iTRAQ proteomic analysis of pancreatic cancer stem cells (Panc1 CSCs)
Identification of 230 differentially expressed proteins of Panc1 CSCs
Activation of glycolysis, pentose phosphate pathway, pyruvate-malate cycle, and lipid metabolism
Downregulation of Krebs cycle, spliceosome and non homologous end joining
Fatty acids synthesis and mevalonate pathways directly involved in Panc1 CSCs viability