Inwardly rectifying potassium channel 4.1 expression in post-traumatic syringomyelia
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- 作者：E. Najafia ; &dagger ; ; atieh.najafi@students.mq.edu.au" class="auth_mail" title="E-mail the corresponding author ; M.A. Stoodleya ; &Dagger ; ; marcus.stoodley@mq.edu.au" class="auth_mail" title="E-mail the corresponding author ; L.E. Bilstonb ; c ; ¶ ; ; l.bilston@neura.edu.au" class="auth_mail" title="E-mail the corresponding author ; S.J. Hemleya ; sarah.hemley@mq.edu.au" class="auth_mail" title="E-mail the corresponding author
- 关键词：AQP4 ; aquaporin 4 ; CSF ; cerebrospinal fluid ; DTT ; dithiothreitol ; EGTA ; ethylene glycol-bis (2-aminoethylether)-N ; N ; N&prime ; N&prime ; -tetraacetic acid ; GFAP ; glial fibrillary acidic protein ; HEPES ; 4-(2-hydroxyethyl)piperazine-1-ethanesulfonic acid ; KCl ; potassium chloride ; Kir4.1 ; inwardly rectifying potassium channel 4.1 ; NDS ; normal donkey serum ; PMSF ; phenylmethylsulfonyl fluoride ; PTS ; post-traumatic syringomyelia ; TPBS ; tris phosphate-buffered saline
- 刊名：Neuroscience
- 出版年：2016
- 出版时间：11 March 2016
- 年：2016
- 卷：317
- 期：Complete
- 页码：23-35
- 全文大小：5528 K
文摘
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Kir4.1 protein expression is altered in a model of post-traumatic syringomyelia.
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Kir4.1 protein expression decreased around the syrinx cavity at 3 days and 6 weeks.
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There is a population of Kir4.1-negative astrocytes surrounding the syrinx cavity.