume T, Akinori A, et al. Suppressive effect of zine ion on iNOS expression induced by interferongamma or tumor necro-sis factor-slpha in murine keratnocytes. J Dermatological Sciemce 2000, 20:27-35.
[141]高宇.心系证候规范性研究[D]:[硕士学位论文].江苏南京:南京中医药大学, 2006. 11-12.
[142]耿建领,黄涛.张子和心系疾病学术经验略要[J].光明中医, 2003,18(109): 47-48.
[143]丁书文,李晓,李运伦.热毒学说在心系疾病中的构建与应用[J].山东中医药大学学报, 2004, 28(6):413-416.
[144]董玉山.781例心系疾病证治特点的研究[D]. [硕士学位论文].山东:山东中医药大学, 2001.
[145]戴天芳,柳芳.《金匮要略》治心七法[J].中医研究, 2005, 18(2):7-8.
[146]李素清.《伤寒论》“悸”的辨治规律探讨[J].中医药研究, 1998, 14(4):1-2.
[147]顾宁,陈红景.《伤寒论》心悸证治浅析[J].中医药研究, 2000, 16(1):3-4.
[148]陈国英.《伤寒论》中温法在心系疾病中的运用考释[J].中医药学刊, 2004,22(5):904-905.
[149]谈文峰,丁倩.柴胡疏肝散治疗心系疾病验案举隅[J].江苏中医,1998,19(10): 33.
[150]白雪,王明杰.从玄府论治心系疾病的经验浅析[J].首都医药, 2005,11(17): 41-42.
[151]李必旭.林钟香治疗心系疾病验案三则[J].辽宁中医杂志,2007,34(12): 1796-1797.
[152]丁成华,曹晓瑞,马丽珍.略论心系疾病从脾胃论治[J].江西中医学院学报, 2007, 19(5):17-18.
[153]刘明武.伤寒论对心系疾病的辨治规律探析[J].辽宁中医, 1997, 24(3):107-108.
[154]胡爱萍.试论《伤寒论》中心系疾病的用药原则与特点[J].甘肃中医, 2000,3:2-3.
[155]苏明.栗锦迁教授经验介绍--心系疾病治疗中化痰法的应用[J].天津中医药, 2006, 23(2):103-104.
[156]梁凤信,郭茂松,孙元莹.郭文勤教授治疗心系疾病学术思想[J].黑龙江中医药, 2001, 6:2-3.
[157]蔚永运,郭艳.从心系疾病论心与表的关系[J].山东中医杂志1999,18(10): 435-436.
[158]范磊,张成博,杨海燕.“心部于表”的理论认识及其临床意义[J].山东中医杂志, 2005, 29(4):261-262.
[159]刘莹,李平.论李永成从脾胃从神论治皮肤病[J].辽宁中医, 2008,35(5): 772-773.
[160]刘大文.面部痤疮从心论治[J].北京中医药, 2008, 27(1):20-21.
[161]谷玉琴,孙月吉.心理因素与痤疮[J].中华皮肤科杂志2006, 39(2):119-120.
[162]李旺,张淑萍.刘完素燥证论治探析[J].浙江中医杂志, 2001, (5):209.
[163]孟繁洁.刘完素燥论阐微[J].中国医药学报, 2004, 19(4):200.
[164]王爱民.吴鞠通论下焦温病阴伤证治浅析[J].江苏中医, 1999, 20(12):6.
[165]马伯艳,李冀.《温病条辨》保阴护津思想探析[J]. ACMP, 2003, 31(1):56.
[166]张晓光.吴鞠通养阴保津学术思想探要[J].实用中医内科杂志, 2003, 17(4):261.
[167]马爱莲.治疗温病当注重养阴[J].现代中西医结合杂志, 2005, 14(13):1698.
[168]周铭心,王树芬.中医时间医学[M].长沙:湖南科学技术出版社, 1989:76
[169]蔡能,史鸿璋,陈钟舜,等.心身病[M].上海:上海科技文献出版社, 1985, 88.
[170]Schafer T, Nienhaus A, Vieluf D, et al. Epidemiology of acne in the general population:the risk of smoking[J]. Br J Dermato, 2001, 145:100-104.
[171]刘叶兰.中医对寻常痤疮病因病机的认识[J].河南中医, 2006, (26)4:6-8.
[172]毛文姣.痤疮病因概述[J].辽宁中医药大学学报, 2008, 10(12):70-71.
[173]张爱清,李克华,王甘棠,等.痤疮患者个性特征及情绪反应的研究[J].中国皮肤性病学杂志, 1997, 13(1):17.
[174]钱江,杨柳,兰海梅,等.南方地区寻常痤疮中医临床流行病学调查分析[J].医学信息, 2006, 19(10):1807-1809.
