刊名:Biochemical and Biophysical Research Communications
出版年:2017
出版时间:15 January 2017
年:2017
卷:482
期:3
页码:388-398
全文大小:1866 K
卷排序:482
文摘
Either insufficient or excess manganese activates mitochondria-mediated cell death. The optimal healthy Mn exposure window is very narrow. Mitochondrial H2O2 production depends on Mn across physiologic to toxicologic range. Integrative omics needed to understand complex Mn interaction in cell fate. Mn is central to redox interface between an organism and its environment.