For whom the endocannabinoid tolls: Modulation of innate immune function and implications for psychiatric disorders

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For whom the endocannabinoid tolls: Modulation of innate immune function and implications for psychiatric disorders

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作者：Rebecca J. Henry^a ; ^c ; ¹ ; Daniel M. Kerr^a ; ^b ; ^c ; ¹ ; David P. Finn^b ; ^c ; Michelle Roche^a ; ^c ; ^{Michelle.roche@nuigalway.ie" class="auth_mail" title="E-mail the corresponding author}
关键词：2-AG ; 2-arachidonyl glycerol ; AEA ; anandamide ; CB ; cannabinoid receptor ; CBD ; cannabidiol ; COX-2 ; cyclooxygenase 2 ; ERK1/2 ; extracellular signal-regulated kinase ½ ; FAAH ; fatty acid amide hydrolase ; IFN ; interferon ; IL ; interleukin ; iNOS ; inducible nitric oxide synthase ; IP10 ; interferon gamma-induced protein 10 ; IRF ; interferon regulatory transcription factor ; LPS ; lipopolysaccharide ; MAGL ; monoacylglycerol lipase ; MAPK ; mitogen-activated protein kinase ; NF-魏B ; nuclear factor kappa B ; NO
刊名：Progress in Neuropsychopharmacology & Biological Psychiatry
出版年：2016
出版时间：4 January 2016
年：2016
卷：64
期：Complete
页码：167-180
全文大小：929 K

文摘

•: The endocannabinoid (EC) system modulates TLR-induced inflammation.
•: 160">Paucity of data on EC effects on TLR immune responses apart from TLR4
•: Psychiatric disorders associated with inflammation and enhanced TLR expression
•: Therapeutic implications for ECS modulation of TLR associated inflammation

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