Ephrin-Bs Drive Junctional Downregulation and Actin Stress Fiber Disassembly to Enable Wound Re-epithelialization

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Ephrin-Bs Drive Junctional Downregulation and Actin Stress Fiber Disassembly to Enable Wound Re-epithelialization

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作者：Robert Nunan¹ ; Jessica Campbell¹ ; Ryoichi Mori¹ ; ² ; Mara E. Pitulescu³ ; ⁴ ; Wen G. Jiang⁵ ; Keith G. Harding⁵ ; Ralf H. Adams³ ; ⁴ ; Catherine D. Nobes¹ ; ⁶ ; Paul Martin¹ ; ⁵ ; ⁶ ; ^{paul.martin@bristol.ac.uk" class="auth_mail" title="E-mail the corresponding author}
刊名：Cell Reports
出版年：2015
出版时间：17 November 2015
年：2015
卷：13
期：7
页码：1380-1395
全文大小：9690 K

文摘

•: Ephrin-B/EphBs are upregulated in the migrating wound epidermis in mouse and man
•: Ephrin-B/EphB signaling drives junction loosening, thus enabling re-epithelialization
•: Ephrin-B/EphB signaling also leads to dissolution of stress fibers and tension release
•: In human chronic wounds ephrin-Bs are misregulated and may be a therapeutic target

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