Adaptation to Stressors by Systemic Protein Amyloidogenesis

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Adaptation to Stressors by Systemic Protein Amyloidogenesis

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作者：Timothy E. Audas¹ ; ² ; ³ ; ⁷ ; Danielle E. Audas¹ ; ² ; ⁴ ; Mathieu D. Jacob³ ; J.J. David Ho¹ ; ² ; Mireille Khacho³ ; Miling Wang¹ ; ⁴ ; J. Kishan Perera³ ; Caroline Gardiner³ ; Clay A. Bennett¹ ; ² ; Trajen Head¹ ; Oleksandr N. Kryvenko² ; ⁴ ; ⁵ ; Mercé ; Jorda² ; ⁴ ; ⁵ ; Sylvia Daunert¹ ; Arun Malhotra¹ ; Laura Trinkle-Mulcahy³ ; ⁶ ; Mark L. Gonzalgo² ; ⁴ ; Stephen Lee¹ ; ² ; ³ ; ⁴ ; ⁸ ; ^{stephenlee@med.miami.edu" class="auth_mail" title="E-mail the corresponding author}
关键词：extracellular stress ; amyloid-bodies (A-bodies) ; physiological amyloidogenesis ; dormancy ; long noncoding RNA (lncRNA) ; β-amyloid ; heat shock chaperones ; Hsp70
刊名：Developmental Cell
出版年：2016
出版时间：24 October 2016
年：2016
卷：39
期：2
页码：155-168
全文大小：6343 K

文摘

•: Nuclear amyloid bodies (A-bodies) form from systemic protein amyloidogenesis
•: An amyloid-converting motif and ribosomal intergenic lncRNA mediate amyloidogenesis
•: The heat-shock chaperone pathway disaggregates A-bodies
•: Upon stimuli, cells activate physiological amyloidogenesis to enter a dormant state

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