Detailed analysis of sulfur and nitrogen mustard-induced PARylation in HaCaT cells
PARylation participates in mustard-induced stress response in a substance-specific manner
PARP inhibitors potentiate CEES-induced genome instability
With limitations, CEES and HN2 can be used as surrogates to study SM-related toxicity
PARP inhibitors should be considered with caution as SM antidotes, due to potential long-term effects