[175]陈慧.痤疮的流行病调查及中医分型和积雪甙对造模大鼠炎症结节作用机理探讨[D].湖北:湖北中医学院, 2006,硕士学位论文.
[176]何弘.乌鲁木齐市女性青春期后痤疮病因初探[J].中国麻风皮肤病杂志, 2007, 23(10):921.
[177]李立明.流行病学[M]. 4版.北京:人民卫生出版社, 2000:237.
[178]EzzatiM, LopeZ AD, Rodgers A, et a. l Selected major risk factor and global and regional burden of disease[J]. Lancet, 2002, 360:1347-1360.
[179]程祖亨,汪师贞.新疆维吾尔族自治区不同民族高血压流行病学因素的调查研究[J].新疆医学院学报, 1982, 5(2):103-111.
[180]Hiroshi Kawamura, Medet. J, Hiromim. 24-hour, blood pressure in Uygur, Kazakh and han elderly subjects in China[J]. Hypertens Res. 2000, 23(2):177-185.
[181]李江,王建平.新疆南疆农村居民饮水现状与相关地方病问题[J].地方病通报, 1997, 1.
[182]王连方,郑宝山,王生玲.新疆水砷及其对开发建设的影响[J].地方病通报, 2002, 17(1).
[183]寇风荣,摆宏萍,居尼斯别克,景兆兰,阮华.米泉市农村饮用水水氟水砷含量的检测[J].现代预防医学, 2007, 14.
[184]史红,周铭心.便秘的病因研究.新疆中医药, 2007;25(3):107-111.
[185]丁建中,龚权,朱惠玲,等.外燥对小鼠呼吸道分泌功能与抗体的影响[J].长江大学学报(自然版). 2005, 2(12):341-342, 351.
[186]丁建中,张六通,陈刚,等.外燥对小鼠气管?肺与皮肤影响的病理学研究[J].时珍国医国药, 2007, 18(3):527-528.
[187]陈刚,丁建中,张六通,等.细菌攻击对外燥小鼠气道与肠道分泌及免疫功能的影响[J].湖北中医杂志, 2007, 29(2):3-5.
[188]丁建中,龚权,陈刚,等.外燥对小鼠气管细胞凋亡与bcl-2基因及bax基因表达的影响[J].时珍国医国药, 2007, 18(1):15-16.
[189]李永军,赵刚,司小莉,等.戈壁气候对新兵肺通气功能的影响[J].总装备部医学学报, 2008, 10(4):195-196.
[190]刘浩,周铭心.西北燥证(新疆)与常年性变应性鼻炎?慢性支气管炎的关系研究.新疆医科大学2005级硕士论文.中国优秀硕士学位论文全文数据库
[191]杨正志,杨利华.气象因素对老年高血压患者血压的影响[J].世界中西医结合杂志, 2009, 4(6):418-419.
[192]王玲,白原,刘小云,等.高血压与气象因素的关系[J].医学综述, 2007,13(3): 239-240.
[193]黄如训,肖小华,李玲,等.气象因素促发卒中的实验研究[J].中华老年医学杂志, 2001, 20(5):366-368.
[194]董蕙青,郭琳芳.脑卒中发病与气象要素变化关系分析[J].广西气象, 2006,20 (2):40-43.
[195]董蕙青,李雄,郭琳芳,等.南宁市脑卒中疾病死亡与气象要素的关系[J].广西科学院学报, 2005, 21(2):127-130.
[196]王健,张新英,窦月华.乌鲁木齐市高血压病的气象诱因和指数预报初探[J].华西医学, 2009, 12:289-295.
[197]陈凤辉,程祖亨,玛依拉·吾甫尔,等.新疆“塔克拉玛干沙漠人”高血压与摄盐量摄钠量相关性研究[J].新疆医科大学学报, 2009. 32(7):882-884.
[198]Momityama Y, Hirano R, Taniguchi H, et al. Effects of interleukin-1gene polymorphisms on the development of coronary artery disease associated with Chlamydia Pneumoniae infection[J]. J Am Coll Cardiol, 2001, 38(3):712-7.
[199]Francis SE, Camp NJ, Dewberry RM, et al. Interleukin-1 receptor antagonist gene polymorphism and coronary artery disease[J]. Circulation, 1999, 99(7):861-6.
[200]Miranda-Malpica E, Martinez-Rios MA, Fragoso JM, et al. The interleukin 1B-511 polymorphism is associated with the risk of developing restenosis after coronary stenting in Mexican patients[J]. Hum Immunol, 2008, 69(2):116-21.
[201]Duff GW. Evidence for genetic variation as a factor in maintaining health[J]. Am J Clin Nutr, 2006, 83(suppl):431S-5S.
[202]Soylu O, Yildirim A, Coker A, et al. Interleukin-1B(-511)gene polymorphism is associated with acute coronary syndrome in the Turkish population[J]. Eur Cytokine Netw, 2008, 19(1):42-8.
