Elevating hippocampal H2S ameliorates cognitive deficits in APP/PS1 mice.
H2S elevation restores hippocampal NMDAR-dependent LTP in APP/PS1 mice.
Elevation of H2S restores synaptic GluN2B expression and response in APP/PS1 mice.
Elevation of H2S enhances NMDAR-dependent signaling in APP/PS1 mice.
Blockade of GluN2B abolishes the beneficial effects of H2S elevation in APP/PS1 mice.