The feeding responses evoked by endogenous cholecystokinin are regulated by different gastrointestinal sites
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Gastrointestinal sites where camostat influences ingestive behavior were examined.

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CCK-A receptor antagonist attenuated camostat-induced decreases in food intake.

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CCK-A receptor action in the celiac artery was necessary for camostat effects on meal size.

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CCK-A receptor action in the cardiac mesenteric artery was necessary for camostat effects on internal interval.

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Camostat affects different aspects of food intake by acting in different gastrointestinal sites.

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