Importance of the central nucleus of the amygdala on sodium intake caused by deactivation of lateral parabrachial nucleus
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- 作者：Glá ; ucia M.F. Andrade-Franzé ; Carina A.F. Andrade ; Silvia Gasparini ; Laurival A. De Luca Jr ; Patrí ; cia M. De Paula ; Dé ; bora S.A. Colombari ; Eduardo Colombari ; José ; V. Menani ; menani@foar.unesp.br" class="auth_mail" title="E-mail the corresponding author
- 关键词：ANG II ; angiotensin II ; CAP ; captopril ; CeA ; central nucleus of amygdala ; FURO ; furosemide ; GABA ; γ-aminobutyric acid ; LPBN ; lateral parabrachial nucleus ; NTS ; nucleus of the solitary tract ; s.c. ; subcutaneously
- 刊名：Brain Research
- 出版年：2015
- 出版时间：2 November 2015
- 年：2015
- 卷：1625
- 期：Complete
- 页码：238-245
- 全文大小：750 K
文摘
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Sodium satiety is controlled by central amygdala and lateral parabrachial nucleus.
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Sodium intake to lateral parabrachial deactivation depends on central amygdala.
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Facilitation sodium intake by central amygdala is inhibited by parabrachial nucleus.
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Deactivation of the lateral parabrachial nucleus disrupts sodium satiety.
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CeA facilitatory and LPBN inhibitory mechanisms interacts to control of NaCl intake.