Interactive effects of hypoxia, carbon monoxide and acute lung injury on oxygen transport and aerobic capacity
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文摘

Hypoxia with acute lung injury attenuated reductions in VO2max compared to the predicted additive effects of the treatments.

Elevated FHbCO administered in combination with impaired pulmonary gas exchange tended to attenuate the decrease in VO2max.

Acute lung injury with hypoxia and elevated FHbCO reduced VO2max by attenuated O2 delivery despite decreased O2 extraction.

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