Electrophysiology in Fisher syndrome

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• 作者：Satoshi Kuwabara ; ^{kuwabara-s@faculty.chiba-u.jp} ; Yukari Sekiguchi ; Sonoko Misawa
• 关键词：DRG ; dorsal root ganglion ; FS ; Fisher syndrome ; GBS ; Guillain&ndash ; Barré ; syndrome ; NCS ; nerve conduction study ; SEP ; somatosensory-evoked potential ; SNAP ; sensory nerve action potential
• 刊名：Clinical Neurophysiology
• 出版年：2017
• 出版时间：January 2017
• 年：2017
• 卷：128
• 期：1
• 页码：215-219
• 全文大小：206 K
• 卷排序：128

文摘

In Fisher syndrome (FS), ophthalmoplegia, ataxia, and areflexia are caused by autoantibodies against ganglioside GQ1b. Major physiologic abnormalities are absent H-reflexes and impaired proprioception in posturography. GQ1b-expressed in ocular motor nerves and dorsal root ganglion 1a neurons represent a target molecule for FS.