Postnatal dexamethasone-induced programmed hypertension is related to the regulation of melatonin and its receptors

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Postnatal dexamethasone-induced programmed hypertension is related to the regulation of melatonin and its receptors

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作者：Hsin-Yu Chang^a ; You-Lin Tain^b ; ^{tainyl@hotmail.com" class="auth_mail" title="E-mail the corresponding author}
关键词：DEX ; dexamethasone ; HDAC ; histone deacetylase ; MEL ; melatonin ; MT1 ; melatonin receptor-1 ; MT2 ; melatonin receptor-2 ; RORα ; retinoid acid receptor-α ; RZRβ ; retinoid Z receptor-β
刊名：Steroids
出版年：2016
出版时间：April 2016
年：2016
卷：108
期：Complete
页码：1-6
全文大小：713 K

文摘

•: Postnatal dexamethasone (DEX) induced programmed hypertension in later life.
•: Maternal melatonin therapy prevented DEX-induced programmed hypertension.
•: Postnatal DEX decreased renal MT2 protein level, which melatonin prevented.
•: Melatonin therapy increased levels of RORα protein and melatonin in the kidney.
•: Postnatal DEX-induced programmed hypertension is related to melatonin receptors.

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