Breast regression protein-39 is not required for experimental autoimmune encephalomyelitis induction
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- 作者：Ester Cantó ; 1 ; Carmen Espejo ; 1 ; carmen.espejo@vhir.org" class="auth_mail" title="E-mail the corresponding author ; Carme Costa ; Xavier Montalban ; Manuel Comabella ; manuel.comabella@vhir.org" class="auth_mail" title="E-mail the corresponding author
- 关键词：AMCase ; acidic mammalian chitinase ; BRP-39 ; breast regression protein 39 ; CHI3L1 ; chitinase 3-like 1 ; CHI3L3 ; chitinase 3-like 3 ; CHIT-1 ; chitotriosidase ; CIS ; clinically isolated syndrome ; CNS ; central nervous system ; EAE ; experimental autoimmune encephalomyelitis ; GFAP ; glial fibrillary acidic protein ; HE ; hematoxylin and eosin ; KB ; Klü ; ver&ndash ; Barrera ; KO ; knock-out ; LEA ; lycopersicum esculentim agglutinin ; LPS ; lipopolysaccharide ; MOG ; myelin oligodendrocyte glycoprotein ; MS ; multiple
- 刊名：Clinical Immunology
- 出版年：2015
- 出版时间：October 2015
- 年：2015
- 卷：160
- 期：2
- 页码：133-141
- 全文大小：1445 K
文摘
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BRP-39 expression is increased during the inflammatory phase in EAE.
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Absence of BRP-39 does not modify EAE disease course.
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Members of the 18 glycosyl hydrolase family are differentially expressed in EAE.