Brassinosteroids (BR) modulate the general stress response (GSR).
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Exogenously applied BR reduces flg22-, but enhances wound-induced GSR.
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Wound-activated GSR in BR-treated plants heightens resistance to Botrytis cinerea.
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Point of BR-GSR interaction is downstream of membrane receptor circuitry.
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Multiple interaction points between BRs and stress signaling tailor plant responses.
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