Physical exercise, reactive oxygen species and neuroprotection

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Physical exercise, reactive oxygen species and neuroprotection

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作者：Zsolt Radak^a ; ^b ; ^{radak@tf.hu" class="auth_mail" title="E-mail the corresponding author} ; Katsuhiko Suzuki^b ; Mitsuru Higuchi^b ; Laszlo Balogh^c ; Istvan Boldogh^d ; Erika Koltai^a
关键词：(AP-1) ; activator protein 1 ; (KGDHC) ; α-ketoglutarate dehydrogenase ; (AD) ; Alzheimer&rsquo ; s disease ; (APE1) ; apurinic/apyrimidinic endonuclease 1 ; (BER) ; base excision repair ; (iPLA) ; Ca(2+)-independent phospholipase A(2) ; (CREB) ; cyclic AMP response element-binding protein ; (EPR) ; electron paramagnetic resonance ; (ERK) ; extracellular-signal-regulated kinases ; (FOXO) ; forkhead box O ; (IGF-1) ; insulin like growth-factor-1 ; (IL-1) ; interleukin-1 ; (IL-6) ; interleukin-6 ; (IL-8) ; interleukin-8 ; (IFN
刊名：Free Radical Biology and Medicine
出版年：2016
出版时间：September 2016
年：2016
卷：98
期：Complete
页码：187-196
全文大小：2781 K

文摘

•: Exercise regulates the cellular redox state of the brain.
•: ROS are involved in the self-renewal and differentiation of neuronal stem cells.
•: Base excision repair enzymes mediate neuroprotective effects.
•: The neuroprotective effects of exercise are partly due to redox-associated adaptation.

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