VIP protects human retinal microvascular endothelial cells against high glucose-induced increases in TNF-α and enhances RvD1

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VIP protects human retinal microvascular endothelial cells against high glucose-induced increases in TNF-α and enhances RvD1

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作者：Haoshen Shi^a ; ^{hshi@med.wayne.edu" class="auth_mail" title="E-mail the corresponding author} ; Thomas W. Carion^a ; ^{tcarion@med.wayne.edu" class="auth_mail" title="E-mail the corresponding author} ; Youde Jiang^a ; ^{youdejiang55@gmail.com" class="auth_mail" title="E-mail the corresponding author} ; Jena J. Steinle^a ; ^b ; ^{jsteinle@med.wayne.edu" class="auth_mail" title="E-mail the corresponding author} ; Elizabeth A. Berger^a ; ^b ; ^{eberger@med.wayne.edu" class="auth_mail" title="E-mail the corresponding author}
关键词：Neuropeptide ; Resolvins ; Diabetic retinopathy ; Treatment
刊名：Prostaglandins and Other Lipid Mediators
出版年：2016
出版时间：March 2016
年：2016
卷：123
期：Complete
页码：28-32
全文大小：1037 K

文摘

class="label">•: VIP treatment reduces high glucose-induced TNF-α levels in HREC.
class="label">•: High glucose reduces RvD1 expression by HREC.
class="label">•: VIP treatment restores RvD1 levels in HREC exposed to high glucose.
class="label">•: ALX/FPR2 and GPR32 are expressed on HREC.
class="label">•: VPAC2 is the predominant receptor expressed on HREC.

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