[203]Latkovskis G, Licis N, Kalnins U. C-reactive protein levels and common polymorphisms of the interleukin-1 gene cluster and interleukin-6 gene in patients with coronary heart disease[J]. Eur J Immunogenet, 2004, 31(5):207-13.
[204]骆秦,李南方,周玲.β2-肾上腺素能受体基因Arg16/G ly多态性与新疆哈萨克族高血压的关系[J].科学技术与工程, 2010, 10(28):6859-6966.
[205]杨忠伟,冯秀丽,吕继荣.新疆不同民族冠心病患者eNOS基因894G/T多态性研究[J].新医学, 2010, 41(1):21-23.
[206]王慧,刘奇迹,黎莉. LRPS/6基因多态与中国汉族人群冠心病的关联分析[J].国际遗传学杂志, 2009, 32(4):12-14.
[207]林雪,尚玉红,马丽.环境地理因素与更年期高血压的关系探微—西北燥证背景研究[J].中华中医药杂志2008, 23(2):125-127.
[208]王素娥.神经衰弱的中医辨证分型治疗[J].中国乡村医药杂志, 2003, 10(6):17.
[209]李慧杰,李德新.郁证之症因及证治规律探析[J].辽宁中医药大学学报, 2007, 9(3):24-25.
[210]徐海燕.神经衰弱的中医辨证施治体会[J].中西医结合与祖国医学, 2005, 9(9):836-837.
[211]高华,沙塔娜提·穆罕默德,周铭心.西北燥证与心理素质关系探讨[J].新疆医科大学学报, 2007, 30(1):17-19.
[212]潘祥龙,谢珉宁,丁佳频.润燥止痒胶囊治疗皮肤瘙痒症的临床研究[J].上海中医药杂志, 2004, 38(8):33-34.
[213]马宽玉,朱璐.润肤止痒汤治疗老年性皮肤瘙痒症30例临床观察[J].四川中医, 2005, 25(12):95-96.
[214]丁仁明,李谦,刘复兴.辨证分型治疗单纯性皮肤瘙痒症62例疗效观察[J].云南中医中药杂志, 2007, 28(4):12-13.
[215]沈乐.痤疮论治杂谈[J].光明中医, 2006, 21(4):14.
[216]李东海,肖红利,林少健,等.从肺热脾虚论治寻常性痤疮64例[J].广州中医药大学学报, 2006, 23(1):32-34.
[217]孟军,王蕾,徐信杰,等.辨病辨证相结合治疗痤疮临床观察[J].中国中西医结合皮肤性病学杂志, 2008, 7(2):93-94.
[218]许嗣立,严石林.小议痤疮从肝阳虚论治[J].四川中医, 2007, 25(6):27-28.
[219]周喜芬.浅析痤疮从肝论治之机理[J].内蒙古中医药, 2008, 1:5-6.
[220]余晓琳,陈军平,林晨.从肝郁肾虚论治寻常性痤疮48例[J].辽宁中医药大学学报, 2008, 10(6):93-94.
[221]张秀美,王秀莲.痤疮从湿热论治探讨[J].天津中医药, 2007, 24(6):521-522.
[222]田维军,魏桂芝.中医虚证全书[M].江西科学技术出版社, 1997, 12.
[223]孙广仁,中医基础理论,普通高等教育”十五”国家级规划教材.北京:中国中医药出版社, 2002, 8:209.
[224]匡调元.中医病理研究[M].上海:上海科学技术出版社, 1980:66.
[225]王琦.中医体质学[M].北京:人民卫生出版社, 2005. 8.
[226]王琦,盛增秀.中医体质学说M.南京:江苏科学技术出版社, 1982:2.
[227]母国成.中医体质学说及其异化[J].新中医, 1983, 15(9):9.
[228]田代华.论体质与证候[J].山东中医学院学报, 1983, 7(1):6.
[229]朱庭仪.中医体质分型100例分析[J].成都中医药大学学报, 1996, 19(4):48.
[230]罗慰慈.鉴别咳嗽症状时应重视环境因素临床肺科杂志[J]. 2009, 14(3):285.
[231]王燕,周铭心.新疆各地不同民族阴阳虚证流行病学调查分析[J]. 2008. 49(4):345-348.
[232]王琦,朱燕波.中国一般人群中医体质流行病学调查—基于全国9省市21948例流行病学调查数据[J].中华中医药杂志, 2009, 24(1):7-12.
[233]吴承玉,骆文斌,王娜娜等.江苏地区2043例中医体质流行病学调查研究[J].中国中医基础医学杂志, 2009, 15(2):138-140.
[234]李杰.中医体质分类的流行病学调查及阳虚体质的相关研究[].江苏:南京中医药大学,博士学位论文, 2008.
