Paralytic peptide activates insect humoral immune response via epidermal growth factor receptor
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- 作者：Liang Song songliang308@126.com" class="auth_mail" title="E-mail the corresponding author ; Fei Wang fwangswu@gmail.com" class="auth_mail" title="E-mail the corresponding author ; Shifeng Dong 1294935836@qq.com" class="auth_mail" title="E-mail the corresponding author ; Cuimei Hu hucuimeiuse@126.com" class="auth_mail" title="E-mail the corresponding author ; Xiaoting Hua huaxiaotingswu@126.com" class="auth_mail" title="E-mail the corresponding author ; Qingyou Xia ; xiaqy@swu.edu.cn" class="auth_mail" title="E-mail the corresponding author
- 关键词：PP ; paralytic peptide ; AMP ; anti-microbial peptide ; p38 MAPK ; p38 mitogen-activated protein kinase ; EGFR ; epidermal growth factor receptor ; RNAi ; RNA interference ; MAPKs ; mitogen-activated protein kinases ; GBP ; Growth-blocking peptide ; qRT-PCR ; quantitative RT-PCR ; FITC-PP ; FITC-labeled active PP
- 刊名：Peptides
- 出版年：2015
- 出版时间：September 2015
- 年：2015
- 卷：71
- 期：Complete
- 页码：20-27
- 全文大小：1629 K
文摘
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BmE cells can be used as a more sustainable and stable system to investigate PP activated signaling pathway in vitro.
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EGFR is involved in PP-induced expression of AMP genes.
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EGFR participates in PP-dependent activation of p38 MAPK.