文摘
Cucurbitacin-I, a triterpenoids, has diverse pharmacological and biological activities. Cucurbitacin-I induced cardiotoxicity (hypertrophy and death) in H9c2 cells. Cucurbitacin-I induced strong autophagy, hypertrophy and apoptosis in H9c2 cells. Cucurbitacin-I induced cardiotoxicity via an ERK-autophagy dependent pathway.