[235]周晓平,杨进.中医气象学现代研究方法探讨[J].中国中医药信息杂志, 2007, 14(2):6.
[236]张学忠,董得平.中国青少儿生长发育不同年龄阶段与地理气象因素关系的灰色关联分析[J].中国体育科技, 1996, 32(4):62.
[237]蔡民坤.心气虚证与气虚证的相互关系浅析[J].实用中医内科杂志, 2008, 22(5):27.
[238]李燕.王昕.心气虚证与心功能指标相关性的meta分析[J].中外医疗, 2009, 12:174-175.
[239]王阶,何庆勇,姚魁武. 1069例冠心病脏腑虚证的多中心前瞻性研究[J].中华中医药杂志, 2009, 1:208-213.
[240]宋红普,何裕民. 476例原发性高血压患者体质特点研究[J].上海中医药大学学报, 2001, 15(2):33-35.
[241]林谦,陈焱木,金法. 371例原发性高血压患者的中医体质辨证研究[J].现代中医药, 2004, 3:17-18.
[242]姚晓天,胡伟,程志清.高血压影响因素与中医体质类型相关性研究[J].中医药学刊, 2004, 22(12):2314-2316.
[243]吴宏东,王琦,董静.高血压病体质因素的多元Logistic回归分析[J].北京中医药大学学报(中医临床版)2009, 16(5):12-13.
[244]魏源.神经衰弱患者五大价格特质研究[J].中国临床康复, 2004, 8(30):6585-6587.
[245]钱铭怡.心理咨询与心理治疗[M].北京:北京大学出版社, 1984:232-54.
[246]冯栋年,刘燕玲,张新,等.阳虚证患者遗传学特征探讨[J].中西医结合杂志, 1989, 9(1):24.
[247]国维,范瑞强,尹玉贞,等.中药消痤灵治疗痤疮的多中心随机对照研究[J].广州中医药大学学报, 1995, 12(3):6-8.
[248]成改霞.寻常痤疮五脏辨治规律及五脏分级相关性研究[D].福建:福建中医学院硕士学位论文, 2005.
[249]郑雨佳.座疮200例中医体质流行病学调查[D].四川:成都中医药大学,硕士学位论文, 2008.
[1]吴辉,于扬文,吴伟等. 116例冠心病患者中医证候及病因分析[J].江苏中医, 2004, 25(10):30.
[2]李芳,樊相军.胸痹诱发因素与辨证分型的调查分析[J].人民军医, 2001, 4(8):484-486
[3]李军,王杰.病证结合的冠心病心绞痛病因病机探讨[J].中国中医基础医学杂志, 2003, 13(7):531-533
[4]林谦.冠心病(胸痹心痛)的病因病机及辨证论治[J].中国临床医生, 2000,28(10): 11-14
[5]杨维益.心绞痛诊治一得.浙江中医杂志, 1981, (9):386.
[6]阁洪臣.略谈胸痹心痛的病理与证治.陕西新医药, 1979, (6):49.
[7]黄河清.《伤寒论》少阴寒化证与冠心病心肌缺血相关性探讨.中医杂志, 1999, 40(10):585.
[8]黄宗鉴.益气活血法治疗冠心病心绞痛临床疗效观察.福建中医药, 1988,(l):27.
[9]张伯臾,蓝建华.中医内科学.北京:人民卫生出版社, 1990:215.
[10]王秀珍等.论心生痰与心痛.黑龙江中医药, 1985, (4):11.
[11]林功铮.曹锡寅论治心绞痛.辽宁中医杂志, 1983, (6):5.
[12]田养年.从痰瘀论治冠心病.辽宁中医杂志, 1983, (4):25.
[13]胡冬裴.胸痹古今证型归类研究团.上海中医药大学学报, 2004, 18(4):8-9
[14]邱少波.浅谈胸痹的病因病机.国医论坛. 2007, 22(l):13-14
[15]李秀峰,张瑜珠.高血压病的病因病机?辨证论治及体会.中国医药指南, 2009, 7(4):109-110
[16]黄力,史载祥.高血压病的病因病机和辨证论治.中国临床医生, 1999, 27(12):10-11
[17]古炽明,丁有钦.高血压病证候文献分析述评[J].中医药学刊, 2003, 21(7):1156.
[18]赵奕,方文岩,赵治等.高血压病中医辨证分型与基底动脉血流动力学探讨[J].天津中医药, 2005, 22(4):297.
[19]冯向阳.辨证分型治疗高血压病112例临床观察[J].中医药导报, 2006, 12(8):34.
[20]白春锦,周濠,王丽等.不同中医证型高血压患者的心血管危险因素分层?高血压分期?分级及血脂等指标变化特征[J].中医康复研究, 2005, 9(23):145.
[21]柳静,张旭生.高血压病从气血论治[J].吉林中医药, 2007, 27(1):14.
