MiR-367 was aberrantly upregulated in sera and tumors of ESCC patients.
MiR-367 was downregulated in ESCC patients after esophagectomy or chemotherapy.
Serum miR-367 was correlated with the clinicopathological properties of ESCC patients.
Serum miR-367 was associated with OS in ESCC patients.
lentivirus-induced miR-367 downregulation inhibited ESCC growth and cell cycle transition.