ULK1/2 deficiency leads to reduction of glycolysis during nutritional stresses
HK1, PFK1, FBP1, and ENO1 are direct substrates of the ULK1 kinase
ULK1 regulates activities of glycolytic enzymes and sustains glycolysis and PPP flux
ULK1/2 reprogram glucose metabolic fluxes independently of autophagy