Effects of zinc oxide nanoparticles on human coronary artery endothelial cells
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- 作者：Kai-Jen Chuanga ; b ; 1 ; kjc@tmu.edu.tw" class="auth_mail" title="E-mail the corresponding author ; Kang-Yun Leec ; d ; 1 ; kangyunlee68@gmail.com" class="auth_mail" title="E-mail the corresponding author ; Chih-Hong Pane ; f ; chpan@mail.iosh.gov.tw" class="auth_mail" title="E-mail the corresponding author ; Ching-Huang Laif ; lgh@ndmctsgh.edu.tw" class="auth_mail" title="E-mail the corresponding author ; Lian-Yu Ling ; hspenos@yahoo.com.tw" class="auth_mail" title="E-mail the corresponding author ; Shu-Chuan Hoc ; h ; shu-chuan@tmu.edu.tw" class="auth_mail" title="E-mail the corresponding author ; Kin-Fai Hoi ; j ; kfho@cuhk.edu.hk" class="auth_mail" title="E-mail the corresponding author ; Hsiao-Chi Chuangc ; h ; r92841005@ntu.edu.tw" class="auth_mail" title="E-mail the corresponding author
- 关键词：AGTR1 ; Inflammation ; Nanoparticles ; Nitric oxide ; Oxidative stress
- 刊名：Food and Chemical Toxicology
- 出版年：2016
- 出版时间：July 2016
- 年：2016
- 卷：93
- 期：Complete
- 页码：138-144
- 全文大小：1315 K
文摘
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Effects of ZnONP were determined in HCEAC.
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Alteration in 8-OHdG, IL-6 and NO occurred by ZnONPs.
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AGTR1 is an important gene in response to ZnONP exposure.
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Calcium signalling pathway is important after ZnONPs exposure.
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ZnONP is associated with coronary artery endothelial cell dysfunction.