Molecular cloning of canine co-chaperone small glutamine-rich tetratricopeptide repeat-containing protein 伪 (SGTA) and investigation of its ability to suppress androgen receptor signalling in androgen-independent prostate cancer

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Molecular cloning of canine co-chaperone small glutamine-rich tetratricopeptide repeat-containing protein 伪 (SGTA) and investigation of its ability to suppress androgen receptor signalling in androgen-independent prostate cancer

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作者：Yuiko Kato^a ; ¹ ; Kazuhiko Ochiai^a ; ¹ ; ^{kochiai@nvlu.ac.jp" class="auth_mail" title="E-mail the corresponding author} ; Masaki Michishita^b ; Daigo Azakami^a ; Rei Nakahira^b ; Masami Morimatsu^c ; Toshina Ishiguro-Oonuma^d ; Yasunaga Yoshikawa^e ; Masato Kobayashi^f ; Makoto Bonkobara^f ; Masanori Kobayashi^g ; Kimimasa Takahashi^b ; Masami Watanabe^h ; Toshinori Omi^a
关键词：Androgen receptor ; Canine ; Prostate ; Cancer ; SGTA
刊名：The Veterinary Journal
出版年：2015
出版时间：November 2015
年：2015
卷：206
期：2
页码：143-148
全文大小：1234 K

文摘

•: Canine SGTA shared a high homology with the human and mouse proteins.
•: SGTA is overexpressed in canine prostate cancer, but not in hyperplasia.
•: Canine SGTA forms homodimers.
•: Canine SGTA suppressed AR signalling in a prostate cancer cell line.