SGT1 contributes to maintaining protein levels of MEK2DD to facilitate hypersensitive response-like cell death in Nicotiana benthamiana
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文摘

SGT1 is required for HR-like cell death induced by MEK2DD.

Silencing SGT1 reduces protein levels of MEK2DD, but not MEK2WT.

SKP1 is not required for HR-like cell death induced by MEK2DD.

SGT1 may have novel functions for maintaining protein levels of MEK2 active form other than those already described.

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