Suicide is a serious public health problem worldwide with an estimated annual mortality rate of 14.5 deaths per 100,000 people.
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FKBP5 and CRHR1 genes have been associated importantly with epigenetic alterations in molecular pathways of the function HPA axis and serotonergic system in patients with suicidal behavior.
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An understanding of the functional consequence of FKBP5 and CRHR1 genes and its polymorphisms from a neuropathological perspective would be convenient to advance our understanding of their role in the HPA axis alterations with suicidal behavior.
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