Sensory-Derived Glutamate Regulates Presynaptic Inhibitory Terminals in Mouse Spinal Cord
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- 作者：Michael Mende1 ; 13 ; 14 ; Emily V. Fletcher2 ; 3 ; 13 ; Josephine L. Belluardo1 ; 6 ; 13 ; Joseph P. Pierce7 ; Praveen K. Bommareddy1 ; Jarret A. Weinrich1 ; 8 ; Zeeba D. Kabir7 ; 9 ; Kathryn C. Schierberl7 ; 9 ; John G. Pagiazitis2 ; 3 ; Alana I. Mendelsohn4 ; Anna Francesconi10 ; Robert H. Edwards11 ; Teresa A. Milner7 ; 12 ; Anjali M. Rajadhyaksha7 ; 9 ; Peter J. van Roessel5 ; George Z. Mentis2 ; 3 ; gzmentis@columbia.edu" class="auth_mail" title="E-mail the corresponding author ; Julia A. Kaltschmidt1 ; 6 ; 8 ; kaltschj@mskcc.org" class="auth_mail" title="E-mail the corresponding author
- 刊名：Neuron
- 出版年：2016
- 出版时间：15 June 2016
- 年：2016
- 卷：90
- 期：6
- 页码：1189-1202
- 全文大小：3685 K
文摘
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Retrograde signals mediate excitatory/inhibitory balance in a spinal reflex circuit
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Glutamatergic sensory activity directs GABAergic interneuron synapse differentiation
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Glutamate and BNDF collaborate hierarchically to regulate presynaptic GAD65 and GAD67
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Presynaptic group I mGluR regulates GABAergic efficacy of an interneuron synapse