胸膜粘连发生机制的研究进展
摘要
胸膜粘连是呼吸内科临床常见病、多发病,其发病机制较复杂,常见的如肺炎、肺结核、胸膜炎及胸部损伤后引发。近年来认为胸膜炎症反应、细胞因子、胸膜局部凝血机制异常在胸膜粘连的发生机制中起着极其重要的作用,但石棉纤维、结核性胸膜炎、类风湿关节炎、冠状动脉搭桥术导致胸膜粘连有其独特的发病机制。下面主要阐述胸膜粘连发生机制的研究进展。
引文
[1]王欢.胸膜纤维化机制的研究进展[J].中国卫生标准管理,2016,7(08):176-177.
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[7]Coxon A,Tang T,Mayadas TN.Cytokine-activated endothelial cells delay neutrophil apoptosis in vitro and in vivo.A role for granulocyte/macrophage colony-stimulating factor[J].J Exp Med,1999,190(7):923-934.
[8]Jonjic N Peri G,Bernasconi S,et al.Expression of adhesion molecules and chemotacticcytokinesin culturedhumanmesothelial[J].JExp Med,1992,176(4):1165-1174.
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[10]LightRW,ChengDS,LeeYC,etal.Asingleintrapleural injectionof transforming growthfactorβ2 producesanexcellent pleurodesisin rahhits[J]. Am J Respir Crit Care Med, 2000,162:98-104.
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[14]Bannai M,Tokunage K,Tanaka H,et al.Five HLA-B22 group alleles in Japanese[J].Tissue Antigens,1997,49(4):376-382.
[15]Myoung H,Young W,Ho I,et al,Association of HLA class I antigens with diffuse panbrochiolitis in korean patients[J].Am J Respir Crit Care Med,1999,159(2):526-529.
[16]Philip-Joet F,Ales i MC,Philip-Joet C,et al.Fibrinolytic and inf lamma-tory proces esin pleural effusions[J].EurRespir J,1995,8:1352-1356.
[17]Rodriguez-Panadero F,Segado A,M artin Juan J,et al.Failure of talc pleurodesis isassociated with increasedpleuralfibrinolysis[J].AmJRespir Crit Care M ed,1995,151:785-790.
[18]Idell S,M azar A,Bitterman P,et al.Fibrin Turnover inLung Inflammation and Neoplasia[J].AmJRespir Crit Care Med,2001,163:578-584.
[19]Huggins JT,Sahn SA.Causes and management of pleural fibrosis[J].Respirology,2004,9(4):441-447.
[20]AKERBERG D,POSARIC-BAUDEN M,ISAKSSON K, et al.Prevension ofpleuraladhesion by bioactive polypeptides-apilot study[J].International Journal of Medical Sciences,2013,10(12):1720-1726.
[21]舒敬奎,刘凌,张剑青,等.内科胸腔镜下结核性胸腔积液胸膜粘连程度分级及影响因素分析[J].中国内镜杂志,2016,22(05):1-5.
[22]Tomita Y,Hashimoto S,Shimizu T,et al.Restriction fragment length polymorphism analysis HLA class IIIgenes of patients with diffuse panbronchiolitis[J].Intern Med,1996,35(9):693-697.
[2]Nagai K,et al. A randomized trial comparingadjuvant chemotherapy versus surgery alone forcompletely resected pN2 non-small cell lung cancer(JCOG9304)[J].Lung Cancer,2004,44:167.
[3]谢灿茂,RichardW.Light.滑石粉浆对兔胸膜固定术的急性反应[J].中山医科大学学报,2000(01):26-28+31.
[4]Whitaker JT,Sahn SA.Causes and management of pleural fibrosis[J].Respirology,2004,9(4):441-447.
[5]Yano S,Shinoajara,Herpst RS,et al.Production of experimental malignant pleural effusion is dependent on invasion of the pleura and expression of vascular endothelial growth factor/vascular permeability factor by human lung cancer cells[J].Am J Pathol,2000,157(6):898-903.
[6]Idell S.Coagulation,fibrinolysis and fibrin deposition in lung injury and repair[J].Pulmonary Fribrosis,1995,80(4):743-776.
[7]Coxon A,Tang T,Mayadas TN.Cytokine-activated endothelial cells delay neutrophil apoptosis in vitro and in vivo.A role for granulocyte/macrophage colony-stimulating factor[J].J Exp Med,1999,190(7):923-934.
[8]Jonjic N Peri G,Bernasconi S,et al.Expression of adhesion molecules and chemotacticcytokinesin culturedhumanmesothelial[J].JExp Med,1992,176(4):1165-1174.
[9]王居平,李月川,张虹.胸膜粘连术粘连形成机制的研究进展[J].河北医药,2009,31(03):329-330.
[10]LightRW,ChengDS,LeeYC,etal.Asingleintrapleural injectionof transforming growthfactorβ2 producesanexcellent pleurodesisin rahhits[J]. Am J Respir Crit Care Med, 2000,162:98-104.
[11]李海明,张华茹.结核性胸膜炎胸液中纤维蛋白原含量对胸膜肥厚及粘连形成的影响[J].中国防痨杂志,2004(04):26-27+30.
[12]Lappi-Blanco E,Soini Y,Kinnula V,et al.VEGF and bFGF are highly expressed in intraluminal fibromyxiod lesions in bronchiolitis obliterans organizing pneumonia[J]. J Pathol,2002;196(2):220.
[13]AntonyVB,NasreenN,MohammedKA,etal.Talcpleurodesis:basicfibroblastgrowthfactormediatespleuralfibrosis[J].Chest,2004,126:1522-1528.
[14]Bannai M,Tokunage K,Tanaka H,et al.Five HLA-B22 group alleles in Japanese[J].Tissue Antigens,1997,49(4):376-382.
[15]Myoung H,Young W,Ho I,et al,Association of HLA class I antigens with diffuse panbrochiolitis in korean patients[J].Am J Respir Crit Care Med,1999,159(2):526-529.
[16]Philip-Joet F,Ales i MC,Philip-Joet C,et al.Fibrinolytic and inf lamma-tory proces esin pleural effusions[J].EurRespir J,1995,8:1352-1356.
[17]Rodriguez-Panadero F,Segado A,M artin Juan J,et al.Failure of talc pleurodesis isassociated with increasedpleuralfibrinolysis[J].AmJRespir Crit Care M ed,1995,151:785-790.
[18]Idell S,M azar A,Bitterman P,et al.Fibrin Turnover inLung Inflammation and Neoplasia[J].AmJRespir Crit Care Med,2001,163:578-584.
[19]Huggins JT,Sahn SA.Causes and management of pleural fibrosis[J].Respirology,2004,9(4):441-447.
[20]AKERBERG D,POSARIC-BAUDEN M,ISAKSSON K, et al.Prevension ofpleuraladhesion by bioactive polypeptides-apilot study[J].International Journal of Medical Sciences,2013,10(12):1720-1726.
[21]舒敬奎,刘凌,张剑青,等.内科胸腔镜下结核性胸腔积液胸膜粘连程度分级及影响因素分析[J].中国内镜杂志,2016,22(05):1-5.
[22]Tomita Y,Hashimoto S,Shimizu T,et al.Restriction fragment length polymorphism analysis HLA class IIIgenes of patients with diffuse panbronchiolitis[J].Intern Med,1996,35(9):693-697.