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CNKI会议论文(1)
CNKI学位论文(87)
知网期刊论文(97)
在“
Elsevier电子期刊
”中,
命中:
43
条,耗时:小于0.01 秒
在所有数据库中总计命中:
185
条
1.
MLL
-
AF4
Spreading Identifies Binding Sites that Are Distinct from Super-Enhancers and that Govern Sensitivity to DOT1L Inhibition in Leukemia
作者:
Jon Kerry
1
;
Laura Godfrey
1
;
Emmanouela Repapi
2
;
Marta Tapia
1
;
Neil P. Blackledge
3
;
Helen Ma
4
;
Erica Ballabio
1
;
Sorcha O&rsquo
;
Byrne
5
;
Frida Ponthan
6
;
Olaf Heidenreich
6
;
Anindita Roy
5
;
Irene Roberts
1
;
5
;
Marina Konopleva
4
;
Robert J. Klose
3
;
Huimin Geng
7
;
Thomas A. Milne
1
;
8
;
thomas.milne@imm.ox.ac.uk
关键词:
MLL
;
MLL
-
AF4
;
DOT1L
;
H3K79me2
;
leukemia
;
epigenetic therapy
;
drug combination therapy
;
epigenetic spreading
刊名:Cell Reports
出版年:2017
2.
Instructive Role of
MLL
-Fusion Proteins Revealed by a Model of t(4;11) Pro-B Acute Lymphoblastic Leukemia
作者:
Shan Lin
1
;
6
;
Roger T. Luo
2
;
6
;
Anetta Ptasinska
3
;
Jon Kerry
4
;
Salam A. Assi
3
;
Mark Wunderlich
1
;
Toshihiko Imamura
2
;
Joseph J. Kaberlein
2
;
Ahmad Rayes
1
;
Mark J. Althoff
1
;
John Anastasi
5
;
Maureen M. O&rsquo
;
Brien
1
;
Amom Ruhikanta Meetei
1
;
Thomas A. Milne
4
;
Constanze Bonifer
3
;
James C. Mulloy
1
;
james.mulloy@cchmc.org" class="auth_mail" title="E-mail the corresponding author
;
Michael J. Thirman
2
;
7
;
mthirman@medicine.bsd.uchicago.edu" class="auth_mail" title="E-mail the corresponding author
关键词:
MLL
-
AF4
;
acute lymphoblastic leukemia
;
chimeric fusion proteins
;
mouse models of cancer
;
acquired resistance to targeted therapy
;
species specificity of oncogenes
刊名:Cancer Cell
出版年:2016
3.
Mll
-
AF4
Confers Enhanced Self-Renewal and Lymphoid Potential during a Restricted Window in Development
作者:
Neil A. Barrett
1
;
4
;
Camille Malouf
1
;
2
;
4
;
Chrysa Kapeni
1
;
2
;
Wendi A. Bacon
1
;
George Giotopoulos
1
;
Sten Eirik W. Jacobsen
3
;
Brian J. Huntly
1
;
Katrin Ottersbach
1
;
2
;
katrin.ottersbach@ed.ac.uk" class="auth_mail" title="E-mail the corresponding author
刊名:Cell Reports
出版年:2016
4.
Development Refractoriness of
MLL
-Rearranged Human B Cell Acute Leukemias to Reprogramming into Pluripotency
作者:
Alvaro Muñ
;
oz-Ló
;
pez
1
;
2
;
16
;
Damià
;
Romero-Moya
1
;
2
;
16
;
Cristina Prieto
1
;
2
;
Veró
;
nica Ramos-Mejí
;
a
3
;
Antonio Agraz-Doblas
1
;
2
;
4
;
Ignacio Varela
4
;
Marcus Buschbeck
1
;
Anna Palau
1
;
Xonia Carvajal-Vergara
5
;
Alessandra Giorgetti
1
;
Anthony Ford
6
;
Majlinda Lako
7
;
Isabel Granada
1
;
8
;
Neus Ruiz-Xivillé
;
1
;
8
;
Sandra Rodrí
;
guez-Perales
9
;
Raul Torres-Ruí
;
z
1
;
9
;
Ronald W. Stam
10
;
Jose Luis Fuster
11
;
Mario F. Fraga
12
;
Mahito Nakanishi
13
;
Gianni Cazzaniga
14
;
Michela Bardini
14
;
Isabel Cobo
1
;
12
;
Gustavo F. Bayon
12
;
Agustin F. Fernandez
12
;
Clara Bueno
1
;
2
;
cbueno@carrerasresearch.org" class="auth_mail" title="E-mail the corresponding author
;
Pablo Menendez
1
;
2
;
15
;
pmenendez@carrerasresearch.org" class="auth_mail" title="E-mail the corresponding author
关键词:
iPSC
;
cancer reprogramming
;
MLL
-
AF4
;
B-ALL
;
Sendai virus
;
transcriptome
;
DNA methylome
刊名:Stem Cell Reports
出版年:2016
5.
