Les angiœdème héréditaires de type III : nouvelle maladie ou nouveau diagnostic ?
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摘要
Current knowledge and key points. – Diseases with a clinical profile close to hereditary angioneurotic edema, but without C1Inh anomaly, have been described recently. It is in fact family cases, concerning only women, where estrogens seem to play a dominant role. Angioedema’s secondary aspects are gathering various pathologies (vasculitis, Gleich’s syndrome, angioedema initiated by physical agents). The role played by some drugs must not be forgotten, mainly angiotensin converting enzyme inhibitors, which are at the origin of angiodema in nearly 0.5%of users.

Future prospect and projects. – Uncontrolled activation of the contact system seems to play a major role in the main part of these angiodemas. The efficiency of the tranexaminic acid (which modulates its activation) is to be taken as evident. The key to the future seems to be the development of plasmin and bradykinin inhibitors.div>div>
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La Revue de Médecine InterneVolume 24, Issue 12December 2003, Pages 838-840
L. Bouillet

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La Revue de Médecine InterneVolume 27, Supplement 3December 2006, Page S350
L. Bouillet, D. Ponard, C. Drouet, H. Rousset, S. Cichon

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Les angiœdème héréditaires de type III : nouvelle maladie ou nouveau diagnostic ?

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