[22]董德保.清脑熄风汤治疗高血压病40例小结[J].光明中医, 2004, 19(6):52.
[23]张喜奎.陈亦人教授医话(连载)[J].国医论坛, 2000, 15(3):14.
[24]吴华堂,范金茄.风眩(高血压病)病因病机证治探析[J].中医药学刊, 2003, 21(3):467-468
[25]李玉兰.从中医学角度探讨高脂血症的病因病机[J].四川中医, 2010, 28(3):17.
[26]郭技,郭宗英.降脂饮治疗高脂血症的临床观察.辽宁中医杂志, 1996(2):69.
[27]张秉志.中药泡饮治疗高脂血症.江苏中医, 1995, 16(2):24.
[28]李长生,郭凤仗,盛子敬. 49例高脂血症的临床观察,实用中西医结合杂志, 1996, 9(2):98.
[29]熊文生,李任先,刘国普等.辨治高脂血症重在理脾化痰.新中医, 1996(11):4.
[30]刘剑,王献民,周心芳等.自制神农双降丸治疗高脂血症临床观察.中医药研究, 1996, 9(1):26-28
[31]曾昭龙,李小敏,尹炳生等.正脂丸对血脂影响的研究.辽宁中医杂志, 1999, 26(5):234-235
[32]屠光英,周晓军. 44例高脂血症的临床观察.上海中医药杂志, 1991(9):8-9
[33]董玉宽.健脾为主治疗老年性高指血症27例.辽宁中医杂志, 1994,21(12): 545-546
[34]王静宇,陈培玉,张立营等.山明乌参三黄二泽汤治疗高脂血症80例临床观察.四川中医, 1994(12):24-25
[35]吴少萍.“降脂汤”治疗高脂血症40例观察.上海中医, 1998(9):21.
[36]胡正松.高脂血症从痰淤论治五法.江苏中医, 1995, 16(12):32-33
[37]张三林,方亚伦,张正臣等.黄炭降脂片治疗高脂血症92例临床分析.实用中西医结合杂志, 1995, 8(4):249.
[38]崔衍富.血塞通结合辨证治疗高脂血症100例.辽宁中医杂志, 1997, 24(8):403
[39]张子明,吴振萍,侯健民等.降脂口服液治疗高脂血症120例.实用中西医结合杂志, 1993, 6(5):279-280
[40]巫奕丽,董卫.降脂饮治疗高脂血症53例临床观察.中医药研究, 1998, 14(3):15-16
[41]周海虹.谈高脂血症的中医证治.实用中西医结合杂志, 1995, 8(7):418.
[42]刘杏鑫,清脂饮治疗高脂血症88例临床分析.实用中西医结合杂志, 1997, 10(7):630.
[43]陈川.补肾、化痰和活血对血脂代谢影响的比较.上海中医药杂志, 1997(2):22-25
[44]郑清莲,薛东波.刘茂甫教授对高脂血症病因病机的认识及治疗经验.中医药研究, 1993(6):17-18
[45]杨大男,杨代勇.张惠琴逍遥丸治疗高脂血症84例?陕西中医, 1995, 16(3):109-110
[46]张绮桂.降脂汤治疗高脂血症30例观察.中医药研究, 1992(6):33-34
[47]解荣.“三子汤”治疗高脂血症30例观察.上海中医药杂志, 1997(10):28.
[48]杨相彬.降脂汤治疗高胆固醉血症临床观察.实用中西医结合杂志, 1995, 8(6):377.
[49]刘少云.外用消脂贴治疗高脂血症60例疗效分析.天津中医, 1999, 16(8):15-16
[50]刘国凤,周静.降脂化淤胶囊治疗高脂血症20例.河北中医, 1999, 21(2):90-91
[51]胡晓灵,李以义,曾斌芳等.复方降脂散治疗老年高脂血症临床观察.中医药研究, 1997, 13(3):11-13
[52]俞兴祥,张受韵.自拟降脂通脉汤治疗高脂血症36例.上海中医药杂志, 1995(4):23-24
[53]杨文瑞,沈王安.降脂叶袋泡茶治疗高脂血症35例报道.上海中医药杂志, 1993(12):29.
[54]朱丽华.自拟降脂汤治疗高脂血症41例临床分析.中医药研究, 1998, 14(3):15.
[55]姚克裘,周曾绮,陈敏先.血通胶囊对老年高脂血症的影响临床研究.上海中医药杂志, 1995(l):37-38
[56]拓步雄,李慧,车玉英.舒肝胶囊对实验性高脂血症及脂肪肝的影响.中医药研究, 1998, 14(5):41-43
[57]李金德,李峰.中西医结合治疗原发性高脂血症62例临床观察.北京中医, 1997(5):17-18
[58]张春荣,王平.中医辨证联合静点丹参?刺五加治疗高脂血症.实用中西医结合杂志, 1996, 9(7):440.
