Glial cell response after aneurysmal subarachnoid hemorrhage — Functional consequences and clinical implications

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Glial cell response after aneurysmal subarachnoid hemorrhage — Functional consequences and clinical implications

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作者：Bart J. van Dijk^a ; ^b ; Mervyn D.I. Vergouwen^b ; Myrna M. Kelfkens^a ; Gabriel J.E. Rinkel^b ; Elly M. Hol^a ; ^c ; ^d ; ^{e.m.hol-2@umcutrecht.nl" class="auth_mail" title="E-mail the corresponding author}
关键词：ADAMTS-13 ; a disintegrin and metalloproteinase with a thrombospondin type 1 motif ; member 13 ; AQP1/4 ; Aquaporin 1/4 ; BBB ; Blood Brain Barrier ; BK ; Big Potassium ; CSD ; cortical spreading depression ; CSF ; cerebrospinal fluid ; DCI ; delayed cerebral ischemia ; EBI ; early brain injury ; ERK1/2 ; Extracellular signal-regulated protein kinases 1 and 2 ; ET-1 ; Endothelin-1 ; GABA ; γ-aminobutyric acid ; GFAP ; Glial fibrillary acidic protein ; Gln ; Glutamine ; GLT-1 ; Glutamate Transporter-1 ; GSN ; Gelsolin ; HMGB1
刊名：Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
出版年：2016
出版时间：March 2016
年：2016
卷：1862
期：3
页码：492-505
全文大小：1104 K

文摘

•: Aneurysmal subarachnoid hemorrhage (SAH) is a severe type of stroke.
•: SAH can cause early brain damage, delayed cerebral ischemia and cognitive impairment.
•: Glial response after SAH is both beneficial and detrimental for brain functioning.
•: Sustained glial response may contribute to cognitive impairment after SAH.

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