Comparisons of NSCs and derivative GSCs reveal elevated WNT5A and EC signature
PAX6/DLX5 bidirectionally regulates WNT5A during differentiation of GSCs into GdECs
WNT5A-mediated GdEC differentiation and EC recruitment support GSC invasive growth
Clinical studies of peritumoral/recurrent GBM reveal increased WNT5A/GdEC expression