Higal-IgG1 ICs inhibits C5a-mediated inflammation via FcγRIIB-Dectin-1 complexes.
Galectin-3 binds murine IgG1, depending on Fc-galactosylation.
Regulation of C5a-induced neutrophil chemotaxis via IgG1-IC depends on Galectin-3.
An interaction of Galectin-3 with FcγRIIB-Dectin-1 receptor complexes is proposed.