Hypoxia with acute lung injury attenuated reductions in VO2max compared to the predicted additive effects of the treatments.
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Elevated FHbCO administered in combination with impaired pulmonary gas exchange tended to attenuate the decrease in VO2max.
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Acute lung injury with hypoxia and elevated FHbCO reduced VO2max by attenuated O2 delivery despite decreased O2 extraction.
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