Chromatin remodeling regulates catalase expression during cancer cells adaptation to chronic oxidative stress
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- 作者：Christophe Glorieuxa ; b ; christophe@sysucc.org.cn" class="auth_mail" title="E-mail the corresponding author ; Juan Marcelo Sandovala ; c ; Antoine Fattacciolid ; Nicolas Dejeansa ; 1 ; James C. Garbee ; Marc Dieuf ; Julien Verraxa ; Patricia Renardd ; Peng Huangb ; g ; Pedro Buc Calderona ; c ; pedro.buccalderon@uclouvain.be" class="auth_mail" title="E-mail the corresponding author
- 关键词：AP-1 ; activator protein 1 ; APEX1 ; ; DNA-(apurinic or apyrimidinic site) lyase ; AP-MS ; ; affinity purification followed by mass spectrometry ; ARID3A ; ; AT-rich interactive domain-containing protein 3A ; Asc/Men ; ; ascorbate/menadione ; ATRA ; ; all-trans-Retinoic acid ; EF1 ; ; elongation factor 1 ; DBHS ; ; Drosophila behavior/human splicing ; Fosl2 ; ; Fos-related antigen 2 ; Fox ; ; forkhead box protein ; FUBP ; ; far upstream element-binding protein ; GAPDH ; ; glyceraldehyde 3-phosphate dehydrogenase ; GATA
- 刊名：Free Radical Biology and Medicine
- 出版年：2016
- 出版时间：October 2016
- 年：2016
- 卷：99
- 期：Complete
- 页码：436-450
- 全文大小：3560 K
文摘
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Cellular adaptation to chronic oxidative stress induces catalase expression.
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RARα and JunB factors control catalase expression in breast cancer cells.
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Chromatin remodeling plays an important role in the control of catalase expression.