Prenatal binge-like alcohol exposure alters brain and systemic responses to reach sodium and water balance
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- 作者：A. Godinoa ; agodino@immf.uncor.edu" class="auth_mail" title="E-mail the corresponding author ; godinoandrea@gmail.com" class="auth_mail" title="E-mail the corresponding author ; P. Abatea ; c ; J.L. Amigoneb ; L. Vivasa ; d ; J.C. Molinaa ; c
- 关键词：5HT ; serotonergic system ; 5-HT-ir ; 5-HT immunoreactivity ; AP ; area postrema ; AVP ; vasopressinergic system ; BNSTL ; lateral division of the bed nucleus of the stria terminalis ; CD ; sham-depleted rats ; CeA ; central amygdaloid nucleus ; CVOs ; circumventricular organs ; DAB ; diaminobenzidine hydrochloride ; DRN ; dorsal raphe nucleus ; DRV ; ventral subdivisions of DRN ; Fra-LI ; Fra like immunoreactivity ; FURO ; Furosemide ; FURO ; + ; LSD ; Furosemide and low-sodium diet ; GD ; gestational day ; LPBN ; the
- 刊名：Neuroscience
- 出版年：2015
- 出版时间：17 December 2015
- 年：2015
- 卷：311
- 期：Complete
- 页码：92-104
- 全文大小：2419 K
文摘
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Prenatal ethanol exposures affect homeostatic responses after body sodium depletion.
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The 5HT – DRN system is chronically affected by prenatal ethanol exposure.
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The AVP hypothalamic neurons are chronically activated by prenatal ethanol exposure.
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Central extended Amygdala subnucleuses are chronically activated by prenatal ethanol exposure.
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Prenatal ethanol exposure increases the preference for alcohol during adolescence.