AS-1 attenuates mechanical stress-induced decreases of LATS1 in cardiac fibroblasts and in vivo.
AS-1 inhibits mechanical stress-induced cardiac fibroblast activity via LATS1.
AS-1 inverses cardiomyocyte hypertrophy induced by fibroblast paracrine and LATS1 modulates pro-hypertrophic signal.
AS-1 ameliorates pressure overload-induced cardiac fibrosis and heart failure.