De-regulation of diabetic regulatory genes in psoriasis: Deciphering the unsolved riddle

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De-regulation of diabetic regulatory genes in psoriasis: Deciphering the unsolved riddle

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作者：Suad AlFadhli^a ; ^{s.alfadhli@hsc.edu.kw" class="auth_mail" title="E-mail the corresponding author} ; ^{suadq8@yahoo.com" class="auth_mail" title="E-mail the corresponding author} ; Alaa A.M. Al-Zufairi^a ; Rasheeba Nizam^a ; Huda A. AlSaffar^b ; Nawaf Al-Mutairi^c
关键词：Ps ; psoriasis ; Ps-T2D ; psoriasis type II diabetes ; T2D ; type II diabetes ; HC ; healthy controls ; B2M ; beta-2-microglobuli ; HPRT1 ; hypoxanthine phosphoribosyl transferase ; RPL13A ; ribosomal protein L13a ; GAPDH ; glyceraldehyde-3-phosphate dehydrogenase ; ACTB ; b-actin ; STX4 ; Syntaxin 4 ; GSK3B ; glycogen synthase kinase 3 beta ; PTPN1 ; protein tyrosine phosphatase ; non-receptor type 1 ; ACE ; angiotensin I converting enzyme (peptidyl-dipeptidase A) 1 ; PWBC ; peripheral white blood cells
刊名：Gene
出版年：2016
出版时间：15 November 2016
年：2016
卷：593
期：1
页码：110-116
全文大小：654 K

文摘

•: Our study highlights the genes involved in the pathogenesis of Ps-T2D.
•: We identified three biomarkers for T2D in Ps patients (GSK3B, PTPN1 and STX4).
•: ACE showed suggestive association with Ps.
•: We hypothesize that Ps and T2D share common causative gene/polygenes.

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