One Step Forward in the Challenging Arena of
MLL
-
AF4
Leukemia
作者:
Nancy J. Zeleznik-Le
1
;
nzelezn@luc.edu" class="auth_mail" title="E-mail the corresponding author
刊名:Cancer Cell
出版年:2016
6.
MLL
-Rearranged Acute Lymphoblastic Leukemias Activate BCL-2 through H3K79 Methylation and Are Sensitive to the BCL-2-Specific Antagonist ABT-199
作者:
Juliana M. Benito
1
;
Laura Godfrey
2
;
Kensuke Kojima
3
;
Leah Hogdal
4
;
Mark Wunderlich
5
;
Huimin Geng
6
;
Isabel Marzo
7
;
Karine G. Harutyunyan
1
;
Leonard Golfman
8
;
Phillip North
2
;
Jon Kerry
2
;
Erica Ballabio
2
;
Triona Ní
;
Chonghaile
9
;
Oscar Gonzalo
7
;
Yihua Qiu
1
;
Irmela Jeremias
10
;
LaKiesha Debose
1
;
Eric O&rsquo
;
Brien
5
;
Helen Ma
1
;
Ping Zhou
1
;
Rodrigo Jacamo
1
;
Eugene Park
6
;
Kevin R. Coombes
11
;
Nianxiang Zhang
11
;
Deborah A. Thomas
1
;
Susan O&rsquo
;
Brien
1
;
Hagop M. Kantarjian
1
;
Joel D. Leverson
12
;
Steven M. Kornblau
1
;
Michael Andreeff
1
;
Markus Mü
;
schen
6
;
Patrick A. Zweidler-McKay
8
;
James C. Mulloy
5
;
Anthony Letai
4
;
Thomas A. Milne
2
;
thomas.milne@imm.ox.ac.uk" class="auth_mail" title="E-mail the corresponding author
;
Marina Konopleva
1
;
mkonople@mdanderson.org" class="auth_mail" title="E-mail the corresponding author
关键词:
apoptosis pathways
;
leukemias
;
bcl-2 family members
;
MLL
/
AF4
;
DOT1L
;
H3K79 methylation
刊名:Cell Reports
出版年:2015
7.
Proteasome Inhibitors Evoke Latent Tumor Suppression Programs in Pro-B
MLL
Leukemias through MLL-
AF4
作者:
Han Liu
1
;
2
;
Todd D. Westergard
3
;
Amanda Cashen
3
;
David R. Piwnica-Worms
4
;
Lori Kunkle
5
;
Ravi Vij
3
;
Can G. Pham
1
;
John DiPersio
3
;
Emily H. Cheng
1
;
6
;
7
;
chenge1@mskcc.org" class="auth_mail
;
James J. Hsieh
1
;
8
;
9
;
hsiehj@mskcc.org" class="auth_mail
刊名:Cancer Cell
出版年:14 April, 2014
8.
t(4;11) leukemias display addiction to
MLL
-
AF4
but not to AF4-MLL
作者:
Ashish R. Kumar
a
;
b
;
ashish.kumar@cchmc.org
;
Qing Yao
a
;
Quanzhi Li
a
;
Thien A. Sam
a
;
John H. Kersey
a
;
b
;
c
关键词:
MLL
;
AF4
;
t(4
;
11)
;
Leukemia
;
11q23
刊名:Leukemia Research
出版年:2011
9.
Harnessing the Hidden Antitumor Power of the
MLL
-
AF4
Oncogene to Fight Leukemia
作者:
Smita Matkar
1
;
Bryson W. Katona
1
;
Xianxin Hua
1
;
huax@mail.med.upenn.edu" class="auth_mail
刊名:Cancer Cell
出版年:14 April, 2014
10.
Significance of
AF4
-
MLL
reciprocal fusion in t(4;11) leukemias?
作者:
Daniel S. Sanders
a
;
Andrew G. Muntean
a
;
Jay L. Hess
;
a
;
jayhess@umich.edu
关键词:
Reciprocal translocations
;
MLL
-
AF4
;
Leukemia
刊名:Leukemia Research
出版年:2011
1
2
3
4
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