[59]高尚社.中风后郁证的辨证论治探析[J].中国中医基础医学杂志, 2005, 11(8):616.
[60]李慧杰,李德新.郁证之症因及证治规律探析[J].辽宁中医药大学学报, 2007, 9(3):24-25
[61]王威.不寐的证治探识[J].实用中医内科杂志, 2006, 20(1):39-40
[62]吕慰秋.顽固性失眠的治疗心法[J].山东中医杂志, 1999, 18(3):105-106
[63]徐凌云.女性失眠探讨[J].中国中医基础医学杂志, 2004, 10(1):75.
[64]方少琼.小儿失眠的病因病机的探讨[J].中国中医基础医学杂志, 1997, (3):23.
[65]武永刚.《伤寒杂病论》治疗失眠证七法辨析[J].中医药学报, 2004, 32(2):5-6
[66]安妮,马智.马智教授治疗不寐经验撷菁[J].时珍国医国药, 2008, 19(5):1288-1289
[67]夏隆庆,赵春霞.瘙痒的发生机制、分型和治疗[J].临床皮肤科杂志, 2003, 32(11):687-689
[68]沈柱,刘玉峰.瘙痒发生与治疗的研究进展[J].中华皮肤科杂志, 2003, 36(7):415-417
[69]尹冀源,秦晓娟,柴立等.瘙痒症患者血浆中阿片样肽的检测[J].临床皮肤科杂志, 2000, 29(6):340-341
[70]徐宜厚,王保方,张赛英.皮肤病中医诊疗学[M].北京:人民卫生出版社, 1997, 14-15
[71]陆德铭.中医外科学汇[M].上海:上海科学技术出版社, 1997, 18:120.
[72]张坤.皮肤痛痒症辨证论治浅析[J].新中医, 2004, 36(7):60.
[73]赵炳南.赵炳南临床经验集[M].第一版.北京:人民卫生出版社, 1974.
[74]刘艳平,刘艳杰.七味地黄益母汤治疗老年皮肤瘙痒症[J].湖北中医杂志, 2002, 24(8):44.
[75]谢贻志.克痒汤治疗皮肤瘙痒症30例[J].实用中医药杂志, 2002, 18(9):32.
[76]贺芳,蔡爱萍.中药治疗皮肤瘙痒58例[J].现代中西医结合杂志, 2000, 9(19):1939-1940
[77]陈达灿,国维.皮肤性病科专病中医临床诊治[M].北京:人民卫生出版社, 2002:281.
[78]江超,姜华静,柯黎黎.仙方活命饮治疗痤疮80例[J].山东中医杂志, 2001, 20(8):475.
[79]艾军.从温病辨治痤疮的体会[J].广西中医药, 1998, 21(4):42.
[80]贾喜花,唐汉钧,林立真.唐汉钧辨证分型治疗痤疮的经验[J].上海中医药杂志, 2002, (6):31-32
[81]徐学武.增液承气汤加味治疗寻常痤疮110例[J].湖北中医杂志, 1991, 13(1):18-19
[82]阎德文.浅谈痤疮的发病、辨证与分型诊断[J].深圳中西医结合杂志, 2000, 10(2):70-71
[83]金起风,周德英.中医皮肤病学[M].北京:中国医药科技出版社, 2000, 376.
[84]李艳萍,张友堂.痤疮病因病机探讨[J].中国中医基础医学杂志, 2004, 10(11):60.
[85]李明亮,杨柳,黄文婧等.基于杨柳教授4种证型分类的痤疮病因病机探讨[J].时珍国医国药, 2010, 21(2):456-457
[86]钱江,杨柳,兰海梅等.南方地区寻常痤疮中医临床流行病学调查分析[J].临床医学, 2006, 19(10):1807.
[87]王作顺.马连珍老中医治疗冠心病经验[J].中国中医急症, 2003, 12(2):145.
[88]李洁,解品启.从瘀论治冠心病心绞痛[J].中国中医急症, 2007, 16(2):179-180
[89]许从真.冠心病心绞痛辨证论治[J].浙江中西医结合杂志, 2003, 13(1):59.
[90]于厚波.冠心病心肌梗死的中医辨证施治[J].黑龙江医学, 2005, 29(6):479.
[91]黄永生.冠心病心绞痛临床诊治体会[J].长春中医学院学报, 2005, 21(1):16-17
[92]王文革,姚红芳.冠心病心绞痛的中医辨证分析[J].中西医结合心血管杂志, 2004, 2(12):730.
