文摘
Current treatment for glioblastoma fails to provide sufficient therapeutic outcomes. Overexpressed cyclooxygenase-2 (COX-2) contributes to the glioblastoma progression. COX-2 plays complex roles in glioma invasion, angiogenesis, immunosuppression, etc. COX-2 inhibitors sensitize glioblastomas to conventional chemo- and radio-therapies. COX-2 downstream signaling pathways might provide alternative targets for gliomas.