Mechanisms of abscisic acid-mediated control of stomatal aperture
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文摘

ABA triggers a robust signal network that controls stomatal closing.

Guard cell ABA levels are controlled by biosynthesis, catabolism, and transport.

SnRK2s and CPKs are key for ABA activation of anion channels in guard cells.

PP2Cs downregulate both Ca2+-independent and Ca2+-dependent ABA signaling branches.

Multiple biotic signal inputs target ABA signaling in guard cells.

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