[93]袁建平,朱玉兰,周会丽.中医辨证加西药治疗冠心病心绞痛疗效观察[J].吉林中医, 2005, 25(7):47-48
[94]李幼勋,王德华.胸痹的病因病机及其证治述略[J].实用中医内科杂志, 2002, 16(4):184-185
[95]张敏州,邹旭,李新梅等.脚屏心痛证冠状动脉造影100例临床分析[J]浙江中西医结合杂志, 2001, 11(8):472-473
[96]徐瑛.中医辨证治疗冠心病60例[J].辽宁中医杂志, 2002, 29(7)409-410
[97]梁秀香.中药治疗胸痹心痛126例临床观察.北京中医, 1997, (3):23-25
[98]宋秀明.王左治疗冠心病的经脸.湖北中医杂志, 1999, 21(5):197.
[99]康路景.辨证论治冠心病心绞痛198例分析.内蒙古中医药, 1996, 15(2):5-7
[l00]高荣林等.路志正调理脾胃法治疗胸痹300例临床观察报告.中医杂志, 1996, 37(10):606.
[101]旷惠挑.冠心病心绞痛临床证型分类探讨.中医杂志, 1997, 38(12):782.
[102]国家中医药管理局.中药新药临床研究指导原则(试行)[M].北京:中国医药科技出版社, 2002:73-77
[103]秦建国,王亚红,粱晋普等.郭维琴治疗高血压病经验[J].中医杂志, 2007, 48(7):586-587
[104]顾月珍,刘旭玲.高血压病的辨证治疗[J].黑龙江中医药, 2007(2):27
[105]王丽艳.高血压病辨证论治探要[J].中医函授通讯, 1999, 18(5):32
[106]魏霞,穆广梅.高血压辨证体会[J].中国医药导报, 2004, 19(1):59
[107]吕伍文.中药治疗高血压病84例临床观察[J].中医药学报, 2004, 132(13):38
[108]张荣珍,陈志祥,胡素颖等. 3 578例高血压中医证型构成临床调查研究[J].中医药临床杂志, 2007, 19(4):360-364
[109]罗玉环,梁昌年.中医辨证分型治疗高血压病72例[J].安徽医药, 2008, 12(3):242-244
[110]王严冬,刘志龙,杨华伟.珠海地区300名高血压患者中医证型与不同类降压药效果分析[J].甘肃中医, 2008, 21(9):54-55
[111]武煦峰.辨证治疗高血压病120例[J].实用中西医结合临床, 2008, 8(2):26-26
[112]谭洁,滕久祥,彭芝配.湖南省家族性高血压病中医证型分布的流行病学调查研究[J].湖南中医药大学学报, 2009, 29(1):70-71
[113]刘福明,陈晓虎,杜午奇等.高血压病中医证型分布规律的临床研究[J].江苏中医药, 2009, 41(10):33-34
[114]吴世才.中医治疗原发性高血压137例的体会[J].河南学院学报, 2009, 15(11):1433-1434
[115]杨秀飞.百合安神治疗神经衰弱30例[J].实用中医内科杂志, 2002, 16(1):28.
[116]林秋兰.中医辨证施治职业因素引起的神经衰弱[J].职业与健康, 2001, 17(11):29.
[117]张赐安.中西医结合治疗神经衰弱70例[J].新中医, 2002, 34(7):61.
[118]李淑萍.针刺兴奋疗法治疗神经衰弱120例[J].中医药学刊, 2001, 18(1):81.
[119]周彬,巨春青.健脑宁神治疗神经衰弱68例[J].湖北中医杂志, 2002, 24(7):31.
[120]李彬,李岩,魏久贞.辨证分型治疗神经衰弱60例[J].山东中医杂志, 2002, 21(3):151.
[121]曹文胜.针药并用辨证治疗神经衰弱96例[J].针灸临床杂志, 2000, 16(11):5.
[122]孙明军,李海军.天麻钩藤饮加减治疗神经衰弱500例[J].河南中医药学刊, 1998, 13(4):52.
[123]梁煜,李逊,曾令芬.辨证治疗神经衰弱30例[J].实用中医药杂志, 1998, 14(11):12.
[124]吕品雅.神经衰弱的证与治[J].南京医学情报, 1998, 18(3):20.
[125]孙秀,于春雷,战惠娟等. 100例神经衰弱综合征的针刺及调护的探讨[J].中医药信息, 1998, 2:53.
[126]冯占元.神经衰弱中医诊断与疗效标准的探讨[J].山东中医杂志, 1997, 16(10):435.
[127]邓禄清.神经衰弱从肾治[J].广东医学, 1992, 13(3):138.
[128]朱仁康.朱仁康临床经验集--皮肤外科[M].北京:人民卫生出版社. 2005:144-150
[129]孙凤琴.许连霈教授辨证分型治疗皮肤瘙痒症经验[J].北京中医药大学学报, 2003, 10(1):31-32
[130]金强.中药为主治疗痤疮75例[J].浙江中医杂志, 1995, 30(9):391-391
[131]李元亚,刘瓦利.中药内服外用治疗寻常痤疮58例疗效分析[J].中级医刊, 1996, 31(11):54-55
[132]吴克永,张新民,沈自尹.辨证分型治疗寻常性痤疮234例[J].上海中医药杂志, 1998, 11(5):28-29
[133]张虹亚,刘峥,葛明等.辨证论治痤疮69例[J].安徽中医学院学报, 1997, 16(4):29-30
[134]赵志新.辨证治疗痤疮190例[J].河北中医, 2000, 22(4):172.
