搜索页_Excitotoxicidad

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在“Elsevier”中，命中：669条，耗时：小于0.01 秒

1.p53 Mediates autophagy activation and mitochondria dysfunction in kainic acid-induced excitotoxicity in primary striatal neurons

作者：X.X. Dong^a ; ^b ; ¹ ; Y.R. Wang^a ; ¹ ; S. Qin^a ; Z.Q. Liang^a ; B.H. Liu^c ; Z.H. Qin^a ; Y. Wang^a ; ^{yanwang@suda.edu.cn}

关键词：p53 ; kainic acid ; excitotoxicity ; autophagy ; mitochondria

刊名：Neuroscience

出版年：2012

2.The effects of A1 and A2A adenosine receptor agonists on kainic acid excitotoxicity in the guinea pig cochlea

作者：Keiji Tabuchi ; ^{ktabuchi@md.tsukuba.ac.jp} ; Shuhei Sakai ; Masahiro Nakayama ; Bungo Nishimura ; Kentaro Hayashi ; Yuki Hirose ; Akira Hara

关键词：Excitotoxicity ; Adenosine A1 receptor ; Adenosine A2A receptor ; Cochlea ; Kainic acid (KA)

刊名：Neuroscience Letters

出版年：2012

3.DGK味 is degraded through the cytoplasmic ubiquitin-proteasome system under excitotoxic conditions, which causes neuronal apoptosis because of aberrant cell cycle reentry

作者：Masashi Okada^a ; Yasukazu Hozumi^a ; Toshiaki Tanaka^a ; Yusuke Suzuki^a ; Mitsuaki Yanagida^b ; Yoshihiko Araki^b ; Camilla Evangelisti^c ; ¹ ; Hitoshi Yagisawa^d ; Matthew K. Topham^e ; Alberto M. Martelli^c ; Kaoru Goto^a ; ^{kgoto@med.id.yamagata-u.ac.jp}

关键词：Diacylglycerol kinase ; Excitotoxicity ; Ubiquitin&ndash ; proteasome system ; Cell cycle reentry

刊名：Cellular Signalling

出版年：2012

4.The mechanism of axonal degeneration after perikaryal excitotoxic injury to the retina

作者：Natalie D. Bull^a ; ¹ ; Glyn Chidlow^d ; ^e ; ¹ ; John P.M. Wood^d ; ^e ; Keith R. Martin^a ; ^b ; ^c ; Robert J. Casson^d ; ^e ; ^{robert.casson@adelaide.edu.au}

关键词：Excitotoxicity ; Wallerian ; Neurodegeneration

刊名：Experimental Neurology

出版年：2012

5.Probucol modulates oxidative stress and excitotoxicity in Huntington's disease models in vitro

作者：Dirleise Colle^a ; ^{dirleise@yahoo.com.br} ; Juliana M. Hartwig^a ; Fé ; lix A. Antunes Soares^b ; Marcelo Farina^a ; ^{farina@ccb.ufsc.br}

关键词：DCF ; 2&prime ; 7&prime ; -dichlorofluorescein ; DCFH-DA ; 2&prime ; 7&prime ; -dichlorofluorescein diacetate ; DMEM ; Dulbecco's modified Eagle's medium ; DMSO ; dimethylsulfoxide ; HD ; Huntington's disease ; HEPES ; 4-(2-hydroxyethyl)-1-piperazineethanesulfonic acid ; K

刊名：Brain Research Bulletin

出版年：2012

6.NR2A and NR2B subunits differentially mediate MAP kinase signaling and mitochondrial morphology following excitotoxic insult

作者：Anthony M. Choo¹ ; Donna M. Geddes-Klein¹ ; Adam Hockenberry ; David Scarsella ; Mahlet N. Mesfin ; Pallab Singh ; Tapan P. Patel ; David F. Meaney ; ^{dmeaney@seas.upenn.edu}

关键词：NMDA receptor ; Calcium ; JNK ; ERK ; Mitochondria

刊名：Neurochemistry International

出版年：2012

7.Mesenchymal stem cells protect CNS neurons against glutamate excitotoxicity by inhibiting glutamate receptor expression and function

作者：A. Voulgari-Kokota^a ; R. Fairless^b ; ^d ; ¹ ; M. Karamita^a ; ¹ ; V. Kyrargyri^a ; ¹ ; V. Tseveleki^a ; M. Evangelidou^a ; B. Delorme^c ; P. Charbord^e ; R. Diem^b ; ^d ; L. Probert^a ; ^{lesley@pasteur.gr}

关键词：BDNF ; brain-derived neurotrophic factor ; bFGF ; basic fibroblast growth factor ; CNS ; central nervous system ; EAE ; experimental autoimmune encephalomyelitis ; GFAP ; glial fibrillary acidic protein ; GFP ; green fluorescent protein ; Iba1 ; ionized calcium bindin

刊名：Experimental Neurology

出版年：2012

8.Enhanced neurogenesis in organotypic cultures of rat hippocampus after transient subfield-selective excitotoxic insult induced by domoic acid

作者：A. Pé ; rez-Gó ; mez ; R.A. Tasker ; ^{tasker@upei.ca}

关键词：neurotoxicity ; neurogenesis ; NeuN ; GFAP ; AMPA/kainate ; dentate gyrus

刊名：Neuroscience

出版年：2012

9.Excitotoxic lesions of the medial amygdala attenuate olfactory fear-potentiated startle and conditioned freezing behavior

作者：Graham A. Cousens ; ^{gcousens@drew.edu} ; Amanda Kearns ; Francesco Laterza ; Jillian Tundidor

关键词：Fear conditioning ; Medial amygdala ; Freezing behavior ; Fear-potentiated startle

刊名：Behavioural Brain Research

出版年：2012

10.鈥楳ild mitochondrial uncoupling鈥?induced protection against neuronal excitotoxicity requires AMPK activity

作者：Petronela Weisová ; ^a ; ^b ; Ujval Anilkumar^a ; Caitriona Ryan^a ; Caoimhí ; n G. Concannon^a ; Jochen H.M. Prehn^a ; ^{jprehn@rcsi.ie} ; Manus W. Ward^a ; ¹

关键词：AICAR ; 5-amino-4-imidazolecarboxamide riboside ; AMP ; 5&prime ; -adenosine monophosphate ; AMPK ; 5&prime ; -adenosine monophosphate-activated protein kinase ; ANOVA ; analysis of variance ; ATP ; adenosine 5&prime ; -(tetrahydrogen triphosphate) ; BSA ; bovine serum al

刊名：Biochimica et Biophysica Acta (BBA)/Bioenergetics

出版年：2012

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