Ligularia fischeri and its constituent 3,4-dicaffeoylquinic acid improve obesity-induced nonalcoholic fatty liver disease by regulating lipid metabolism and activating AMPK
Ligularia fischeri suppressed lipid accumulation and activated AMPK in HepG2 cells. L. fischeri inhibited liver injury and steatosis in HFD-induced mice L. fischeri reduced oxidative stress and insulin resistance in HFD-induced mice