[135]孟涛.辨证治疗痤疮120例[J].陕西中医, 2001, 22(4):212.
[136]杨健芳. 148例痤疮中医辨证分型与临床疗效观察[J].湖南医科大学学报, 2001, 26(3):219.
[137]王上游.清肺法治疗青春期痤疮205例[J].浙江中医杂志, 1994, 29(9):421-422
[138]李征.辨证分型治疗寻常性痤疮136例疗效观察[J].中国皮肤性病学杂志, 1997, 11(5):294.
[139]游曦闽,辨证分型治疗痤疮83例[J].福建中医学院学报, 2001, 11(3):71.
[140]余方. 80例痤疮的中医分型与治疗[J].广西中医药, 1995, 18(4):5-6
[141]杨艳华中药辨证分型治疗痤疮80例[J].现代中西医结合杂志, 1999, 8(10):1642-1643
[142]李惠林.中医辨证治疗痤疮的体会[J].新中医11, 43(1):131-132
[1]秦长林.燥证琐谈[J].湖北中医杂志, 2001, 23(8):5-6.
[2]清·王冰.黄帝内经素问[M].北京:人民卫生出版社, 1963:499, 391.
[3]金·刘完素.素问玄机原病式(注释本)[M].北京:人民卫生出版社, 1983:206.
[4]孙莹,连博,杨梅.费伯雄治疗燥证经验探要[J].云南中医学院学报, 2009, 32(2):55-57.
[5]陈亦宜,陈涛.新安医家余国佩对燥邪的认识初探[J].中医药临床杂志, 2004, 16(6):598-599.
[6]王殿华.关于中医辛润法的理论思考[J].山东中医药大学学报, 1999, 38(8):493.
[7]张年顺.燥邪的阴阳属性争鸣概述[J].北京中医学院学报, 1990, 13(5):10-12.
[8]魏妮,孟凡冰,王宗柱.浅谈“燥”的阴阳属性[J].国医论坛, 2007, 22(6):9-10.
[9]林旭红.浅议燥邪的发病季节[J].时珍国医国药, 2006, 17(9):1804.
[10]牟全胜,周铭心.西北多燥说[J].新疆中医药, 1991, 9(4):1-6.
[11]周铭心,陶培勇.燥邪病因属性辨析[J].新疆中医药, 2005, 23(6):1-5.
[12]周铭心.西北燥证研究概述[J].上海中医药杂志, 2005, 39(11):43-45.
[13]周铭心,陶培勇.沙漠燥证初探——沙漠石油工人保健研究[J].中医杂志, 1997, 38(8):493-496.
[14]沙塔拉提,孙红友,周铭心.环境地理因素与亚健康状态的关系初探——西北燥证背景研究[J].新疆中医药, 2006, 24(4):74-76.
[15]周铭心,宋晓平,单丽娟,等.新疆各地不同民族居民西北燥证罹患情况流行病学调查分析[J].新疆医科大学学报, 2006, 29(11):1034-1038.
[16]王燕,周铭心.吴鞠通治燥特色探讨—西北燥证治法相关文献研究[J].新疆中医药, 2006, 24(4):1-3.
[17]明·李梃.医学入门[M].上海:上海锦章图书局印行本. 1930.
[18]连博,孙莹,杨梅.浅析喻嘉言《医门法律》燥证辨治之贡献[J].云南中医学院学报, 2009, 32(2):58-59.
[19]何廉臣.重印全国名医验案类编.福州:福建科技出版社, 2003:234.
[20]白君伟,梁苹茂.《医原》“燥?湿”论及其在2型糖尿病中的应用[J].中医杂志, 2008, 49(4):377-378.
[21]王秀莲.《医原》外感燥湿论探析[J].江苏中医, 2000, 21(11):6-8.
[22]余杨,张德新.从“冬三月,水冰地坼”浅谈辛润治燥法的运用[J].时珍国医国药, 2005, 16(1):80.
[23]何宽其.类燥证探讨[J].中医杂志, 2009, 50(6):569.
[24]丁建中,张六通,邱幸凡.外燥致病特点与辨证论治综议[J].时珍国医国药, 2006, 17(3):442-443.
[25]李民,路伟.阳生阴长法治燥浅识[J].湖北中医杂志, 2001, 23(1):20.
[26]周光,刘爱玲,周铭心.燥证证治八法解析[J].辽宁中医杂志, 2009, 36(3):360-361.